ライフサイエンスコーパス: neurologic sign

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します

1 eight loss, fever, anorexia, depression, and neurologic signs.

2 of arrhythmia, age > 70 years) or with acute neurologic signs.

3 t EAE animals therapeutically after onset of neurologic signs.

4 nts with neurosyphilis who develop transient neurologic signs.

5 Among the 20 subjects with soft neurologic signs, 10 (50%) had abnormal MRI, 13 (65%) ha

6 fter cardiac surgery in the absence of focal neurologic signs, a poorly understood but potentially de

7 g anti-TNFalpha therapy immediately when new neurologic signs and symptoms occur, pending an appropri

8 urrent headaches, with or without additional neurologic signs and symptoms, and prolonged but reversi

9 use of a wide variety of other, more subtle, neurologic signs and symptoms.

10 cation (n=4), cardiac arrest (n=2), or other neurologic signs and symptoms.

11 ad life-threatening hypoxic episodes or soft neurologic signs, and 10 were normal based on neurologic

12 e is manifested as vascular necrosis, edema, neurologic signs, and death.

13 y 4 or 5 after infection due to anorexia and neurologic signs, but the SA EEEV-infected animals remai

14 Neurologic signs decreased with age in the healthy compa

15 of the infected macaques showed any fever or neurologic signs during the experimental period.

16 The persistence of neurologic signs in the adolescents with schizophrenia,

17 Without IL-12Rbeta2, no neurologic sign of ECM developed upon PbA infection.

18 sulted in the development of severe clinical neurologic signs of EAE with 100% mortality by day 17 po

19 ith a JHR, which possibly contributed to the neurologic signs of JHR.

20 testing is rarely helpful unless additional neurologic signs or symptoms are present (diagnostic yie

21 transient ischemic attack, stroke, or other neurologic signs or symptoms.

22 e selected from 220 samples from horses with neurologic signs resembling EPM and examined for inhibit

23 Only one pig developed clinical neurologic signs suggestive of prion disease.

24 Resolution of neurologic signs/symptoms occurred a median of 4 days an

25 ons, escalating corticosteroids, progressive neurologic signs/symptoms, or non-CNS progression--the t

26 Despite progressive neurologic signs, the proviral load in tissues, includin

27 samples, including samples from horses with neurologic signs typical of EPM or with histologically o

28 Neurologic signs were noted clinically, and subsequent i

WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。