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1 the gut, and it is also related to renal and neurologic symptoms.
2 omyopathy 11 months prior to presentation of neurologic symptoms.
3 luding gastrointestinal, musculoskeletal and neurologic symptoms.
4 orresponding evidence by clinical imaging or neurologic symptoms.
5 o a highly overlapping phenotype without the neurologic symptoms.
6 ical manifestations include hypertension and neurologic symptoms.
7 associated with bone, connective tissue, and neurologic symptoms.
8 R imaging of the brain for the evaluation of neurologic symptoms.
9 h rituximab and who subsequently develop new neurologic symptoms.
10 n of mutant animals and delayed the onset of neurologic symptoms.
11 ied an average of 13 days after the onset of neurologic symptoms.
12 ed in some cases with transient worsening of neurologic symptoms.
13 ppression, elevated liver enzyme levels, and neurologic symptoms.
14 vere xeroderma pigmentosum/Cockayne syndrome neurologic symptoms.
15 , in the other patient, resulted in improved neurologic symptoms.
16  and 67% of patients younger than age 80 had neurologic symptoms.
17 with intellectual disability and progressive neurologic symptoms.
18 infarction, and all had consistent, acquired neurologic symptoms.
19 juni in their stools at the time of onset of neurologic symptoms.
20 tients have shown symptomatic improvement in neurologic symptoms, 1 patient has developed heart failu
21 rmed for gastrointestinal obstruction (34%), neurologic symptoms (23%), pain (12%), dyspnea (9%), jau
22  100% had gastrointestinal symptoms, 34% had neurologic symptoms, 43% had hemorrhagic manifestations,
23 , conjunctivitis, headache, and other severe neurologic symptoms 7 to 24 hours after hemodialysis dre
24 lidocaine to reduce the problem of transient neurologic symptoms after spinal blockade.
25  loads were detected in 42% of patients with neurologic symptoms and 44% of voluntary blood donors.
26 s suggest that the presence of preprocedural neurologic symptoms and a history of CAD are associated
27 tionship usually exists between the onset of neurologic symptoms and an infection or a vaccination.
28                      Plaques associated with neurologic symptoms and asymptomatic plaques were compar
29  of immune response associated with distinct neurologic symptoms and cancers.
30 ic regression to evaluate the association of neurologic symptoms and examination findings with FM sta
31                       The patient denies any neurologic symptoms and has no significant findings on n
32  with vision loss or diplopia accompanied by neurologic symptoms and in the absence of an intraocular
33  healthy subjects and 33 of 37 patients with neurologic symptoms and normal CSF and imaging studies c
34 tion carries a significant risk of transient neurologic symptoms and should be avoided.
35 f cyclovertical muscles, in association with neurologic symptoms and signs and with posterior fossa l
36          We report on 7 female patients with neurologic symptoms and signs including bilateral thalam
37 ith severe and persistent headache and other neurologic symptoms and signs should be considered for n
38 mboembolic events, seizures, fluctuations in neurologic symptoms, and adverse effects from corticoste
39  his hands and feet, with associated fevers, neurologic symptoms, and arthritis, who required hospita
40 ytopenia, microangiopathic hemolytic anemia, neurologic symptoms, and renal insufficiency, making the
41 , stage of disease, duration and severity of neurologic symptoms, and therapeutic intervention.
42                                              Neurologic symptoms are more severe and more likely to b
43 n abnormal human HD gene (line R6/2) develop neurologic symptoms at 9-11 weeks of age through an unkn
44        Forty-three (52%) of the patients had neurologic symptoms at diagnosis.
45         The efavirenz group experienced more neurologic symptoms at week 1 (P < 0.001) but not at wee
46 g and occurs in two distinct forms, an acute neurologic symptom complex that occurs within hours or d
47 s, in these experiments, the distribution of neurologic symptoms depends on the expression level and
48          Quantified HCHO was associated with neurologic symptoms (difficulty concentrating POR 1.47;
49 led trial, efavirenz use was associated with neurologic symptoms distinct from depression and anxiety
50                                     However, neurologic symptoms do not manifest until late stages of
51 ay provide an anatomic basis for some of the neurologic symptoms found in FIV-infected cats and HIV-i
52 in the treatment of CNS metastases; however, neurologic symptoms frequently are not fully reversible,
53 performed on 54853 patients with unexplained neurologic symptoms identified 1992 patients (4%) who we
54 available 1 week prior to the development of neurologic symptoms in 11 patients.
55                              The presence of neurologic symptoms in aceruloplasminemia is unique amon
56 ing plerixafor had an increased incidence of neurologic symptoms in association with CNS infiltration
57 er, LPL(-/-) mice developed much less severe neurologic symptoms in experimental autoimmune encephalo
58 opment is consistent with the development of neurologic symptoms in med and jolting mice, which have
59 ination is largely responsible for sustained neurologic symptoms in multiple sclerosis (MS).
60                           Increased focus on neurologic symptoms in patients with rosacea may be warr
61 eloped into adulthood, did not develop overt neurologic symptoms in the first year of life, and had n
62                                         Some neurologic symptoms in the form of seizures and nystagmu
63 ients), new or worsening back pain (in 50%), neurologic symptoms (in 48%), nausea (in 39%), and stiff
64                                        Other neurologic symptoms included eye movement abnormalities
65                                Preprocedural neurologic symptoms included transient ischemic attack,
66                                              Neurologic symptoms lasting at least 3 months were asses
67 e finding of immunity to Ma proteins is that neurologic symptoms may improve or resolve.
68  studies are best reserved for children with neurologic symptoms, neuropsychometric deficits, or elev
69                                              Neurologic symptoms occurred in 38% of reports.
70               After logistic regression, the neurologic symptoms (odds ratio: 48.7, 95% confidence in
71  concentrations but do not display the usual neurologic symptoms of hyperammonemia.
72  being applied to the study of the transient neurologic symptoms of the aura, as well as the painful
73                                Median age at neurologic symptom onset was 42 years (range, 21-73 year
74                                    He denied neurologic symptoms or worsening of pain while lying dow
75 re referred for feeding difficulties, subtle neurologic symptoms, or delayed psychomotor development
76 ar cardiac phenotype but a greater burden of neurologic symptoms (pain, numbness, tingling, and walki
77 siologic processes leading to development of neurologic symptoms, particularly in patients with syste
78  anthrax include the presence of nonheadache neurologic symptoms (positive likelihood ratio cannot be
79                                    Immediate neurologic symptoms postinjury were used to identify mil
80    None of the patients had ever experienced neurologic symptoms prior to the episode of optic neurit
81 l brain lesions in patients with unexplained neurologic symptoms represents a challenge.
82 yperactivity are more susceptible to develop neurologic symptoms such as mental retardation and autis
83                        The FM group had more neurologic symptoms than did the controls, with moderate
84 milarly, the FM group had significantly more neurologic symptoms than the control group in 27 of 29 c
85 ist therapy is supported by the worsening of neurologic symptoms that occurred in our index patient f
86          Among older patients and those with neurologic symptoms, the syndrome often progresses to re
87  7% of HIV-infected patients presenting with neurologic symptoms; the diagnosis of VZV-related CNS di
88 ght patients with preeclampsia-eclampsia and neurologic symptoms underwent magnetic resonance (MR) im
89 d cases demonstrate partial reversibility of neurologic symptoms upon restoration of CNS creatine lev
90                       Median age at onset of neurologic symptoms was 58 (range, 27-83) years, and 87
91 lings (P=0.004), and the respective rates of neurologic symptoms were 4.0% and 9.4% (P=0.01).
92   The incidence of HAM/TSP and new signs and neurologic symptoms were computed in a group of patients
93              Changes in efavirenz-associated neurologic symptoms were correlated to efavirenz plasma
94                                Preprocedural neurologic symptoms were present in 32%.
95 4 consecutive HIV-infected patients with new neurologic symptoms were tested for VZV DNA by a polymer
96                              Neutropenia and neurologic symptoms were the most frequent reasons for e
97 s with newly diagnosed SLE, who had no focal neurologic symptoms, were studied.
98  model was instituted on animal's display of neurologic symptoms, which usually led to rapid demise.
99                                Resolution of neurologic symptoms with initial steroid therapy, relaps

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