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1 ion that formed nonadsorbing products (i.e., p-benzoquinone).
2 te duroquinone (2.5 A) (2,3,5, 6-tetramethyl-p-benzoquinone).
3 , Bu3SnD, and pyridine.BD3 with 2,5-dichloro-p-benzoquinone.
4 tD oxidizes the latter to 5-chloro-2-hydroxy-p-benzoquinone.
5 echol, hydroquinone, 1,2,4-benzenetriol, and p-benzoquinone.
6 sence of other quinones such as 2,5-dichloro-p-benzoquinone, 2, 5-dimethyl-p-benzoquinone, and p-benz
7 asily oxidized hydroquinone (hydroquinone<-->p-benzoquinone + 2H+ + 2e).
8 cide DBMIB (2,5-dibromo-3-methyl-6-isopropyl-p-benzoquinone), a well-known inhibitor of photosyntheti
9 ensitive to 2,5-dibromo-3-methyl-6-isopropyl-p-benzoquinone, a cytochrome b6f complex inhibitor.
10 e/relaxation assay, topo II was inhibited by p-benzoquinone and hydroquinone at 10 microM and 10 mM,
11 ia direct electron transfer reactions, while p-benzoquinone and terephthalic acid are not.
12 inistration of N-acetyl-p-benzoquinoneimine, p-benzoquinone and the electrophilic TRPA1 activator cin
13 methoxy-3-[(Z,Z)-8',11',14'-pentadecatriene]-p-benzoquinone) and its analogs.
14 h an analyte concentration (e.g., 0.1-2.5 mM p-benzoquinone) and with an analyte feeding rate (i.e.,
15 photodeprotection process by the presence of p-benzoquinone, and absence of a labeled carbonyl final
16 s 2,5-dichloro-p-benzoquinone, 2, 5-dimethyl-p-benzoquinone, and p-benzoquinone, QA could be reduced
17              Macrocyclic metal complexes and p-benzoquinones are commonly used as co-catalytic redox
18 ith the enzymatic oxidation of lactose using p-benzoquinone as electron acceptor and the electrochemi
19                                         With p-benzoquinone as the electron acceptor, cell extracts o
20 nisms of p-nitrophenol, p-methoxyphenol, and p-benzoquinone at a porous Ti4O7 reactive electrochemica
21                                 Reduction of p-benzoquinone at the cathodic end of the shared electro
22 SSG mixture via a 1,4-addition reaction with p-benzoquinone (BQ), followed by enzymatic kinetic measu
23 c inhibitor 2,5-dibromo-3-methyl-6-isopropyl-p-benzoquinone but this induction requires the presence
24 metabolites N-acetyl-p-benzoquinoneimine and p-benzoquinone, but not acetaminophen itself, activate m
25 diate O2 reduction and generate the reactive p-benzoquinone co-catalyst.
26 nditions, however, a buffer containing 50 mM p-benzoquinone completely suppressed both cathodic reduc
27 rgetics of such an intermediate, cyclopropyl-p-benzoquinone (CPBQ) is shown to be a specific inhibito
28  derivative 2,5-dibromo-3-methyl-6-isopropyl-p-benzoquinone (DBMIB), a known inhibitor of the bc1 and
29 none analog 2,5-dibromo-3-methyl-6-isopropyl-p-benzoquinone (DBMIB), and the oxidized form of DBMIB,
30  and 75% by 2,5-dibromo-3-methyl-6-isopropyl-p-benzoquinone (DBMIB), which inhibits electron transfer
31 none analog 2,5-dibromo-3-methyl-6-isopropyl-p-benzoquinone (DBMIB).
32 evolution rates of 42-57% using 2,6-dichloro-p-benzoquinone (DCBQ) as an artificial electron acceptor
33 ide abstractions by 2,3-dichloro-5,6-dicyano-p-benzoquinone (DDQ) from 13 C-H hydride donors (acyclic
34        Reactions of 2,3-dichloro-5,6-dicyano-p-benzoquinone (DDQ) with silyl enol ethers, silyl keten
35 erived heats of hydrogenation of o-, m-, and p-benzoquinone (Delta(hyd)H degrees (1o, 1m, and 1p) = 4
36 haracterized inducing factor, 2, 6-dimethoxy-p-benzoquinone (DMBQ), can be used to trigger in vitro h
37 p-chlorobenzoic acid, terephthalic acid, and p-benzoquinone) for use in EAOPs.
38 Alder adducts of cyclopentadiene and 2-allyl-p-benzoquinone, has been devised.
39 he Diels-Alder adduct of cyclopentadiene and p-benzoquinone, has been devised.
40 onverted to the reactive metabolite N-acetyl-p-benzoquinone-imide (NAPQI) (r= 0.739;P= 0.058).
41 icity through enhanced formation of N-acetyl-p-benzoquinone imine (NAPQI) via induction of cytochrome
42                                     N-acetyl-p-benzoquinone imine (NAPQI), a reactive metabolite of a
43  that the acetaminophen metabolite, N-acetyl-p-benzoquinone imine (NAPQI), covalently binds to the ac
44 uinone metabolite of acetaminophen, N-acetyl-p-benzoquinone imine (NAPQI), inhibits both the isomeras
45 ate APAP to the reactive metabolite N-acetyl-p-benzoquinone imine (NAPQI).
46 production of its toxic metabolite, N-acetyl-p-benzoquinone imine (NAPQI).
47             The reactive metabolite N-acetyl-p-benzoquinone imine has long been proven to be responsi
48 ugation and reduced accumulation of N-acetyl-p-benzoquinone imine, a toxic electrophile that is produ
49 that produced the toxic metabolite, N-acetyl-p-benzoquinone imine, higher levels of reduced glutathio
50 avenging of the reactive metabolite N-acetyl-p-benzoquinone imine, protective mechanisms at later tim
51 tive intermediates benzoquinone and N-acetyl-p-benzoquinone imine, which can subsequently react with
52 rt acetaminophen to highly reactive N-acetyl-p-benzoquinone imine.
53 hrome P4502E1 protein expression or N-acetyl-p-benzoquinone-imine formation.
54       The Diels-Alder reaction between 5 and p-benzoquinone in boiling glacial acetic acid yields an
55 ling of aromatic aldehydes (or alcohols) and p-benzoquinone led to an ester in the presence of the Cu
56 sed), but a value of about 10 kJ mol(-1) for p-benzoquinone loss, which is consistent with formation
57 2, with either DDQ (2,3-dichloro-5,6-dicyano-p-benzoquinone) or TBHP (tert-butyl hydroperoxide), alon
58       The EGB, obtained when electrolysis of p-benzoquinone, or 1,4-naphthoquinone, is carried out at
59 s, dihydroxybenzoquinone, dichloro-dihydroxy-p-benzoquinone, or benzene decorated by -COOH groups exh
60 unction of organic compounds (p-nitrophenol, p-benzoquinone, p-methoxyphenol, and oxalic acid) and cu
61 r AP sites or the chemically unrelated bulky p-benzoquinone (pBQ) derivatives of dC, dA and dG, all o
62                                              p-Benzoquinone (pBQ), one of the major benzene metabolit
63  metabolite of the human carcinogen benzene, p-benzoquinone (pBQ).
64 zoquinone, 2, 5-dimethyl-p-benzoquinone, and p-benzoquinone, QA could be reduced but could not effici
65 nal theory (DFT) calculations indicated that p-benzoquinone removal was primarily due to reaction wit
66                                 Formation of p-benzoquinone replaces the formation of oxygen gas, eff
67 (-)(1), respectively, indicating that o- and p-benzoquinone should be excellent radical traps.
68 ha, beta-dehydrogenated derivatives of nonyl-p-benzoquinones that originated by hydroxylation induced
69  chromophore trianisylamine and nonabsorbing p-benzoquinone, the phase angle difference between absor
70 (Et(4)N)(2) (4) reacts rapidly with TEMPO or p-benzoquinones to generate diferric and deprotonated [F
71                Radical anions of o-, m-, and p-benzoquinone were produced in a Fourier transform mass
72               3-Amino-6-chloropyridazine and p-benzoquinone were responsible for the increased toxici
73 roquinone as the substrate 2,3-disubstituted p-benzoquinones were isolated.
74 rting materials the 2,3,5,6-tetrasubstituted p-benzoquinones were isolated.
75                           Highly substituted p-benzoquinones were obtained in yields ranging from 39%
76 In almost all cases the 2,3,5-trisubstituted p-benzoquinones were obtained.
77            In contrast to the thermolysis of p-benzoquinone, which does not decompose until the tempe
78 -trichlorophenol (2,4,5-TCP) to 2,5-dichloro-p-benzoquinone, which is chemically reduced to 2,5-dichl
79 ant was replaced by 2,3-dichloro-5,6-dicyano-p-benzoquinone, which is frequently used at the oxidizin
80 r oxygen oxidized to the corresponding nonyl-p-benzoquinones-yielding a complex mixture of potentiall

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