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1 r by ascorbate + TMPD (N,N,N',N'-tetramethyl-p-phenylenediamine).
2 ascorbate and the electron donor tetramethyl-p-phenylenediamine.
3 lenediamine subunit compared to that of free p-phenylenediamine.
4 itis is commonly associated with exposure to p-phenylenediamine.
5 ally initiated reaction with aqueous diethyl-p-phenylenediamine.
6 -glycerophosphate, and N,N,N',N'-tetramethyl-p-phenylenediamine.
7 donor system ascorbate-N,N,N',N'-tetramethyl-p-phenylenediamine.
8 as also inhibited when N,N,N',N'-tetramethyl-p-phenylenediamine (0.5 mM) and ascorbate (5 mM) were us
9 n of doubly trimethylene-bridged tetrabenzyl-p-phenylenediamine 1(Bz) in its singly and doubly charge
10 y spray detection with N,N,N',N'-tetramethyl-p-phenylenediamine and densitometric scanning of the pur
11 sulted in either a loss or retention of both p-phenylenediamine and ferroxidase activities, indicatin
12 toring is demonstrated using the reaction of p-phenylenediamine and isobutyraldehyde to form the diim
13                                        m- or p-phenylenediamine and m- or p-chlorophenyl-substituted
14 s butylated hydroxytoluene and N,N'-diphenyl-p-phenylenediamine and the iron chelator deferoxamine.
15 nctional redox mediator, 2,3,5,6-tetramethyl-p-phenylenediamine, and presents superb energy density a
16 pable of oxidizing other substrates, such as p-phenylenediamine, and there is still a question of whe
17 razolyl-substituted anilines and o-, m-, and p-phenylenediamine as pi-conjugated spacers.
18 compared to those of the linear tetra-phenyl-p-phenylenediamine as well as the tetra-p-anisyl-p-tetra
19 ch are derived from cholic acid, lysine, and p-phenylenediamine, can produce pores in lipid bilayers
20                          Using a tetramethyl-p-phenylenediamine cytochrome c oxidase screen, 27 oxida
21              The electrochemistry of several p-phenylenediamine derivatives, in which one of the amin
22 pamine (DA), tyrosine (Tyr) and N,N-dimethyl-p-phenylenediamine (DMPA), were evaluated using methanol
23 e formed radicals converted the N,N-dimethyl-p-phenylenediamine (DMPD) probe to the colored DMPD(+) r
24 acterized DbetaM reductant, N,N-dimethyl-1,4-p-phenylenediamine (DMPD), were parallel to the ascorbic
25         A modified acid-quenched N,N-diethyl-p-phenylenediamine (DPD) assay was used to measure the a
26  response of a 10% (by weight) N,N'-diphenyl-p-phenylenediamine (DPPD) and 90% (by weight) carbon and
27 umption rate supported by 0.4 mM tetramethyl-p-phenylenediamine in antimycin-inhibited uncoupled inta
28 e artificial reductant N,N,N',N'-tetramethyl-p-phenylenediamine in place of ubiquinol was, however, u
29 on between hydrogen sulfide and N,N-dimethyl-p-phenylenediamine in the presence of iron(III) chloride
30 s attached with a dansyl group, in which the p-phenylenediamine moiety serves as electron donor and t
31  match and charge density sensitivity of the p-phenylenediamine moiety.
32 his locus displayed an N,N,N',N'-tetramethyl-p-phenylenediamine oxidase-negative phenotype, elicited
33 e coupled to quinol or N,N,N',N'-tetramethyl-p-phenylenediamine oxidation, and the activity was sensi
34 s of 1,3,5-triformylphloroglucinol (Tp) with p-phenylenediamine (Pa-1) and 2,5-dimethyl-p-phenylenedi
35 h p-phenylenediamine (Pa-1) and 2,5-dimethyl-p-phenylenediamine (Pa-2), respectively, in 1:1 mesityle
36 e aim of this study was to determine whether p-phenylenediamine (PPD) and/or Bandrowski's base (BB) s
37                                  Exposure to p-phenylenediamine (PPD) is associated with the developm
38 individuals' T-lymphocytes after exposure to p-phenylenediamine (PPD).
39 ne (6TDA), 1,5-naphthalenediamine (NDA), and p-phenylenediamine (PPDA) in human urine.
40                 The novel biocompatible poly(p-phenylenediamine) (PpPDA)-Fe3O4 nanocomposite (PpPDA@F
41     The lipophilic antioxidant N,N'-diphenyl-p-phenylenediamine protected TLF-1-treated T. brucei bru
42 eceptors, 1-4, based on the incorporation of p-phenylenediamine(s) within a urea framework, were synt
43 g of PPD to cells and serum, did not prevent p-phenylenediamine-specific stimulation of patient lymph
44 ced electron-transfer sensors were made from p-phenylenediamine-substituted azacrown ethers attached
45 n increase in the oxidation potential of the p-phenylenediamine subunit compared to that of free p-ph
46 s similar conformations as the other dimeric p-phenylenediamines, such as derivatives 1(Me) and 1(Et)
47 of unsymmetrically functionalized tetraalkyl-p-phenylenediamine (TAPD) units which are difficult to s
48 e rate with ascorbate-N,N,N',N',-tetramethyl-p-phenylenediamine (TMPD) as a reductant.
49 otable finding is the ability of tetramethyl-p-phenylenediamine (TMPD) to oxidize (interrogate) H(ads
50           Addition of N,N,N',N'-tet-ramethyl-p-phenylenediamine (TMPD) to the ascorbate-reduced potas
51 cin A and cyanide, and N,N,N',N'-tetramethyl-p-phenylenediamine (TMPD) was oxidized by antimycin A-po
52  The radical cation of N,N,N',N'-tetramethyl-p-phenylenediamine (TMPD), formed through oxidation by (
53 ogical electron donor, N,N,N',N'-tetramethyl-p-phenylenediamine (TMPD), may overcome the resistance o
54 ate in the presence of N,N,N',N'-tetramethyl-p-phenylenediamine (TMPD).
55 s were cut in cross-section and stained with p-phenylenediamine to visualize myelin.
56  from N,N'-bis(carboxymethyl)-N,N'-dinitroso-p-phenylenediamine using an assay that combines catalase
57  or by ascorbate plus N,N,N', N'-tetramethyl-p-phenylenediamine via cytochrome c1 and the iron-sulfur
58 redox reactions of ferrocene and tetramethyl-p-phenylenediamine were obtained in supercritical CO2 in
59 ansfer complex (TMPD = N,N,N',N'-tetramethyl-p-phenylenediamine) with a 1.492 (2) A central sp(2)[bon

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