ライフサイエンスコーパス: passive smoking

1 fects of acute exposure to sidestream smoke (passive smoking).

2 The ill health effects have been extended to passive smoking.

3 % CI, 1.00-1.45), compared with never active/passive smoking.

4 t ownership, presence of older siblings, and passive smoking.

5 ecreases were primarily due to reductions in passive smoking.

6 history of asthma, higher social class, and passive smoking.

7 d VEGF and PEDF expression in the context of passive smoking.

8 k of heart disease in individuals exposed to passive smoking.

9 percent had cotinine levels consistent with passive smoking.

10 from a database of symposium proceedings on passive smoking.

11 nt confidence interval, 2.4 to 7.3) and with passive smoking among nonsmokers (odds ratio, 2.5; 95 pe

12 (CIs) for former and current smoking and for passive smoking among nonsmokers compared with a referen

13 ever smokers or lifetime intensity-years of passive smoking among nonsmokers, whereas it decreased w

14 sk of coronary heart disease associated with passive smoking among nonsmokers.

15 The associations observed between passive smoking and adverse health outcomes have generat

16 Adjusting for passive smoking and antiretroviral therapy had little ef

17 We examined the relation between passive smoking and COPD and respiratory symptoms in an

18 ls were used to estimate the associations of passive smoking and genetic susceptibility with birth we

19 es modified the association between maternal passive smoking and infant birth weight in this study, w

20 s a significant interaction between maternal passive smoking and maternal EPHX1 genotype for birth we

21 t examined the association between active or passive smoking and miscarriage were included in the met

22 a modest association between first-trimester passive smoking and oral clefts that was consistent acro

23 examined the associations between active or passive smoking and perinatal death were included in the

24 s the relation between household exposure to passive smoking and serum concentrations of retinol, toc

25 estigating the relationship between maternal passive smoking and the risk of preterm birth have reach

26 dings support a positive association between passive smoking and the risk of very preterm birth.

27 ercent for cigarette smoking, 17 percent for passive smoking, and 14 percent for chronic illness.

28 and clinical outcomes were adjusted for sex, passive smoking, and body mass index.

29 various smoking behaviours (active smoking, passive smoking, and smoking cessation) with risk of typ

30 Active and passive smoking are associated with significantly increa

31 The effects of even brief (minutes to hours) passive smoking are often nearly as large (averaging 80%

32 Active and passive smoking are the first and third leading preventa

33 male nurses, in whom we assessed exposure to passive smoking at home and at work as well as duration

34 n risk of COPD and self-reported exposure to passive smoking at home and work (adjusted odds ratio 1.

35 these data suggest that regular exposure to passive smoking at home or work increases the risk of CH

36 8) among women reporting regular exposure to passive smoking at home or work.

37 ashington County, MD, to compare exposure to passive smoking at home, recorded in a private census of

38 FVR increased with passive smoking, but this increase was not significantly

39 esults were stratified by maternal genotype, passive smoking conferred a significantly negative effec

40 Exposure to passive smoking during pregnancy was associated with an

41 reported respiratory symptoms and increasing passive smoking exposure (1.16, 1.07-1.25 for any sympto

42 moking (p(interaction) = 0.004) or postnatal passive smoking exposure (p(interaction) = 0.004).

43 We measured passive smoking exposure at home and work by two self-re

44 rmation about lifetime smoking and household passive smoking exposure in 1995 and about lifetime expo

45 The association between passive smoking exposure in childhood and adverse cardio

46 owever, previous studies did not account for passive smoking exposure in the never-smoking reference

47 ons remained and did not appear to be due to passive smoking exposure in utero.

48 heart attack in individuals attributable to passive smoking exposure is poorly understood.

49 In the passive smoking group, there was a remarkable decrease i

50 osure in 1995 and about lifetime exposure to passive smoking in 3 settings (household, workplace, and

51 piratory distress syndrome patients and with passive smoking in 41% of nonsmokers (vs 20% and 40% in

52 he relationship of HDL cholesterol levels to passive smoking in children and adolescents referred to

53 ssociation of decreased HDL cholesterol with passive smoking in children but have not adjusted for po

54 strong evidence for urgent measures against passive smoking in China.

55 blems, eczema, rehospitalization in infancy, passive smoking in infancy, family history of asthma, an

56 among never smokers could be attributable to passive smoking in the current population in China.

57 ew studies have taken account of exposure to passive smoking in the workplace.

58 of the associations observed for active and passive smoking indicates that exposure to passive smoki

59 and radon) and other environmental factors (passive smoking, indoor radon, and air pollution).

60 onal studies are needed to determine whether passive smoking is a risk factor for bladder cancer.

61 e was to review the scientific evidence that passive smoking is associated with 1 or more health outc

62 Passive smoking is associated with a small increase in t

63 Exposure to passive smoking is associated with an increased prevalen

64 Reduction of passive smoking is important not only for the prevention

65 rall, 37% (39/106) of reviews concluded that passive smoking is not harmful to health; 74% (29/39) of

66 only factor associated with concluding that passive smoking is not harmful was whether an author was

67 Passive smoking may be a modifiable risk factor for dent

68 oking alone or in combination with postnatal passive smoking may be important in the risk of childhoo

69 These findings suggest that passive smoking may increase the risk of malignant lymph

70 ent study provides evidence that smoking and passive smoking may influence NHL etiology, particularly

71 d passive smoking indicates that exposure to passive smoking may result in decreased circulating conc

72 Passive smoking may worsen the risk profile for later at

73 The effect of passive smoking on the risk of coronary heart disease is

74 ha authors examined the effect of active and passive smoking on ventilatory function assessed by spir

75 active smoking, current active smoking, and passive smoking only were, respectively, 1.36 (95% CI, 1

76 focus specifically on the health effects of passive smoking or if they were not written in English.

77 1.03, 2.22) for >40 years vs. </=5 years of passive smoking; P for trend = 0.03), particularly for f

78 [95% CI 0.92-1.15]), but was associated with passive smoking (pooled RR 1.10 [95% CI 1.06-1.15]).

79 ive (pooled RR, 1.36 [95% CI 1.17-1.46]) and passive smoking (pooled RR, 1.06 [95% CI 1.01-1.11]).

80 ive (pooled RR, 1.21 [95% CI 1.14-1.29]) and passive smoking (pooled RR, 1.07 [95% CI 1.03-1.12]).

81 tive (pooled RR, 1.40 (95% CI 1.24-1.59) and passive smoking (pooled RR, 1.09 [95% CI 1.04-1.14]).

82 for maternal age, education, parity, stress, passive smoking, prepregnancy weight and height, and inf

83 (MI) and feedback of urine cotinine to stop passive smoking (PS) in children at risk for asthma.

84 creased with increasing lifetime exposure to passive smoking (relative risk = 1.51 (95% CI: 1.03, 2.2

85 age, sex, exercise, and dietary fat intake, passive smoking remained a significant risk factor for d

86 There is a publication delay for passive smoking studies with nonsignificant results comp

87 Among nonsmokers, exposure to passive smoking tended to be associated with lower serum

88 The associations were weaker for passive smoking than for active smoking.

89 Compared with women not exposed to passive smoking, the relative risks of total CHD-adjuste

90 imated the population attributable risk from passive smoking to be 27% for decayed and 14% for filled

91 Evidence linking active or passive smoking to the incidence of adult-onset asthma i

92 luences of asthma and household environment (passive smoking, use of a gas stove, and having a dog or

93 Despite the fact that exposure to passive smoking was assessed by self-report and only at

94 st increased risk for allergic diseases, and passive smoking was associated with an increased risk fo

95 y, we examined whether childhood exposure to passive smoking was associated with carotid atherosclero

96 In asthmatic girls, passive smoking was associated with reduced lung functio

97 There was little evidence that exposure to passive smoking was associated with reduced serum concen

98 Passive smoking was consistently associated with an incr

99 Although passive smoking was not associated with the risk of peri

100 for trend = 0.02) when women with household passive smoking were excluded from the reference categor

101 1.57) higher risk when women with household passive smoking were excluded from the reference categor

102 ant when the number of cigarettes smoked and passive smoking were included as covariates (relative ri

103 ues, the effects of postnatal ETS, mimicking passive smoking, were equivalent to (AC) or greater than

104 cal studies have suggested an association of passive smoking with coronary heart disease (CHD).

105 yr113His, affect the association of maternal passive smoking with infant birth weight.

106 e smoking, the public health consequences of passive smoking with regard to coronary heart disease ma

107 e examined prospectively the relationship of passive smoking with risk of CHD in a cohort of women.

108 d stroke, but the relationship of active and passive smoking with the progression of atherosclerosis

109 population is also exposed to high levels of passive smoking, yet little information exists on the ef