戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 and the CPEB4 autoamplification loop induces pathologic angiogenesis.
2 d regression in vivo in both physiologic and pathologic angiogenesis.
3  the present study, we show that SOCS3 curbs pathologic angiogenesis.
4 to the progression of both developmental and pathologic angiogenesis.
5 ole in vascular development, remodeling, and pathologic angiogenesis.
6 tifies a potential new target for control of pathologic angiogenesis.
7 protein, and mRNA levels of key mediators of pathologic angiogenesis.
8 expressed in endothelial cells and regulates pathologic angiogenesis.
9 inhibitors as a probable strategy to control pathologic angiogenesis.
10 e past decades to study both physiologic and pathologic angiogenesis.
11 (VEGFR2) plays a key role in physiologic and pathologic angiogenesis.
12  an enigmatic vascular tumor and a model for pathologic angiogenesis.
13 ctor is a key regulator of physiological and pathologic angiogenesis.
14 lls and plays a role in both physiologic and pathologic angiogenesis.
15 s, one must understand the many dualities of pathologic angiogenesis.
16 served that was specifically associated with pathologic angiogenesis.
17 th anti-CD31 antibodies, suggesting impaired pathologic angiogenesis.
18  helps to regulate vascular permeability and pathologic angiogenesis.
19  by endothelial cells at sites of normal and pathologic angiogenesis.
20                       Tie2 was implicated in pathologic angiogenesis.
21 g endogenous endothelial cell populations in pathologic angiogenesis.
22 ks the proinflammatory cytokine TNF alpha to pathologic angiogenesis.
23 nal vascular growth may provide insight into pathologic angiogenesis.
24 rtant new therapeutic agents for controlling pathologic angiogenesis.
25 ssociated with retinal diseases that involve pathologic angiogenesis.
26        VEGF is a key regulator of normal and pathologic angiogenesis.
27  growth factor (VEGF) is a major mediator of pathologic angiogenesis, a process necessary for the for
28 is during normal development, as well as the pathologic angiogenesis accompanying several ocular diso
29 s a pivotal endogenous feedback inhibitor of pathologic angiogenesis and a potential therapeutic targ
30 that plays a pivotal role in physiologic and pathologic angiogenesis and is a therapeutic target for
31 e in both embryonic vascular development and pathologic angiogenesis, and they are consistent with a
32 oxygen-induced retinopathy model, a model of pathologic angiogenesis, and this model was also used to
33  their transport and delivery in vivo during pathologic angiogenesis are not well understood.
34 could be a potential endogenous regulator of pathologic angiogenesis associated with atherosclerosis
35 tal hypertension and cirrhosis: reduction of pathologic angiogenesis, attenuation of liver fibrogenes
36 rther suggest that TR3/Nur77 is required for pathologic angiogenesis but not for developmental/physio
37 caffeine attenuated not only hypoxia-induced pathologic angiogenesis, but also hyperoxia-induced vaso
38 e master regulator of both developmental and pathologic angiogenesis, composed of an oxygen-sensitive
39 gents with the capacity to completely ablate pathologic angiogenesis could be of substantial utility
40                                              Pathologic angiogenesis has emerged as an important ther
41  found in several conditions associated with pathologic angiogenesis; however, its distinct role in t
42  blocking KDR may be beneficial for treating pathologic angiogenesis in adult tissue.
43 ine whether the Tie2 pathway was involved in pathologic angiogenesis in adult tissues, a soluble form
44                                              Pathologic angiogenesis in chronically inflamed tissues
45 CS3 in vessels and studied developmental and pathologic angiogenesis in murine models of oxygen-induc
46     However, Cd151-null mice showed impaired pathologic angiogenesis in other in vivo assays (Matrige
47 ssess effects on retinal vascular repair and pathologic angiogenesis in relation to glial cell injury
48 the human disease, including erythrocytosis, pathologic angiogenesis in the brain and retina, and abe
49  that FZD4 is required for physiological and pathologic angiogenesis in the retina and for regulation
50 key regulator of EC function in vitro and of pathologic angiogenesis in vivo and is potentially an at
51 al cells, which are needed for some types of pathologic angiogenesis in vivo.
52                                              Pathologic angiogenesis induced by cav-1 in prostate can
53 urally defective, leaky blood vessels typify pathologic angiogenesis induced by vascular endothelial
54 ment in the posterior segment of the eye and pathologic angiogenesis into the normally avascular, mat
55 The role of placenta growth factor (PlGF) in pathologic angiogenesis is controversial.
56 he future development of therapy directed at pathologic angiogenesis is dependent upon an understandi
57                Selective effects of CD151 on pathologic angiogenesis make it a potentially useful tar
58 wth factor (VEGF) has been implicated in the pathologic angiogenesis observed in psoriasis and other
59  unique potential in ocular gene therapy for pathologic angiogenesis or tumors.
60     Moreover, as KS may serve as a model for pathologic angiogenesis, our results further provide the
61 a demonstrate a role for the Tie2 pathway in pathologic angiogenesis, suggesting that targeting this
62 inal vasculature, which is quintessential of pathologic angiogenesis that occurs in blinding diseases
63 te that synthetic oligo G is an inhibitor of pathologic angiogenesis that reduces cell-surface levels
64  this signaling pathway in physiological and pathologic angiogenesis, the authors generated a monoclo
65 as an essential regulator of physiologic and pathologic angiogenesis, vascular endothelial growth fac
66 fine the role of the alpha2beta1 integrin in pathologic angiogenesis, we investigated tumor-associate
67 -dependent pathways to synergistically block pathologic angiogenesis, which may lead to a safer and m
68                               PAI-1 inhibits pathologic angiogenesis without adversely affecting norm

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。