1 ligious Order Study, a longitudinal clinical-
pathological study.
2 e epilepsy and explored this through clinico-
pathological study.
3 = 381) longitudinal community-based clinical-
pathological studies.
4 s caps, have been well characterized through
pathological studies.
5 d across comparisons and are consistent with
pathological studies.
6 However,
pathological studies about hyperammonemia have shown tha
7 CC and with a diagnosis of HCC-CC or I-CC by
pathological study;
and control group: 84 patients with
8 ssue culture models, animal models and human
pathological studies are discussed.
9 So far, large
pathological studies comparing axonal damage between pri
10 Bioinformatic analyses and
pathological studies corroborate the clinical relevance
11 However, and in agreement with recent
pathological studies,
degeneration of chronically demyel
12 Pathological study did not demonstrate injury to the rig
13 Pathological studies found severe abnormalities of LMNs
14 Pathological studies from the brains of patients and fro
15 indings, patients were stratified into three
pathological study groups by using the World Health Orga
16 Serological and
pathological studies have associated atherosclerosis wit
17 Few
pathological studies have been carried out on patients w
18 Pathological studies have been successful in distinguish
19 Pathological studies have determined that fibrillar form
20 In multiple sclerosis (MS),
pathological studies have identified substantial demyeli
21 However, numerous surgical and
pathological studies have identified widespread cystic m
22 Recent
pathological studies have re-emphasized that axonal inju
23 However,
pathological studies have shown microdysgenesis in grey
24 Physiological and
pathological studies have shown that ACE inhibitors have
25 Pathological studies have shown that first-generation DE
26 In a separate
pathological study,
histological specimens were obtained
27 Recent
pathological studies implicated proinflammatory cytokine
28 Clinical, imaging, and
pathological studies in multiple sclerosis have generall
29 These clinical data are supported by the
pathological study,
in which higher overall cortical Lew
30 CC and with a diagnosis of HCC-CC or I-CC by
pathological study is limited.
31 Based on epidemiological and
pathological studies,
it is becoming increasingly clear
32 Genetic and
pathological studies link alpha-synuclein to the etiolog
33 ous system (PNS) to direct physiological and
pathological study,
neuropathies have traditionally play
34 We conducted
pathological studies of brain tissues to define the chan
35 here correlate well with epidemiological and
pathological studies of GBS chorioamnionitis.
36 Detailed
pathological studies of healed ruptures, however, are la
37 ropathies cause CRPS-like abnormalities, and
pathological studies of nerves from chronic CRPS-I patie
38 ssociated with human retroviral disease, but
pathological studies of TTP lesions have been unable to
39 ition to this clinical study, we performed a
pathological study of autopsy cases from this same commu
40 The framework was validated by a
pathological study of poplar leaves infected by fungal M
41 siological, functional imaging, and clinical-
pathological studies point to disturbances in frontal co
42 Pathological studies report consistent loss of PAX2 in b
43 Pathological studies reveal a variety of structural abno
44 Pathological studies revealed an encephalitis of the mid
45 Pathological studies revealed moderate cervical lymphade
46 Pathological studies revealed that the heart-specific Rc
47 Pathological studies show severe loss of myelin sparing
48 receptor superfamily members and our recent
pathological studies show that TNFRII is deficient in th
49 These findings support recent
pathological studies showing an ascending pattern of Lew
50 Our combined imaging and
pathological study shows that multi-gradient echo magnet
51 Pathological studies suggest that calcified coronary nod
52 s is not well established; and (ii) although
pathological studies suggest that complement activation
53 However, epidemiological and
pathological studies suggest that excessive mucin secret
54 the possibility of employing human iPSCs in
pathological studies,
therapeutic screening, and autolog
55 A prospective longitudinal clinical-
pathological study was conducted in a tertiary research
56 In this clinico-
pathological study we examined the cervical spinal cord
57 ights the continuing role of careful clinico-
pathological studies which have set the stage for each m