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1 RNA mapped the PTB-binding site to the UCUAA pentanucleotide repeats.
2 ulated by slipped-strand mispairing across a pentanucleotide repeat, ACAGC, within the 5' end of alxA
3 10 has been associated with expansion of the pentanucleotide repeat (ATTCT)(n).(AGAAT)(n) from a norm
4 st variation was detected in the number of a pentanucleotide repeat (CAAAA) that controls production
5 by variation in the number of short-sequence pentanucleotide repeats (CAAAA) located immediately down
7 n contrast to other expandable repeats, this pentanucleotide repeat does not form stable intra- or in
8 d activation required a TATA box sequence, a pentanucleotide repeat immediately upstream of the TATA
10 a indicate that the new SCA10 intronic ATTCT pentanucleotide repeat in SCA10 patients is unstable and
11 Long tracts of CCG trinucleotide or CCGNN pentanucleotide repeats in DNA have previously been show
14 icant allelic association of a CYP11a 5' UTR pentanucleotide repeat polymorphism with hirsute PCOS su
15 sex hormone binding globulin (SHBG) gene, a pentanucleotide-repeat polymorphism [(TAAAA)(n)] and a s
16 instabilities) of repeating tri-, tetra- and pentanucleotide repeating sequences associated with a nu
17 e ability of differing numbers of (CCTTT)(n) pentanucleotide repeats to induce transcription of the N
19 DNAs containing 76 CCG triplets or 48 CCGNN pentanucleotide repeats were 2.0 +/- 0.2-fold and 2.1 +/
20 ontaining 76 tandem CCG triplets or 48 CCGNN pentanucleotide repeats were methylated with SssI methyl
22 zed the allele distribution of a polymorphic pentanucleotide repeat within the 5' upstream promoter r
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