ライフサイエンスコーパス: pouch

1 s and four nest types (plate, cup, dome, and pouch).

2 tion or neutrophil infiltration into the air pouch.

3 such as neutrophil infiltration into the air pouch.

4 rates of cell proliferation within the wing pouch.

5 ty is down regulated in the notum and distal pouch.

6 t-sided structures, called the left coelomic pouch.

7 owed the presence of globular bodies in this pouch.

8 was considered false positive due to Dacron pouch.

9 ination of oral ectoderm that forms Rathke's pouch.

10 l roles in patterning and growth of Rathke's pouch.

11 Gli2 occurs prior to the closure of Rathke' pouch.

12 s and form ectopic foci surrounding Rathke's pouch.

13 le novelty among vertebrates, the male brood pouch.

14 externally located gills, possibly housed in pouches.

15 tion of Wnt11r and Fgf8a rescues tbx1 mutant pouches.

16 ll micromeres to the left and right coelomic pouches.

17 ation of the more anterior arch arteries and pouches.

18 emaining at high levels in the more anterior pouches.

19 e from the endoderm of the caudal pharyngeal pouches.

20 ed into mouse synovial-like subcutaneous air pouches.

21 n and are not incorporated into the coelomic pouches.

22 mp7 and PRDC are expressed in the pharyngeal pouches.

23 nan-induced inflammation in footpads and air pouches.

24 increases junctional E-cadherin in maturing pouches.

25 enerate a late-forming portion of pharyngeal pouch 1 (termed late-p1) and skeletal elements adjacent

26 119 patients (3.2%) required excision of the pouch, 32 (0.8%) had a nonfunctioning pouch, and 46 pati

27 d parathyroids develop from third pharyngeal pouch (3rd pp) endoderm.

28 ndodermal primordium in the third pharyngeal pouch (3rd pp).

29 gastrojejunal (GJ) anastomosis (37), gastric pouch (4), gastric remnant (2), jejuno-jejunostomy (1),

30 In the center of each pouch, a fusion plate forms where cells lose their epith

31 s of the pituitary are derived from Rathke's pouch, a pocket formed by an invagination of the oral ec

32 packs and 40% of RYO (Roll-Your-Own) tobacco pouches, almost exclusively lower priced brands, were di

33 d prematurely differentiated within Rathke's pouch along with correlated ectopic expression of Mash1

34 opy evaluation confirmed the presence of the pouch along with some indication of residual degenerated

35 These pouches also retard the formation of some common off-fla

36 Restorative proctocolectomy with ileal pouch anal anastomosis (IPAA) is associated with tubal f

37 Ileal pouch anal anastomosis (IPAA) is the treatment of choice

38 rmine whether immediate colectomy with ileal pouch-anal anastamosis (IPAA) after diagnosis of severe

39 ), 118 subtotal colectomies (19%), 134 ileal pouch-anal anastomoses (21%), 23 segmental colectomies (

40 le-center experience of transabdominal ileal pouch-anal anastomoses (IPAA) redo surgery for a failed

41 d ileostomy, and 1172 (47.3%) received ileal pouch-anal anastomoses.

42 A prospectively collected ileal pouch-anal anastomosis (IPAA) database was reviewed retr

43 Restorative proctocolectomy with ileal pouch-anal anastomosis (IPAA) has substantially reduced

44 hough restorative proctocolectomy with ileal pouch-anal anastomosis (IPAA) substantially reduces the

45 Pouchitis is common after ileal pouch-anal anastomosis (IPAA) surgery for ulcerative col

46 compare surgical outcome of transanal ileal pouch-anal anastomosis (ta-IPAA) with transabdominal min

47 Total proctocolectomy with ileal pouch-anal anastomosis is considered the procedure of ch

48 Although ileal pouch-anal anastomosis is recommended after colectomy fo

49 f surgery among patients who underwent ileal pouch-anal anastomosis was associated with higher 90-day

50 ients, 71 (65.7%) underwent subsequent ileal pouch-anal anastomosis, 2 died of other causes, and 3 we

51 r a combined total proctocolectomy and ileal pouch-anal anastomosis.

52 an important issue even in the era of ileal pouch-anal anastomosis.

53 s (UC) undergoing proctocolectomy with ileal pouch-anal anastomosis.

54 A total of 3707 patients underwent primary pouch and 328 underwent redo pouch surgery.

55 er of transcription factors expressed in the pouch and by inductive signals from the ventral dienceph

56 amber (France), preserved with its marsupial pouch and content.

57 lish distinct molecular domains in the third pouch and control the subsequent separation of these org

58 eded to maintain progenitors within Rathke's pouch and for the restriction of differentiated cells to

59 ver, total leukocyte infiltration in the air pouch and IL-1beta production were attenuated in Cryo(-Z

60 at RvE1 decreases neutrophil influx into the pouch and increases neutrophil phagocytosis of P. gingiv

61 Prop1 mutant, cells are trapped in Rathke's pouch and N-cadherin expression remains high.

62 pendent rodent models of inflammation in air pouch and peritoneum.

63 full-thickness plications within the gastric pouch and stoma using the StomaphyX device with SerosFus

64 in highly regionalised patterns in the third pouch and that sprouty gene deletion results in upregula

65 However, the shape and organization of the pouch and the anterior/intermediate pituitary lobes are

66 dies have shown that development of Rathke's pouch and the generation of distinct populations of horm

67 wo mouse models of inflammation, zymosan air pouch and thioglycolate-induced peritonitis models, the

68 and 'normal' phosphate (Pi) supply using a 'pouch and wick' system, and had been screened previously

69 luding 11 (0.36%) with adenocarcinoma of the pouch and/or the anal-transitional zone (ATZ), 1 (0.03%)

70 ndow results in morphological defects to the pouches and craniofacial skeleton.

71 f foxY results in a failure to form coelomic pouches and disrupts the expression of virtually all tra

72 ononuclear cell recruitment in vivo into air pouches and injured muscles depends on the heterocomplex

73 r and the Pak2a kinase in the development of pouches and the posterior facial skeleton that depends o

74 of the pouch, 32 (0.8%) had a nonfunctioning pouch, and 46 patients (1.2%) had redo IPAA.

75 randomized left-right asymmetry of coelomic pouches, and disorganized circumesophageal muscle causin

76 ling in patterning the vertebrate pharyngeal pouches, and show that Hh signalling may be involved in

77 e propose that signals intrinsic to Rathke's pouch are necessary for cell specification between e11.5

78 st of the cell divisions in the central wing pouch are oriented along the proximal-distal (P-D) axis

79 The pharyngeal pouches are a segmental series of epithelial structures

80 The endodermal pouches are a series of reiterated structures that segme

81 In sea urchins, the coelomic pouches are the major contributor to the adult, but how

82 el of pouch (AreaM) and the highest level of pouch (AreaH).

83 were measured at the middle height level of pouch (AreaM) and the highest level of pouch (AreaH).

84 or a notochord, cartilaginous arcualia, gill pouches, articulations within the proboscis, and multipl

85 d ex vivo using mice with a subcutaneous air pouch as a model for inflammation.

86 ed diagnosis are worse but half retain their pouch at 10 years with good functional outcomes.

87 neutron diffraction study of a large format pouch battery cell.

88 egulated and cells are able to exit Rathke's pouch but have lost their migrational cues and form ecto

89 Defect-engineered graphene flexible pouch capacitors with energy densities of 500% higher th

90 neoplasia and 0.6%, 1.4%, 2.1%, and 3.3% for pouch carcinoma.

91 ctively in myeloid cells infiltrating an air pouch cavity upon injection of carrageenan failed becaus

92 r contributor to the adult, but how coelomic pouch cells (CPCs) are specified during embryogenesis is

93 gnaling induce the rearrangement of maturing pouch cells into bilayers through junctional localizatio

94 n of beta-catenin in small clusters of canal pouch cells prevents their resorption, causing instead t

95 The performance of 8Ah C6/LiFePO4 pouch cells were measured following periods of calendar

96 Hodgkin lymphomas with golden hamster cheek pouch cells, resulting in serially-transplanted (over 5-

97 Wnt11r-dependent guidance cue for migrating pouch cells.

98 th respectively 4.35 V and 4.4 V UCV in 2 Ah pouch cells.

99 ve gastrectomy (approximately 100 mL gastric pouch), closed duodenal stump, end-to-side duodenoileost

100 ariable analyses among patients in the ileal pouch cohort (odds ratio, 1.38; 95% CI, 1.05-1.82).

101 ns were observed among patients in the ileal pouch cohort who received anti-TNF agents preoperatively

102 CCI, registered during 90 days after pouch construction, was compared between the transanal a

103 nts had a defunctioning ileostomy at time of pouch construction.

104 Nylon/LLDPE pouches containing N2 are the most suitable packaging fo

105 f the DiGeorge syndrome gene TBX1, a lack of pouches correlates with severe craniofacial defects, yet

106 ed subcutaneously within Sernova Corp's Cell Pouch (CP).

107 regate pharyngeal arch artery and pharyngeal pouch defects in RA receptor mutants, and show that RA s

108 es of wnt11r and fgf8a phenocopy tbx1 mutant pouch defects, and mesoderm-specific restoration of Wnt1

109 e conducted a birth dating study of Rathke's pouch derivatives to determine whether the location of s

110 the Hoxa3 null mutant lacks third pharyngeal pouch derivatives, the thymus and parathyroids by E18.5,

111 ved regulatory mechanism that acts to define pouch derivatives.

112 as required for survival of third pharyngeal pouch-derived organs, but expression in either tissue wa

113 craniofacial defects, yet how Tbx1 controls pouch development remains unclear.

114 -specific roles of Hoxa3 in third pharyngeal pouch development, we analyzed tissue-specific mutants u

115 ch-related problems including band slippage, pouch dilation, and hiatal hernia were studied.

116 nclude stomal stenosis, malpositioned bands, pouch dilation, band slippage, perforation, gastric volv

117 unction, hiatal hernia, wound infection, and pouch dilation.

118 fic bacterial populations correlate with the pouch disease phenotype and inflammatory activity.

119 for assessment of polyp burden in the rectum/pouch, duodenal polyposis and pre-colectomy groups, resp

120 ropagates from the center to the edge of the pouch during the third larval instar.

121 ncer of colon or rectum is a risk factor for pouch dysplasia or adenocarcinoma.

122 SHH pathway activation throughout pouch endoderm activated ectopic Tbx1 expression and par

123 SHH signaling is active in both dorsal pouch endoderm and neighboring neural crest (NC) mesench

124 ial body epithelium emerging from pharyngeal pouch endoderm in early organogenesis, differential Foxa

125 icate NCCs in regulating patterning of third pouch endoderm into thymus- versus parathyroid-specified

126 oderm, core paraxial mesoderm and pharyngeal pouch endoderm of the mandibular arch as well as more ca

127 is among the prominent markers of pharyngeal pouch endoderm, but to date no role has been assigned to

128 in upregulated FGF signalling throughout the pouch endoderm.

129 glands are derived from the third pharyngeal pouch endoderm.

130 phB-Pak2a signaling is required to stabilize pouch epithelial cells at the end of branching morphogen

131 ver, an unexplored possibility is that brood pouch evolution was partly shaped by parent-offspring or

132 with good functional results, and may avoid pouch excision and permanent ileostomy in carefully sele

133 sensitivities were analysed, and changes in pouch faecal and mucosal microbiota assessed by 16S rRNA

134 l pouch surgery was the primary indicator of pouch failure (hazard ratio, 3.691; 95% confidence inter

135 se patients who had IPAA at our institution, pouch failure occurred in 197 patients (5.3%).

136 rences in fistula or abscess formation or in pouch failure.

137 s was a significant risk factor for ultimate pouch failure.

138 rmal recruitment of leukocytes to murine air pouches filled with unmodified CCL2.

139 the recruitment of leukocytes to murine air pouches filled with wild-type CXCL12.

140 imen, control CT and puncture of the Douglas pouch fluid was made.

141 atrices were randomly placed in subcutaneous pouches for 7 and 28 days and evaluated by histology and

142 he posterior facial skeleton that depends on pouches for its segmentation.

143 head, endodermal branches, called pharyngeal pouches, form through the transient stratification, coll

144 Studies evaluating ileal pouch formation and antireflux surgery showed conflictin

145 rly phase during which FGFs drive pharyngeal pouch formation, and a later phase when they directly re

146 ely to be secondary to defects in pharyngeal pouch formation.

147 Wnt-dependent epithelial destabilization of pouch-forming cells with their collective migration towa

148 pithelium to promote the lateral movement of pouch-forming cells.

149 the mesoderm to promote the morphogenesis of pouch-forming endoderm through wnt11r and fgf8a expressi

150 he proportion of patients with a functioning pouch, frequency of defecation and incidence of incontin

151 d of clinical examination with assessment of pouch function and QOL.

152 The clinical significance of these cementum pouches has yet to be determined but bacterial contamina

153 is and control the relative expansion of the pouch, hinge, and notum.

154 0.069) and an increased rate of giant venous pouch in children in whom no mutation was identified (p

155 tion of the small micromeres to the coelomic pouches in the sea urchin embryo provides an exceptional

156 distinct roles in the segmental formation of pouches in zebrafish.

157 nd timing of key regional markers within the pouch, including Tbx1, Bmp4 and Fgf8, were altered.

158 not restore leukocyte recruitment to the air pouch, indicating a role for endothelial CD47.

159 in inflammatory bacteria are associated with pouch inflammation in patients with UC who underwent pou

160 to groups based on their degree and type of pouch inflammation.

161 However, cells at the periphery of the wing pouch instead tend to orient their divisions perpendicul

162 hat endodermal Eph-ephrin signaling promotes pouch integrity by targeting Pak2a to the plasma membran

163 Rathke's pouch is formed and endocrine cell lineages are generate

164 lar to the Hesx1-deficient embryos, Rathke's pouch is initially expanded in Six3+/- ;Hesx1Cre/+ compo

165 s are restricted to developing in the caudal pouches is unclear.

166 c bypass are radiologically contained GJ and pouch leaks and can be safely managed nonoperatively.

167 M evaluation showed the presence of a narrow pouch-like opening between cementum and enamel in 15 of

168 This study reports on the presence of a pouch-like opening between the enamel and cementum in ma

169 ermined but bacterial contamination of these pouch-like structures in areas of furcation periodontal

170 On multivariate analysis, pouch loss was associated with delayed diagnosis (P = 0.

171 ed glass and two commercial bag-in-box (BIB) pouches (low density polyethylene - LDPE and ethylene vi

172 monocyte and neutrophil accumulation in the pouch lumen as well as in the wall.

173 proliferating cells are enriched around the pouch lumen, and they appear to delaminate as they exit

174 lipopolysaccharide (LPS) or OxPAPC into the pouch lumen.

175 anal-transitional zone (ATZ), 1 (0.03%) with pouch lymphoma, 3 with squamous cell cancer of the ATZ,

176 Patients with a failed ileoanal pouch may be offered redo pouch surgery with a high like

177 dance of species suggestive of a "healthier" pouch microbiota.

178 t mice were subjected to the carrageenan-air pouch model of inflammation and then treated with D-T(4)

179 om M(r) 5,000 to 1,500,000 in the rodent air pouch model of inflammation to determine their potential

180 In an air pouch model of inflammation, CCL20 triggered recruitment

181 on in RAW264.7 cells and also in a mouse air-pouch model of inflammation.

182 nse to tumor necrosis factor-alpha in an air-pouch model of leukocyte migration.

183 nz[a]anthracene (DMBA)-induced hamster cheek pouch model of oral squamous cell carcinoma (SCC).

184 induced neutrophilic inflammation in the air-pouch model of synovitis, and they show decreased joint

185 as neutrophils and monocytes in a dermal air pouch model of TNF-alpha-induced inflammation.

186 A humanized mouse air-pouch model showed that intravenous treatment with the C

187 The mouse dorsal air pouch model was used to evaluate the in vivo impact of R

188 ites have inflammatory properties in the air pouch model, and rHuPH20 can instead inhibit some aspect

189 First, in a murine air-pouch model, apoptotic cell supernatants induced a three

190 In an air-pouch model, CS-induced neutrophil recruitment is depend

191 cells (BMSCs) to form bone in a mouse muscle pouch model, exhibiting specificity that BMPs lack.

192 Utilizing the chimeric murine air pouch model, MK2 signaling differentially regulated CCL3

193 Using a dorsal air-pouch model, passive transfer of apoptotic neutrophils b

194 esponse to delivery of CXCL1 through the air pouch model.

195 ase of AnxA1 and CRAMP in a subcutaneous air pouch model.

196 simile synovium in vivo using the murine air-pouch model.

197 during local inflammation in the murine air pouch model.

198 nium particle-induced inflammation in an air pouch model.

199 d signal, Hedgehog, is required for coelomic pouch morphogenesis and institution of laterality, but d

200 However, the role of Bmp signaling in pouch morphogenesis is poorly understood.

201 chemical inhibitor approaches, we show that pouch morphogenesis requires Bmp signaling from 10-18 h

202 n the pharyngeal arches, aberrant endodermal pouch morphogenesis, and hypoplastic cranial cartilages.

203 t in jawed vertebrates, including pharyngeal pouch morphogenesis, patterning of the oral skeleton and

204 right midabdomen (n = 2), under the gastric pouch (n = 1), and right lower quadrant (n = 1).

205 20 years were 1.0%, 2.0%, 3.7%, and 6.9% for pouch neoplasia and 0.6%, 1.4%, 2.1%, and 3.3% for pouch

206 We calculated the cumulative incidence of pouch neoplasia and performed a case-control study to id

207 Cumulative incidences for pouch neoplasia at 5, 10, 15, 20, and 25 years were 0.9%

208 inflammatory bowel disease (IBD), subsequent pouch neoplasia can develop.

209 med to determine the cumulative incidence of pouch neoplasia in patients with IBD and identify risk f

210 The incidence of pouch neoplasia in patients with IBD without a history o

211 ized features, risk factors, and outcomes of pouch neoplasia in patients with inflammatory bowel dise

212 e Cox model, the risk factor associated with pouch neoplasia was a preoperative diagnosis of UC-assoc

213 Thirty-eight patients (1.19%) had pouch neoplasia, including 11 (0.36%) with adenocarcinom

214 ents with IBD and IPAA; 25 (1.83%) developed pouch neoplasia, including 16 adenocarcinomas.

215 IBD and identify risk factors for developing pouch neoplasia.

216 sia was the only risk factor associated with pouch neoplasia.

217 ncrease in risk, respectively, of developing pouch neoplasia.

218 Mucosectomy did not protect against pouch neoplasia.

219 biliopancreatic limb of 50 cm and a gastric pouch of about 25 mL.

220 hat absence of SOX2 in the developing Rathke pouch of conditional embryos led to severe anterior lobe

221 al disease sites (P = .0025) and presence of pouch of Douglas implants (P = .0045) were associated wi

222 Hence, the male brood pouch of syngnathid fishes, which nurtures offspring, al

223 factors asymmetrically in the left coelomic pouch only, which launch the pathway that eventually lea

224 m compliance at the serving-size level (of a pouch or approximately 40-50g).

225 of head and neck cancer in the hamster cheek pouch oral cancer model is presented.

226 filtered cigars, pipe tobacco, hookah, snus pouches, other smokeless tobacco, dissolvable tobacco, b

227 ics show that Bmp signaling is necessary for pouch outpocketing via the Fibroblast growth factor (Fgf

228 ukocyte recruitment when added to murine air pouches (p < 0.05).

229 A total of 27 pouch patients with inflammatory bowel diseases, who und

230 e epithelial integrity of the vertical canal pouch perimeter (presumptive anterior and posterior SSCs

231 gest time to first FAP-related event [rectal/pouch polyposis]), intermediate (duodenal polyposis) and

232 orable pouch voiding mechanics, insufficient pouch pressure generation or failure of external sphinct

233 s originates from bilateral third pharyngeal pouch primordia containing endodermal progenitors of bot

234 rder shift increases the allocation of third pouch progenitors to the thymus domain and corresponding

235 duction and subsequent expansion of Rathke's pouch progenitors.

236 , a large amount of fluid within the Douglas pouch raised the oncological concern.

237 ients with failing IPAA, partial or complete pouch reconstruction can be done safely with good functi

238 (UC) who required partial or complete ileal pouch reconstruction due to poor function or infectious

239 Reoperation for pouch-related problems after LAGB is safe and effective.

240 Patients who required revision for pouch-related problems including band slippage, pouch di

241 ocalization of Dachs in cells throughout the pouch requires the movement of the Ds transition region

242 6th arch arteries and to the 4th pharyngeal pouch, respectively, suggesting that different regulator

243 Time-to-diagnosis and pouch retention rates were analyzed using Kaplan-Meier c

244 Overall 10-year pouch retention was 71%.

245 ical, and pathologic factors associated with pouch retention were evaluated with log-rank test and Co

246 % of patients whereas 59% had their original pouch revised and retained.

247 Pouch revision was partial in 57% of patients and comple

248 required to specify LHX3(+)/LHX4(+) Rathke's pouch (RP) progenitor identity.

249 enous RvD4 from resolving exudates in dorsal pouch S. aureus infections.

250 e RA signaling occur secondary to pharyngeal pouch segmentation defects, although this model has neve

251 ized, but total proctocolectomy and ileoanal pouch should be strongly considered.

252 Between the two plastics, the LDPE lined pouch showed a considerably higher aroma sorption as com

253 e RYGB model that closely replicates gastric pouch size of human RYGB surgery of about 5% of total ga

254 In vivo dorsal air pouch studies revealed that RvE1 decreases neutrophil in

255 tive study of patients with UC who underwent pouch surgery (N = 131) from 1981 through 2012 and were

256 ts with familial adenomatous polyposis after pouch surgery (n = 9), individuals with intact colons un

257 The relation between laparoscopic pouch surgery and fertility, however, has not been studi

258 Pelvic sepsis developing after redo ileal pouch surgery was the primary indicator of pouch failure

259 a failed ileoanal pouch may be offered redo pouch surgery with a high likelihood of success in terms

260 derwent primary pouch and 328 underwent redo pouch surgery.

261 my and do not choose to undergo later pelvic pouch surgery.

262 flammation in patients with UC who underwent pouch surgery.

263 ve protein in patients with UC who underwent pouch surgery.

264 ia, so there is no consensus on the need for pouch surveillance.

265 The probability of pouch survival after reconstruction was 93% at 1 year an

266 ADAM17-mediated shedding of IL-6R within the pouch that orchestrates this inflammatory process.

267 ng development, the canals are sculpted from pouches that protrude from the otic vesicle, the embryon

268 Of the pouches that subsequently failed, 75% occurred in patien

269 Diverticular disease is characterized by pouches (that is, diverticulae) due to weakness in the b

270 development of the infundibulum and Rathke's pouch - the precursor of the anterior pituitary.

271 along the proximodistal axis into the distal pouch, the hinge, the surrounding pleura, and the notum.

272 Otx2 expression was intact in Rathke's pouch, the precursor to the anterior lobe, but the anter

273 t Eya1 expression to the somites, pharyngeal pouches, the preplacodal ectoderm (the common precursor

274 osing epithelial walls in the center of each pouch, thereby creating a hollow canal.

275 hanges in pregnant versus non-pregnant brood pouch tissue and characterize the genomic organization o

276 contained 11 +/- 6 ppm, and the tumor/normal pouch tissue boron concentration ratio was 10:1.

277 microcirculation as an in situ assay, cheek pouch tissue was exteriorized in anesthetized (phentobar

278 on in cells as they transition from Rathke's pouch to the anterior lobe appears to be essential for t

279 by a thicker carbon layer in micrometre-size pouches to act as an electrolyte barrier.

280 mesodermal derivatives such as the coelomic pouches to which the small micromeres contribute.

281 R) inhibited plasma leakage in hamster cheek pouch topically exposed to tissue culture trypomastigote

282 2.6 (95% confidence interval [CI] 1.1-6.5)), pouch-vaginal fistula (P = 0.01, HR 2.8 (95% CI 1.3-6.4)

283 la type by gender: females (rectovaginal and pouch-vaginal) and males (rectourethral).

284 ted from dysfunctional voiding - unfavorable pouch voiding mechanics, insufficient pouch pressure gen

285 are not comparable to human RYGB in gastric pouch volume for a large or absent gastric volume.

286 re quantified in the lavage fluid and in the pouch wall after staining with Giemsa or after enzymatic

287 uitment of monocyte/macrophages into the air-pouch wall, but not in the lumen.

288 A new pouch was created in 41% of patients whereas 59% had the

289 Initial patterning in the E10.5 pouch was normal suggesting that the observed change in

290 In vivo, Th17 cell recruitment into the air pouch was reduced in CD43(-/-) mice in response to CCL20

291 Absence of colonic pouch was the only independent factor of pelvic sepsis (

292 duced epithelial tumors in the hamster cheek pouch were treated.

293 Air pouches were raised by 2 injections of sterile air, and

294 d ectodermal components, as well as Rathke's pouch, were similarly sampled and profiled using both mi

295 ed or highly reduced in the posterior/caudal pouches, where the parathyroid marker Gcm2 is expressed,

296 o early gene expression defects in the third pouch, whereas thymus hypoplasia is caused by reduced pr

297 males brood their offspring in a specialized pouch, which presumably evolved to facilitate male paren

298 induced neutrophil infiltration into the air pouch, which was not observed with rHuPH20 treatment.

299 The pharyngeal pouches, which form by budding of the foregut endoderm,

300 roughly uniform throughout most of the wing pouch with a steep transition region that propagates fro

301 Challenging the air pouch with LPS or BTH stimulated production of cytokines