ライフサイエンスコーパス: protection of @1 from

1 to the skin induced a robust time-dependent protection of mice from a lethal Klebsiella pneumoniae p

2 Complete protection of cells from acute apoptosis and prolongatio

3 s have been confirmed to be potent in actual protection of mice from acute toxicity (convulsion and l

4 ole above N7 of the purine, which results in protection of N7 from alkylating agents.

5 these studied surfactant systems, suggesting protection of citral from an acidic environment once it

6 ac as measured by T cell stimulation and the protection of mice from an infectious pathogen.

7 TAT3 as well as in insulin- or IGF-1-induced protection of cells from anoikis.

8 nd Cyp2e1 mRNAs, reduced JNK activation, and protection of mice from APAP-induced hepatotoxicity.

9 ated by this pathway plays a key role in the protection of cells from apoptosis and the mediation of

10 ogical function for the CDK inhibitor p27 is protection of cells from apoptosis by constraining CDK2

11 of the PKB/Akt activation is sufficient for protection of cells from apoptosis induced by the deplet

12 gen-regulated protein kinase involved in the protection of cells from apoptosis, the promotion of cel

13 hematopoiesis is partly attributable to the protection of cells from apoptosis.

14 appaB (NF-kappaB) has been implicated in the protection of cells from apoptosis.

15 these proteins together provides significant protection of cells from apoptosis.

16 the ILK-alpha-parvin complex is crucial for protection of cells from apoptosis.

17 nstream caspase-3 cleavage and near-complete protection of cells from apoptotic death.

18 r overexpression of p89 c-Myb results in the protection of cells from apoptotic death.

19 Better protection of individuals from asbestos-related health e

20 g that pigments are essential not simply for protection of spores from biotic and abiotic stresses bu

21 onist resulted in a dose- and time-dependent protection of cartilage from blood-induced damage.

22 high degree (in one case 100%) post-exposure protection of mice from both viruses.

23 lts point to a unique function for desmin in protection of mitochondria from calcium exposure that ca

24 ct evidence that perinatal IgG transport and protection of IgG from catabolism are mediated by FcRn,

25 We conclude that FcRn-mediated protection of IgG from catabolism is a generic mechanism

26 timulation of cell survival is mainly due to protection of cells from cell death rather than by stimu

27 pensable for B7-DC cross-linking Ab-mediated protection of DC from cell death caused by cytokine with

28 electivity of LC migration from the skin and protection of LCs from cell death are considered.

29 prior to excitotoxic insult resulted in the protection of neurons from cell death.

30 e, in agreement with preliminary data on the protection of lymphocytes from chemotoxicity in fasting

31 1 in hyperglycemia appeared to be due to the protection of IAP from cleavage that occurred during exp

32 times with cDNA encoding VP2 led to partial protection of mice from CNS demyelinating disease as det

33 may represent an important mechanism for the protection of EC from complement-mediated injury during

34 lectrochemical water oxidation and effective protection of Si from corrosion at high pH (pH 13.6).

35 Protection of neurons from cytotoxic effects of activate

36 Our data reveal a mechanism of efficient protection of telomeres from damage through Nek7-depende

37 nhibitor, survivin, in endothelial cells and protection of endothelium from death-inducing stimuli.

38 tramuscular (i.m.) vaccination provided full protection of hamsters from death and diarrhea while the

39 -3511 (26) displayed concentration-dependent protection of neurons from degeneration in vitro and dem

40 LK in retina results in robust and sustained protection of RGCs from degeneration after optic nerve i

41 TTP-14-3-3 complex formation appeared to be protection of TTP from dephosphorylation by inhibition o

42 howed that HBsu altered alpha/beta-type SASP protection of pUC19 from DNase digestion, induced negati

43 preservation of mitochondrial networking and protection of cardiomyocytes from doxorubicin-mediated c

44 o of 20:1 (N/P approximately 27.3), complete protection of siRNA from early enzymatic degradation was

45 findings may have potential applications in protection of buildings from earthquakes and isolating s

46 ine and human cell lines and induces in vivo protection of mice from encephalomyocarditis virus infec

47 ng prokaryotic mRNA decay theories: ribosome protection of mRNA from endonucleases, 5' binding and sl

48 e placement, usually measured by quantifying protection of DNA from enzymatic digestion, can regulate

49 he maintenance of immunological homeostasis, protection of lymphocytes from excessive cell death is e

50 Significant protection of amides from exchange with D2O buffer in me

51 n, and microtubules at costameres as well as protection of muscle from exercise-induced injury.

52 on of their proliferation cannot account for protection of spermatogenesis from exposure to cytotoxic

53 Disease control measures emphasized protection of workers from exposure, use of containment

54 nmentally transmitted and to function in the protection of eggs from fouling and infection.

55 es; action of matrix metalloproteinases; and protection of tissue from free radical damage.

56 all model dehydrin (Vitis riparia K2) on the protection of membranes from freeze-thaw stress.

57 ll as for production and storage of food and protection of crops from frost.

58 questered in separate sites by PgDps provide protection of DNA from H(2)O(2)-mediated free radical da

59 ly high levels of H(2)O(2), resulting in the protection of PTPs from H(2)O(2)-mediated oxidation.

60 ponsiveness of macrophages that leads to the protection of mice from hepatic injury and death caused

61 A single dose of vaccine conferred complete protection of ferrets from homologous wt A/Anhui/1/2013

62 ted T cells do not play an important role in protection of mice from HSV, an important role for these

63 rticular, pig CD59 play an important role in protection of PAEC from HuC.

64 Protection of women from human rights abuses, prevention

65 that may explain the experimentally observed protection of amides from hydrogen exchange and the exis

66 bstrate concentrations, suggesting substrate protection of P450 from hydroperoxide inactivation.

67 In thrombin-induced protection of astrocytes from hypoglycemia, pretreatment

68 We tested whether peritransplant protection of islets from IBMIR with a monoclonal anti-T

69 a definite role for FLIP in the SCF-induced protection of ECFCs from IFNgamma-initiated apoptosis.

70 balamin indicates that MeaB is necessary for protection of mutase from inactivation during catalysis.

71 rheumatoid arthritis patients, and enhanced protection of mice from inflammatory colitis.

72 ur study reveals a mechanism of DNA-mediated protection of p53 from interactions with partners involv

73 erase micro gene in 32Dcl3 cells resulted in protection of cells from interleukin-3 withdrawal-induce

74 ed exocytosis plays an important role in the protection of kidneys from ischemia-reperfusion injury.

75 rc tyrosine kinase family/ERK signaling, and protection of neurons from ischemic injury and cell deat

76 in human umbilical cord blood cell-mediated protection of oligodendrocytes from ischemic conditions.

77 cine and both provided complete, single-dose protection of macaques from lethal challenge with the Ma

78 indicate that while IL-6 plays a role in the protection of mice from lethal HSV infection, it does no

79 g the classical pathway of complement in the protection of mice from lethal infection with virulent S

80 In this study, we report the successful protection of mice from lethal MCMV infection with gene-

81 A primary role for chaperones appears to be protection of effectors from Lon-associated degradation

82 n of AM in preventing metastasis, leading to protection of mice from lung metastases.

83 d that the exosporium may play a role in the protection of spores from macrophages.

84 The protection of p53 from MDM2 by PTEN and the damage-induc

85 tious conditions, which may be important for protection of insects from microbial infection during mo

86 evant in vivo target of miR-21 and show that protection of pdcd4b from miR-21 binding results in fail

87 enervation also prevented ultrasound-induced protection of kidneys from moderate (26-minute ischemia)

88 hod determines Cu binding sites based on the protection of His from modification by diethyl pyrocarbo

89 -specific B-cell responses are essential for protection of macaques from monkeypox virus, a variola v

90 Several studies have implicated TRF2 in the protection of telomeres from NHEJ, but the underlying me

91 ammals, thus providing passive immunity, and protection of IgG from normal serum protein catabolism.

92 ial determinant of high-level, MRP-dependent protection of cells from NQO toxicity.

93 ns of drug transporters may involve both the protection of bacteria from outside toxins and the trans

94 ormation through mechanisms that may include protection of LDL from oxidation and suppression of vasc

95 he benefits of olive oil polyphenols for the protection of LDL from oxidation.

96 phism in the NQO1 gene, which is involved in protection of cells from oxidative damage.

97 s and antioxidant proteins, resulting in the protection of cells from oxidative insults.

98 e1 is a multifunctional protein operating in protection of cells from oxidative stress via its DNA re

99 lothioneins (MT) have been implicated in the protection of cells from oxidative stress.

100 l lines of evidence have supported a role in protection of cells from oxidative stress.

101 ole in the generation of glutathione and the protection of cells from oxidative stress.

102 stained PI3K inhibition leads to significant protection of neurons from oxidative stress-induced neur

103 substituted by respiratory O2 uptake in the protection of nitrogenase from oxidative damage and, thu

104 Expression of BDNF also caused strong protection of photoreceptors from oxidative damage-induc

105 ons but they are unable to provide wild-type protection of nitrogenase from oxygen, so they cannot by

106 Although immune responses may provide good protection of plants from pathogen attack, excessive imm

107 dus SSP in pollen-stigma interactions and in protection of stigmas from pathogen attack.

108 len-stigma interactions, or alternatively in protection of stigmas from pathogen attack.

109 en-specific receptor can result in effective protection of hosts from pathogens.

110 Thus, our data suggest that the protection of DC from PCa-induced apoptosis might signif

111 upport further study of these strategies for protection of newborns from pertussis.

112 ation of mice is likely due to CD47-mediated protection of exosomes from phagocytosis by monocytes an

113 cofilin action appears to be a result of the protection of phosphocofilin from phosphatase-mediated d

114 teolysis and whether p26 is required for the protection of eIF2alpha from phosphorylation, we have an

115 tion of excess absorbed light energy and the protection of plants from photo-oxidative damage.

116 vides a molecular mechanism for the observed protection of Tibetans from polycythemia at high altitud

117 eterminants are required for Alpha4-mediated protection of PP2Ac from polyubiquitination and degradat

118 einylglycine, GSH) is essential for adequate protection of pneumocytes from potential toxicity mediat

119 ent intracellular delivery of siRNAs and the protection of siRNAs from premature degradation before r

120 PML mediates this function through protection of CIITA from proteasomal degradation.

121 and pentapeptide binding of CheB fragments, protection of CheB from proteolysis by pentapeptide, and

122 nt consequence of this binding appears to be protection of IP3Rs from proteolysis.

123 on, up-regulation of p21(CIP1), and complete protection of cells from rapamycin-induced apoptosis.

124 ancy of exosite 1 partially accounts for the protection of thrombin from rapid inactivation by PAI-1

125 the scavenging of the toxic heavy metals and protection of cells from reactive oxygen intermediates.

126 single dose of VLPs resulted in the complete protection of mice from RSV replication in lungs.

127 of these particles resulted in the complete protection of mice from RSV replication in the lungs.

128 for the maintenance of genome stability and protection of humans from several types of cancer.

129 effective as the CSF1R/IR in mediating CSF-1 protection of cells from staurosporine-induced apoptosis

130 Since protection of cells from stress-induced apoptosis by the

131 ve been considered as prebiotics and for the protection of humans from Stx.

132 of heat shock proteins (hsps) results in the protection of cells from subsequent stresses.

133 sion analysis suggested pathways involved in protection of males from T1D by microbiota.

134 expense of host-cell growth rate; and (iii) protection of cells from the toxic effects of overexpres

135 This resulted in the protection of cells from the toxicity of l-Abeta42 at co

136 nding phosphonic acids (EC50 = 50-343 nM) in protection of cells from the viral infection.

137 nhibition of host gene expression and to the protection of EEEV from the antiviral effects of IFNs.

138 tivity, alphaA-crystallin may be involved in protection of hepatocytes from the toxic effects of high

139 In vitro, post-challenge protection of macrophages from the action of the holotox

140 ency), determining the efficacy in practical protection of mice from the acute cocaine toxicity and f

141 al role that trypomastigote CRP plays in the protection of parasites from the deleterious effects of

142 Optimal protection of patients from the harms associated with HA

143 t the telomeric overhang is required for the protection of telomeres from the DNA damage response.

144 cytokines and plays an important role in the protection of tissues from the damaging consequences of

145 some provides significant, but not complete, protection of trypanosomes from the dangerous design of

146 Protein aggregation is irreversible, and protection of proteins from thermal aggregation is a str

147 he template-primer has a critical bearing on protection of RT from thermal inactivation.

148 s are essential for NF-kappaB activation and protection of cells from TNF-alpha-induced apoptosis.

149 ings provide a mechanistic rationale for the protection of hepatocytes from TNF-induced cell death by

150 ssion resulted in a consistent, but partial, protection of cells from undergoing IR-induced apoptosis

151 tion of Nrf2 is a promising strategy for the protection of tissues from various types of insults and

152 tions of today's understanding of CPs in the protection of crops from viral infection and use in the

153 , induction of strong adaptive immunity, and protection of mice from wild-type (WT) WNV infection.