ライフサイエンスコーパス: rCBF

1 rCBF changes were assessed with a three-dimensional voxe

2 rCBF changes were evaluated with statistical parametric

3 rCBF data were recorded with a flow-sensitive alternatin

4 rCBF decreases in limbic/paralimbic areas, temporal and

5 rCBF was measured in 38 control subjects, 29 MCI patient

6 i) declined from 6.95+/-0.04 to 6.60+/-0.05, rCBF declined from 48+/-7 to 10+/-3 ml/100 g/min, and NA

7 to conditionally express recombinant CBF-A (rCBF-A) activate the transcription of FSP1 and undergo E

8 e scanning interval, most of the accelerated rCBF changes seen in the subsequently impaired group occ

9 In addition, rCBF measurement indicated that alcohol-fed rats had les

10 lyses revealed regional abnormalities in all rCBF and rCMR measurements that were consistent in locat

11 The main effects in healthy subjects, an rCBF increase in subgenual cingulate Brodmann's area 25

12 epression Rating Scale were administered and rCBF was measured by means of single photon emission com

13 nverse relationship between 11C-PIB BPND and rCBF MR imaging in the voxel-based analysis that was abs

14 emory experiment, both FCS-rCBF coupling and rCBF/FCS ratio were modulated by task load in the ECN an

15 positive association of between craving and rCBF in the left orbitofrontal cortex.

16 Finally, task-induced changes of FCS and rCBF in the lateral-parietal lobe positively correlated

17 n functional connectivity strength (FCS) and rCBF during resting and an N-back working-memory task.

18 llected at each of these glycemic levels and rCBF measurements within the previously described networ

19 s between selective motor manifestations and rCBF in specific regions suggest possible regional selec

20 d for in-depth evaluation of amyloid PET and rCBF data.

21 ed with significant emotional reactivity and rCBF reductions in the ventromedial prefrontal cortex, s

22 Associations between extraversion scores and rCBF in each olfactory stimulus condition were assessed

23 to movement were relatively unaffected, and rCBF did not change in cerebellum or visual cortex.

24 cipital perfusion deficits but with anterior rCBF deficits in a pattern often described in the litera

25 We assessed rCBF during baseline, rectal distention, and anticipatio

26 en-group differences in correlations between rCBF and response time.

27 ent in the frontal and temporal regions, but rCBF changes in men with ADHD were more widespread and p

28 in regional synaptic activity as indexed by rCBF.

29 cortex, and a larger increase in cerebellar rCBF.

30 was calculated as a percentage of cerebellar rCBF, analysis of covariance found decreases in HD cauda

31 For each hormone condition, rCBF was measured with [15O]H2O PET, and BOLD signal was

32 In this region, a consistent rCBF response to stimulation was observed across subject

33 Conversely, rCBF in the cerebellum increased.

34 the Gambling Task, but orbitofrontal cortex rCBF did not.

35 ated with lower resting orbitofrontal cortex rCBF in cocaine-dependent subjects.

36 cingulate and dorsolateral prefrontal cortex rCBF is significantly related to decision making, as ass

37 late and left dorsolateral prefrontal cortex rCBF significantly correlated with performance on the Ga

38 Left dorsolateral prefrontal cortex rCBF was lower in the cocaine-dependent subjects than in

39 Cortical rCBF was extracted using a 3-dimensional stereotactic su

40 These data confirm limbic cortical rCBF changes associated with effective antidepressant tr

41 Decreased rCBF to the frontotemporal region characterized the cere

42 Amphetamine decreased rCBF to motor cortex, visual cortex, fusiform gyrus, pos

43 CBF in the left frontal region and decreased rCBF in the left amygdala.

44 BF bilaterally in the thalamus and decreased rCBF in the left occipital lobe, right cerebellum, and t

45 teral thalami, right midbrain, and decreased rCBF in the right premotor cortex (P < 0.05, corrected).

46 etween improvement of rigidity and decreased rCBF in the SMA (r(s) = -0.4, P < 0.02) and between impr

47 MCI and AD patients had decreased rCBF in the posterior cingulate gyrus (P = .01) with ext

48 AD patients had decreased rCBF relative to that in control subjects and MCI patien

49 postural reflexes correlated with decreased rCBF in the PPN (r(s) = -0.38, P < 0.03).

50 icance, but a trend for a pronounced delayed rCBF rise was seen for surface seizures (p = 0.08).

51 r model analyses were performed to determine rCBF differences between OA and control groups, rCBF dif

52 ups of children with significantly different rCBF behavior were identified.

53 ndritic arborization, we complimented direct rCBF comparisons with connectivity analyses to determine

54 hip was connection-distance dependent; i.e., rCBF correlated stronger with long-range hubs than short

55 pinal neurons in RVLM to reflexively elevate rCBF and slow the EEG as part of the oxygen-conserving (

56 s relay signals from the MCVA, which elevate rCBF in response to hypoxia, and (3) the SVA is a functi

57 jection of NaCN (300 pmol in 20 nl) elevated rCBF (17 +/- 5 %) and synchronized the EEG from RVLM, bu

58 rats electrical stimulation of RVLM elevated rCBF (laser-Doppler flowmetry) by 31 +/- 6 %, reduced ce

59 rs, ultra-high-risk subjects showed elevated rCBF in the hippocampus, basal ganglia, and midbrain.

60 nchronizes the EEG and coordinately elevates rCBF; (b) the responses are mediated by excitation of ne

61 ly restricted region of subthalamus elevates rCBF, (2) these neurons relay signals from the MCVA, whi

62 he hypothesis that whisker movement elicited rCBF changes are input frequency dependent and are most

63 wise, whole-brain analysis revealed enhanced rCBF only in the left posterior hippocampus (pHp) in the

64 basal forebrain failed to modify SVA-evoked rCBF increase.

65 e carpometacarpal (CMC) joint and to examine rCBF variability across sessions.

66 The authors examined rCBF in 17 healthy volunteers and 18 schizophrenia patie

67 The PTSD group exhibited rCBF decreases in medial frontal gyrus in the traumatic

68 Only MCVs exhibited rCBF increases in the left amygdala.

69 ring the working-memory experiment, both FCS-rCBF coupling and rCBF/FCS ratio were modulated by task

70 ative cerebral blood volume (rCBV) and flow (rCBF) maps were acquired before treatment and after 1 an

71 nd measures of regional cerebral blood flow (rCBF) (a marker of neuronal activity) to describe the fu

72 elevations of regional cerebral blood flow (rCBF) and synchronization of the electroencephalogram (E

73 Regional cerebral blood flow (rCBF) and volume (rCBV) were measured with gadolinium-ba

74 Using regional cerebral blood flow (rCBF) as a marker of basal neuronal activity, this study

75 erformed using regional cerebral blood flow (rCBF) as a marker of neuronal activity.

76 s, we measured regional cerebral blood flow (rCBF) as a marker of synaptic activity at rest and durin

77 ; P<0.01), and elevates cerebral blood flow (rCBF) by 18+/-5% (P<0.05).

78 dependent from regional cerebral blood flow (rCBF) changes during moderate focal cerebral ischemia.

79 f longitudinal regional cerebral blood flow (rCBF) changes in the years preceding measurement of amyl

80 was to compare regional cerebral blood flow (rCBF) changes related to working memory in adults with a

81 n to determine regional cerebral blood flow (rCBF) changes representing ongoing pain experienced by p

82 The regional cerebral blood flow (rCBF) changes that occur during cortical spreading depre

83 ps resulted in regional cerebral blood flow (rCBF) decreases in medial orbitofrontal cortex Brodmann'

84 ith concurrent regional cerebral blood flow (rCBF) determination before and after administration of b

85 Regional cerebral blood flow (rCBF) determinations by positron emission tomography and

86 ygenation, and regional cerebral blood flow (rCBF) did not reach significance, but a trend for a pron

87 if changes in regional cerebral blood flow (rCBF) during hypoglycemia relative to euglycemia are sim

88 resting state regional cerebral blood flow (rCBF) during normal aging and investigated its influence

89 thors measured regional cerebral blood flow (rCBF) during performance of a task that required unmedic

90 PET to assess regional cerebral blood flow (rCBF) during rest and tested for between-genotype differ

91 ant changes in regional cerebral blood flow (rCBF) during states of cardiovascular arousal common to

92 hange in tumor relative cerebral blood flow (rCBF) from baseline and area under the plasma concentrat

93 sed to examine regional cerebral blood flow (rCBF) in 30 patients with chronic schizophrenia and 30 n

94 ges in resting regional cerebral blood flow (rCBF) in 32 healthy men.

95 easure resting regional cerebral blood flow (rCBF) in 52 individuals at ultra-high risk for psychosis

96 H treatment on regional cerebral blood flow (rCBF) in ADHD patients.

97 gnosis on glutamate and cerebral blood flow (rCBF) in adults with SZ and healthy controls.

98 ne craving and regional cerebral blood flow (rCBF) in distributed sites.

99 l volumes, and regional cerebral blood flow (rCBF) in healthy controls (HC) (n = 24), patients diagno

100 DBS increases regional cerebral blood flow (rCBF) in immediate downstream targets but does not revea

101 normalities of regional cerebral blood flow (rCBF) in major depression.

102 SPECT to study regional cerebral blood flow (rCBF) in patients with HD during rest and maze testing.

103 ET) to measure regional cerebral blood flow (rCBF) in sighted and congenitally blind subjects perform

104 ect of Alos on regional cerebral blood flow (rCBF) in the absence and presence of rectal or sigmoid s

105 lution, and of regional cerebral blood flow (rCBF) in the brain of transgenic APP23 mice.

106 covaried with regional cerebral blood flow (rCBF) in the dorsal medial prefrontal cortex, rostral an

107 owed increased regional cerebral blood flow (rCBF) in the vmPFC on both versions of the IGT compared

108 frontal cortex regional cerebral blood flow (rCBF) is lower in cocaine-dependent subjects than in non

109 cerebral blood volume (rCBV) and blood flow (rCBF) maps were acquired before chemoradiotherapy and at

110 to placebo on regional cerebral blood flow (rCBF) measured by SPECT in healthy volunteers to charact

111 lectrode and changes in cerebral blood flow (rCBF) measured with a laser Doppler probe placed over th

112 fy D3-mediated regional cerebral blood flow (rCBF) responses in living primates.

113 identified on regional cerebral blood flow (rCBF) SPECT scans of adolescent children and young adult

114 In regional cerebral blood flow (rCBF) studies with isoflurane and sevoflurane, there is

115 less change in regional cerebral blood flow (rCBF) to the anterior cingulate and supplementary motor

116 ess changes in regional cerebral blood flow (rCBF) upon awakening from stage 2 sleep.

117 We measured regional cerebral blood flow (rCBF) using pseudo-continuous arterial spin labelling.

118 Regional cerebral blood flow (rCBF) was assessed by using the bolus (15)O-labeled wate

119 Regional cerebral blood flow (rCBF) was measured within and outside the perfusion defi

120 Regional cerebral blood flow (rCBF) was monitored by Laser-Doppler flowmetry.

121 Regional cerebral blood flow (rCBF) was monitored through a Laser-Doppler flow probe a

122 e experiments, regional cerebral blood flow (rCBF) was recorded by laser Doppler flowmetry.

123 phy (SPECT) of regional cerebral blood flow (rCBF) was used to compare the central nervous system res

124 in normalized regional cerebral blood flow (rCBF) were assessed while participants repetitively wrot

125 traversion and regional cerebral blood flow (rCBF) while participants were exposed to olfactory stimu

126 ing to measure regional cerebral blood flow (rCBF) while they performed kinematically matched sequenc

127 activation of regional cerebral blood flow (rCBF) with PASAT in patients with mild TBI to explore me

128 of hippocampal regional cerebral blood flow (rCBF) with pulsed ASL.

129 nal changes in regional cerebral blood flow (rCBF), assessed by (15)O-water PET, over a mean 7 year p

130 ology, such as regional cerebral blood flow (rCBF), remains incompletely understood.

131 s a measure of regional cerebral blood flow (rCBF), we investigated the relationship between reduced

132 ET) imaging of regional cerebral blood flow (rCBF).

133 hich increases regional cerebral blood flow (rCBF).

134 obally elevate regional cerebral blood flow (rCBF).

135 d reduction in regional cerebral blood flow (rCBF).

136 ed hippocampal regional cerebral blood flow (rCBF).

137 ts of lesional relative cerebral blood flow (rCBF): 1.89 +/- 0.32 (0.72 mg E2) vs. 1.32 +/- 0.19 (P),

138 dated measure (relative cerebral blood flow [rCBF], <30%), thrombectomy patients had a smaller median

139 ed as a percentage of cerebellar blood flow, rCBF in the striatum and orbital cortex in patients with

140 The finding of focal rCBF abnormalities in the right hemisphere of 2 right-ha

141 n) and arterial spin labeling evaluation for rCBF.

142 hronic MJ users showed significantly greater rCBF than controls in the vmPFC on the standard IGT and

143 In the OA group, rCBF increases representing ongoing pain were identified

144 F differences between OA and control groups, rCBF differences between sessions within each group, and

145 several DMN and ECN regions exhibited higher rCBF per unit connectivity strength (rCBF/FCS ratio); wh

146 ith Val homozygotes, Met carriers had higher rCBF in prefrontal (BA25 extending into BA10) and hippoc

147 rtex) in which Val homozygotes showed higher rCBF in females than males, but Met carriers showed the

148 stigmine significantly decreased hippocampal rCBF in control subjects (P < .0005) and veterans with s

149 .05) but significantly increased hippocampal rCBF in veterans with syndrome 2 (P < .005) and veterans

150 a longitudinal reduction in left hippocampal rCBF that was not evident in subjects who remained in a

151 sociated with significantly less hippocampal rCBF.

152 ASL MR imaging examination of hippocampal rCBF in a cholinergic challenge experiment may be useful

153 found for working memory-related hippocampal rCBF change, which was uniquely attenuated in Met allele

154 However, rCBF increases in the prefrontal cortex were significant

155 In response to hypoglycemia, rCBF was significantly increased in the thalamus, medial

156 effects of L-NAME inhibition on brain pH(i), rCBF, and NADH redox state during 3 h of severe focal ce

157 ss BBB disruption; PHD3-/- mice had impaired rCBF upon early reperfusion but comparable functional ou

158 t whether visually apparent abnormalities in rCBF constitute statistically significant differences be

159 activity, this study assessed alterations in rCBF and related resting state functional connectivity (

160 However, we noted no changes in rCBF and flux in presymptomatic carriers compared with c

161 Changes in rCBF correlated positively with NO(x) production; increa

162 ss expensive method for assessing changes in rCBF during hypoglycemia without radiation exposure.

163 Changes in rCBF in the caudate nucleus predicted gene status (P = 0

164 There were significant changes in rCBF in the dorsal rostral pons, anterior cingulate cort

165 d quantification of IN-OT-induced changes in rCBF in the living human brain unaffected by cognitive,

166 Task-related changes in rCBF in the men without ADHD were more prominent in the

167 SPECT was used to assess changes in rCBF induced by amphetamine in 16 healthy volunteers.

168 )C-PIB show greater longitudinal declines in rCBF in certain areas, representing regions with greater

169 ual cortex and cerebellum show a decrease in rCBF, in a dose range of 0.2-1 minimum alveolar concentr

170 e produced statistically robust decreases in rCBF in bilateral orbitofrontal cortex, thalamus, opercu

171 ine induces focal increases and decreases in rCBF in healthy volunteers in areas primarily innervated

172 Greater longitudinal decreases in rCBF in the high-(11)C-PIB group than in the low-(11)C-P

173 efrontal cortex and significant decreases in rCBF in the vicinity of the limbic/paralimbic areas (i.e

174 Areas of relative increases or decreases in rCBF were measured by using the [(15)O]H(2)O method.

175 t were greater than or equal to decreases in rCBF.

176 for age was used to test for differences in rCBF after the cholinergic challenge across the four gro

177 d increase in rCBF after the initial drop in rCBF at the onset of MCAO.

178 icantly reduced, by over 59 %, elevations in rCBF and, by 78 %, changes in EEG evoked from RVLM.

179 and subthalamus revealed that elevations in rCBF were elicited only from a limited area, which encom

180 rats had less regulatory rebound increase in rCBF after the initial drop in rCBF at the onset of MCAO

181 The abnormal increase in rCBF was found to have progressed to the left hippocampu

182 differences such as the delayed increase in rCBF.

183 Greater longitudinal increases in rCBF are also observed in those with higher amyloid load

184 sitively with NO(x) production; increases in rCBF during HBO2 exposure were associated with large inc

185 maging techniques found similar increases in rCBF in the thalamus, medial prefrontal cortex, and glob

186 obese men produced significant increases in rCBF in the vicinity of the ventromedial and dorsolatera

187 was indication of compensatory increases in rCBF of the occipital cortex during incremental learning

188 Greater increases in rCBF over time in the high-(11)C-PIB group were found in

189 These increases in rCBF precede the onset of O2-induced convulsions.

190 Increases in rCBF suggest a cellular and vascular compensatory proces

191 reflected by both decreases and increases in rCBF.

192 ne, there is a consistent pattern of rise in rCBF in the anterior cingulate cortex and insula while t

193 s expected, bilateral STN DBS also increased rCBF in the bilateral thalami, right midbrain, and decre

194 Amphetamine increased rCBF in two mesial prefrontal zones (Brodmann's areas 8

195 en improvement in bradykinesia and increased rCBF in the thalamus (r(s) = 0.31, P < 0.05).

196 MCI patients had increased rCBF in the left hippocampus (P < .001), right amygdala

197 AD patients had increased rCBF in the right anterior cingulate gyrus (P = .02) com

198 while nicotine (1.2 nmol in 40 nl) increased rCBF by 13 +/- 5 % and synchronized the EEG from MCVA.

199 rametric mapping analysis revealed increased rCBF bilaterally in the thalamus and decreased rCBF in t

200 n (10 sec train) at an active site increased rCBF by 25 +/- 6%.

201 exercise and mental stress tasks, increased rCBF in cerebellar vermis, right anterior cingulate and

202 tion initiates CNS O2 toxicity by increasing rCBF, which allows excessive O2 to be delivered to the b

203 ects of electrical stimulation by increasing rCBF.

204 us study showing significant STN DBS-induced rCBF change in the thalamus, midbrain and supplementary

205 y involved in mechanical stimulation-induced rCBF changes and thus may represent therapeutic targets

206 dium ion channel blocker was able to inhibit rCBF changes in both the cat and rats.

207 study the authors prospectively investigated rCBF and clinical response to venlafaxine, a novel antid

208 verapamil brain distributional clearance, K1/rCBF).

209 Voxel-level rCBF was compared among groups by using an analysis of v

210 Longitudinal rCBF changes differed significantly between high- (n=10)

211 nt showed significantly greater longitudinal rCBF increases in orbitofrontal, medial frontal, and ant

212 Differences in longitudinal rCBF changes between high- and low-(11)C-PIB groups were

213 e modified suture technique produced a lower rCBF, larger infarct size, smaller variance of infarct s

214 aine-addicted subjects showed markedly lower rCBF in the bilateral orbitofrontal cortex than the comp

215 ingulate and right insula covaried with MAP; rCBF in pons, cerebellum and right insula covaried with

216 significantly decreased cortical grey matter rCBF in the occipital lobe (mean difference -11.1 mL/100

217 Mean rCBF (ml/100 g/min) for S1BF were: S1BF [0 s] left corte

218 Mean rCBF levels were compared by using random effects regres

219 resence of hypertension and related the mean rCBF in those clusters to the presence of MCI and AD.

220 overnight and underwent PET scans to measure rCBF responses to bilateral STN DBS.

221 oton emission computed tomography to measure rCBF, after which they completed the Gambling Task.

222 (11)C-verapamil, and (15)O-water to measure rCBF.

223 We measured rCBF and EEG responses in rats exposed at 4 to 6 atmosph

224 When the subjects were not taking MPH, rCBF was higher in the motor, premotor, and the anterior

225 In rats pretreated with L-NAME, rCBF remained maximally decreased throughout 75 min of H

226 eived placebo at both times to assess normal rCBF variability.

227 e degree of "left-handedness" and normalized rCBF during right-hand writing.

228 Nicotine increased normalized rCBF in the left frontal region and decreased rCBF in th

229 Observed rCBF changes potentially indicate dysregulated CNS appra

230 h as ion channel manipulation, and observing rCBF changes may help our understanding of migraine aura

231 l stimulus conditions revealed activation of rCBF in the left medial prefrontal and left anterior cin

232 A principal-components analysis of rCBF data pooled from the two studies identified three f

233 Fully quantified assessments of rCBF and rCMR for glucose were obtained while subjects w

234 hy subjects: there was a greater decrease of rCBF in lateral and medial premotor areas, putamen, and

235 ncentration causes a predominant decrease of rCBF in the cortical regions and increase of rCBF in the

236 apping was used to analyze the PET images of rCBF changes.

237 rCBF in the cortical regions and increase of rCBF in the subcortical regions.

238 ected by AD is reduced and is independent of rCBF.

239 ha-smooth muscle actin, and the induction of rCBF-A appropriately alters their expression as well.

240 with automated three-dimensional matching of rCBF images was used to coregister and quantify results.

241 positron emission tomography measurements of rCBF at baseline and up to eight annual follow-up visits

242 e amyloid plaques, and the quantification of rCBF.

243 Reduction of rCBF appeared greatest in the absence of visceral stimul

244 tion, higher values and rapid restoration of rCBF were observed in group 2, while rCBF in both hemisp

245 mic region and allowing rapid restoration of rCBF.

246 We reinvestigated a series of rCBF SPECT scans obtained several years ago on drug-naiv

247 Our results support the usefulness of rCBF SPECT imaging as a diagnostic aid in PPA.

248 The effects of mecamylamine on rCBF were generally opposite to those of nicotine.

249 Pattern recognition on rCBF maps indicated that IN-OT-induced changes were sust

250 Neither the percentage change of rCBV or rCBF predicted survival, whereas the regional response e

251 n-of-interest analysis for change in rCBV or rCBF to the change in perfusion parameters on the basis

252 contrast, change in average percent rCBV or rCBF, MR tumor volume changes, age, extent of resection,

253 During exposures to hyperbaric oxygen rCBF decreased at 4 ATA, decreased for the initial 30 mi

254 Together, both increased pHp rCBF and strengthened pHp-PCC rsFC predicted relapse wit

255 eters on the basis of PRM (PRM(rCBV) and PRM(rCBF)) for their accuracy in predicting overall survival

256 However, for both subgroups with PTSD, rCBF changes in medial frontal gyrus were inversely corr

257 h placebo, improved IBS symptoms and reduced rCBF in 5-HT3R containing regions of the EMS, but not in

258 llow-up, symptomatic improvement and reduced rCBF in the hippocampus and ventral striatum were observ

259 Correlational analysis between reduced rCBF and BNT was performed.

260 minating the confounding effect that reduced rCBF has on assessment of BBB P-glycoprotein activity an

261 Analysis of learning-related rCBF in network regions revealed improvement in baseline

262 the optimal ischemic core threshold remained rCBF <30% (AUC, 0.83; 95% CI, 0.77, 0.85).

263 tients with HD showed an increase in resting rCBF for all brain regions measured except the caudate n

264 xploratory analyses of interregional resting rCBF covariation revealed a specific and significant dia

265 evidence of a distinctive pattern of resting rCBF abnormalities associated with CFS.

266 Resultant rCBF changes were evaluated parametrically through the f

267 Sessionwise rCBF differences in the OA group in the postcentral, ros

268 No significant sessionwise rCBF differences were observed in controls.

269 s within each group, and whether sessionwise rCBF differences were related to variability in perceive

270 dent longitudinal increases in resting state rCBF in brain regions intrinsic to memory processes.

271 higher rCBF per unit connectivity strength (rCBF/FCS ratio); whereas, this index was lower in poster

272 ts who achieve rapid reperfusion, a stricter rCBF threshold to estimate the ischemic core should be c

273 auditory localization in the blind subjects, rCBF activity in the right posterior parietal cortex was

274 The rCBF data were acquired 15 min before and up to 78 min a

275 The rCBF in the right hemisphere reticular system was relate

276 n inter-regional correlation analysis on the rCBF data set.

277 Their rCBF data were automatically normalized to whole-brain c

278 Although the cause of these rCBF changes in HD patients is unclear, nitric oxide syn

279 of pramipexole's effects suggest that these rCBF responses indicate functional effects of a D3-prefe

280 dy, and clinical response appears related to rCBF changes.

281 the right amygdala and negatively related to rCBF in medial frontal gyrus.

282 , symptom severity was positively related to rCBF in the right amygdala and negatively related to rCB

283 ed group, mood provocation produced a unique rCBF decrease in pregenual anterior cingulate 24a.

284 e ischemic core in thrombectomy patients was rCBF <20% (area under the curve [AUC], 0.89; 95% CI, 0.8

285 When rCBF was calculated as a percentage of cerebellar blood

286 When rCBF was calculated as a percentage of cerebellar rCBF,

287 se men than in lean men (P < 0.005), whereas rCBF decreases in the hypothalamus and thalamus were att

288 tion of rCBF were observed in group 2, while rCBF in both hemispheres was significantly decreased in

289 posed to pleasant and unpleasant odors while rCBF was measured using [(15)O] water PET.

290 , postural stability and gait correlate with rCBF responses in a priori determined regions.

291 frontal gyrus were inversely correlated with rCBF changes in the left amygdala and the right amygdala

292 s sessions was significantly correlated with rCBF decreases in the 5-HT3R-rich amygdala, ventral stri

293 condition, extraversion was correlated with rCBF in the amygdala and occipital cortex.

294 condition, extraversion was correlated with rCBF in the occipital cortex and inferior temporal gyrus

295 S showed a striking spatial correlation with rCBF, and the correlation was stronger in the default-mo

296 y, prestimulus cortisol levels covaried with rCBF in the subgenual anterior cingulate cortex.

297 Prestimulus cortisol levels covaried with rCBF responses in the rostral anterior cingulate cortex.

298 een-genotype group differences covaries with rCBF in other nodes throughout the brain in a genotype-

299 Glu and WM rCBF decreased linearly with age while Gln and Gln/Glu i

300 Glu and WM rCBF were correlated with the UCSD Performance-Based Ski

301 Glu, Gln, Gln/Glu, and AC white matter (WM) rCBF.