ライフサイエンスコーパス: reticular

1 ctivating structures including the ascending reticular activating system (ARAS), contributing to neoc

2 running and is associated with the ascending reticular activating system.

3 ive regions, with its peak in the brain stem reticular activating system.

4 located at the confluence of the limbic and reticular activating systems.

5 bservations that behavioral state influences reticular activity.

6 ith OLP from saliva samples of two subtypes (reticular and erosive) of OLP patients and healthy contr

7 f substrates across and into the endoplasmic reticular and mitochondrial membranes of eukaryotes.

8 more precisely the segmental imprint in the reticular and motor basal hindbrain of adult goldfish.

9 lexes at discrete plasmalemmal, sarcoplasmic reticular and myofilament sites, reveals differential ki

10 These reticular and vesicular structures are more abundant in

11 Notably, pex3 mutant cells already contain reticular and vesicular structures that harbor key prote

12 opodia to spread along the cell base and are reticular and/or fibrous in character.

13 ways compartment: epithelium (basal region), reticular basement membrane (Rbm) and underlying lamina

14 We assessed airway inflammation, reticular basement membrane (RBM) thickness, airway smoo

15 n BAL and assessed smooth muscle area (SMA), reticular basement membrane (RBM) thickness, and epithel

16 but not IL-13, was associated with increased reticular basement membrane thickness in EB specimens fr

17 modeling using image analysis, together with reticular basement membrane thickness, mucus gland area,

18 n of extracellular matrix, collagen markers, reticular basement membrane thickness, or glandular perc

19 n many cell types by sensing low endoplasmic reticular Ca(2+) levels and then coupling to plasma memb

20 addition, we identify the sarco/endoplasmic reticular Ca(2+)-ATPase 1A and the alpha1S subunit of th

21 sitive HCM, whereas sarcoplasmic endoplasmic reticular calcium ATPase 2 abundance and sarcoplasmic re

22 of genotype, as was sarcoplasmic endoplasmic reticular calcium ATPase 2/phospholamban protein ratio (

23 (45)Ca sarcoplasmic endoplasmic reticular calcium ATPaseuptake assay showed reduced upta

24 Down regulation of dStim, the endoplasmic reticular calcium sensor and a principal component of SO

25 rteriolar endothelial cells, CXCL12-abundant reticular (CAR) cells, and cells of the osteoblastic lin

26 ch as CXC chemokine ligand (CXCL)12-abundant reticular (CAR) cells, stem cell factor-expressing cells

27 at lymphotoxin signaling in the fibroblastic reticular cell (FRC) stromal subset was required for pro

28 However, the mechanisms that regulate reticular cell function are not well understood.

29 and pathological alterations of fibroblastic reticular cell networks in the draining lymph nodes.

30 Mice lacking fibroblastic reticular cell PDPN or platelet CLEC-2 exhibited signifi

31 lymph nodes, dendritic cells (DCs) maintain reticular cell survival in multiple compartments.

32 in (PDPN) signalling in stromal fibroblastic reticular cells (FRCs) and its modulation by CLEC-2 expr

33 Fibroblastic reticular cells (FRCs) are known to inhabit T cell-rich

34 In lymph nodes, fibroblastic reticular cells (FRCs) form a collagen-based reticular n

35 Fibroblastic reticular cells (FRCs) form the cellular scaffold of lym

36 Fibroblastic reticular cells (FRCs) in the T cell zone of lymph nodes

37 anized in a rigid 3D network of fibroblastic reticular cells (FRCs) that are a rich cytokine source.

38 gp38(+) stromal fibroblastic reticular cells (FRCs) that express VEGF are enriched fo

39 LN) stromal cells, particularly fibroblastic reticular cells (FRCs), provide critical structural supp

40 Fibroblastic reticular cells (FRCs), through their expression of CC c

41 al expansion and differentiation of marginal reticular cells (MRCs), a population of lymphoid stromal

42 xpressing host cells, including fibroblastic reticular cells and follicular dendritic cells.

43 romal organizers give rise to adult marginal reticular cells and form a dedicated stromal niche for i

44 stromal cells, which include CXCL12-abundant reticular cells and osteoblasts, results in constitutive

45 icroscopy showed that SFTSV colocalized with reticular cells but did not colocalize with dendritic ce

46 ectional crosstalk with MAdCAM-1(+) marginal reticular cells by providing tumor-necrosis factor (TNF)

47 The major target cells of SFTSV appear to be reticular cells in lymphoid tissues of intestine and spl

48 s support lymphocyte function, and targeting reticular cells is a potential strategy for controlling

49 component of the CHT niche, and mature into reticular cells lining and interconnecting sinusoids.

50 critical mediators, and LTbetaR signaling on reticular cells mediated cell survival by modulating pod

51 Fibroblastic reticular cells responded rapidly to DST by transcribing

52 Within secondary lymphoid tissues, stromal reticular cells support lymphocyte function, and targeti

53 occurs in the lymph node, where fibroblastic reticular cells support the maintenance of naive T cells

54 rents in the postsynaptic thalamic relay and reticular cells were dramatically elevated, favoring reb

55 Fibroblastic reticular cells were flow-sorted at different timepoints

56 s, uncontrolled proliferation of bone marrow reticular cells, and fibrosis of the marrow compartment.

57 including endothelial cells, CXCL12-abundant reticular cells, leptin-receptor-positive stromal cells,

58 immunoregulatory properties of fibroblastic reticular cells, we reviewed the most recent advances in

59 hat gives rise exclusively to adult marginal reticular cells.

60 follicular dendritic cells, and fibroblastic reticular cells.

61 ells, a profile associated with fibroblastic reticular cells.

62 Reticular chemistry approach was successfully employed t

63 Here, we demonstrate the successful use of reticular chemistry as an appropriate strategy for the d

64 We report the use of reticular chemistry for the fabrication of a chemically

65 selective catalysts, based on the premise of reticular chemistry in MOFs and the ability to incorpora

66 Successful implementation of reticular chemistry using a judiciously designed rigid o

67 This work illustrates two principles of reticular chemistry: first, selectivity for helical over

68 ectivity-based optogenetic tagging, we found reticular circuits to be composed of distinct subnetwork

69 However, when Hh signalling is inhibited the reticular dermis does not respond to epidermal beta-cate

70 to the transition between the papillary and reticular dermis.

71 basaloid cells present in the papillary and reticular dermis.

72 ceous unit but also within the papillary and reticular dermis.

73 adipogenic differentiation in the developing reticular dermis.

74 score was significantly worse in those with reticular drusen (mean score +/- standard deviation [SD,

75 unctional analysis in patients with AMD with reticular drusen (RDR) have focused on photopic sensitiv

76 Reticular drusen identified from photographs were confir

77 Current smoking at baseline predicted higher reticular drusen incidence (OR 2.1, 95% CI 1.0-4.5) afte

78 95% CI, 2.1-4.4) were associated with higher reticular drusen incidence.

79 A higher proportion of eyes with reticular drusen located outside versus within the macul

80 Neither total area nor central location of reticular drusen predicted 5-year progression to late AM

81 sured were significantly lower in those with reticular drusen than in those without.

82 ith decreased likelihood of progression from reticular drusen to late AMD (adjusted OR, 0.5; 95% CI,

83 ese risk factors and 5-year progression from reticular drusen to late AMD.

84 The 15-year cumulative incidence of reticular drusen was 4.0% (n = 95).

85 SNP rs10490924, and the 15-year incidence of reticular drusen were analyzed in discrete logistic regr

86 Of 118 eyes with reticular drusen, 40 (33.9%) developed late AMD over 5 y

87 re associated with greater long-term risk of reticular drusen.

88 ndrial intermembrane space and is mutated in reticular dysgenesis (RD), a rare form of severe combine

89 D and 32 with leaky SCID, Omenn syndrome, or reticular dysgenesis.

90 ied as having leaky SCID, Omenn syndrome, or reticular dysgenesis; and 3% had a history of enzyme rep

91 ot affect TGF-beta target gene expression in reticular fibroblasts, and TGF-beta inhibition does not

92 ation, differentiation and ECM production by reticular fibroblasts.

93 ive cell cluster is centered on a tegmental (reticular) field traversed by fibers of the superior cer

94 ulomotor area from the central mesencephalic reticular formation (cMRF), a region implicated in horiz

95 The mesencephalic reticular formation (MRF) is formed by the pedunculopont

96 he two major components of the mesencephalic reticular formation (MRF), namely the pedunculopontine a

97 lycinergic fibers ascending from the pontine reticular formation (PRF) of the brainstem evoked fast a

98 characteristic locations within the pontine reticular formation (PRF).

99 ting with the parabrachial nucleus (PBN) and reticular formation (RF), and those interconnecting NST

100 vibrissa-related region of the intermediate reticular formation (vIRt).

101 of startle response neurons of the mammalian reticular formation [4], and studies of this circuit hav

102 lon (pretectum and thalamus), mesencephalon (reticular formation and nucleus ruber), rhombencephalon

103 of Darkschewitsch, mesencephalic and pontine reticular formation and pontine nuclei.

104 estigated the convergence of inputs from the reticular formation and sensory afferents on presynaptic

105 he PCC to the ventral tegmental area/pontine reticular formation and thalamus, in addition to the LC,

106 formed a column of scattered neurons in the reticular formation and were found in the octavolateral

107 projections, several parts of the medullary reticular formation as well as the spinally projecting r

108 he lateral part of the SC and the underlying reticular formation corresponding to locations where rea

109 The reticular formation in the brainstem controls motor outp

110 ated by a central amygdala projection to the reticular formation in the brainstem.

111 superior colliculus (SC) and the underlying reticular formation is correlated with the initiation an

112 cingulate cortex (1.6-fold increase) and the reticular formation of the medulla (6.5-fold increase).

113 ells localized in the hindbrain intermediate reticular formation were noncholinergic in nature (nonmo

114 cMRF input by injecting this portion of the reticular formation with anterograde tracers in combinat

115 ncerta (ZI), anterior pretectum, and pontine reticular formation) provides temporally precise and foc

116 cularis, octavolateralis area, parvocellular reticular formation), many of the ASP-immunonegative neu

117 ruber), rhombencephalon (cerebellar nucleus, reticular formation, and inferior olive), and spinal cor

118 AG, the cuneiform nucleus, the mesencephalic reticular formation, and the superior colliculus.

119 al midbrain tegmentum, posterior tuberculum, reticular formation, and viscerosensory lobe.

120 hetized animals by stimulating the brainstem reticular formation, basal forebrain, or thalamus.

121 to a broad network of regions including the reticular formation, basal ganglia, thalamus, posterior

122 em in the vicinity of the raphe nucleus, and reticular formation, hypothalamus, and septum/striatum o

123 neocortex (middle frontal gyrus), brainstem reticular formation, precerebellar nuclei, and the red n

124 ensory afferents and premotor neurons of the reticular formation, where central pattern generator cir

125 deep layers of the lateral SC and underlying reticular formation.

126 us, solitary tract nucleus, motoneurons, and reticular formation.

127 lung occupied by ground glass, ground glass-reticular (GGR), honeycombing, emphysema, and normal lun

128 mphadenopathy (16%), pleural effusion (12%), reticular infiltration (4%), and pericardial effusion (4

129 tic input to thalamocortical relay cells and reticular interneurons and activate intrathalamic circui

130 in the ER but changes its distribution from reticular into punctate spots.

131 a subpopulation of Dbx1-derived intermediate reticular (IRt) neurons are rhythmically active during i

132 The magnitude and phase differences between reticular lamina and basilar membrane vibrations are abs

133 ell into a narrow fluid-filled space between reticular lamina and tectorial membrane.

134 r flow that arises from shearing between the reticular lamina and the tectorial membrane.

135 ng subnanometer vibrations directly from the reticular lamina at the apical ends of outer hair cells

136 s travelling wave vibrates in phase with the reticular lamina at the best frequency, and results in m

137 expected; instead, they actively vibrate the reticular lamina over a broad frequency range.

138 s can generate sufficient force to drive the reticular lamina over all audible frequencies in living

139 The phase relation of reticular lamina to basilar membrane vibration changes w

140 The outer hair cell-driven reticular lamina vibration collaboratively interacts wit

141 living mouse cochleae that the sound-induced reticular lamina vibration is substantially larger than

142 ial vibrations of the tectorial membrane and reticular lamina were tuned.

143 band and slow sharply tuned responses of the reticular lamina, but only a slow tuned response of the

144 t the OHC bundle, the tectorial membrane and reticular lamina, to the transverse motion of the basila

145 cell stereocilia, the tectorial membrane and reticular lamina, were sharply tuned in the radial direc

146 t junctions that outline the scala media and reticular lamina.

147 ere rather located in both the papillary and reticular layers of the LP.

148 This reticular-limbic pathway may thus function in processing

149 and generate osteoblasts, chondrocytes, and reticular marrow stromal cells, but not adipocytes.

150 CM/adhesion coupling, whereas highly pliable reticular matrices promote adhesion retraction.

151 71(-) infected red blood cell devoid of host reticular matter, a process that normally spans 24 hours

152 the trans side of the plasma or endoplasmic reticular membrane, respectively.

153 s of Parkin did produce abnormal tubular and reticular mitochondria restricted to the motor cell bodi

154 talization to the mitochondria and preserves reticular mitochondrial morphology in response to sI.

155 neonatal rat cardiomyocytes (NRCMs) retain a reticular mitochondrial phenotype after simulated ischem

156 s and T cells, which resulted in an expanded reticular network and enhanced immunity.

157 e vasculature, expansion of the fibroblastic reticular network and maintenance of lymphoid stromal ph

158 The cardiomyocyte reticular network is the ideal location of sensors for b

159 alveoli into contiguous alveoli, creating a reticular network that leads to death by asphyxiation.

160 reticular cells (FRCs) form a collagen-based reticular network that supports migratory dendritic cell

161 Abnormal subepithelial cells, which formed reticular networks deep to the basal epithelial cells, w

162 s partially resemble the cells that form the reticular networks in organized lymphoid tissues, potent

163 Thalamic reticular neurons are inhibitory cells interconnected by

164 thors show that the receptive field sizes of reticular neurons are small enough to provide localized

165 biophysical properties interconnecting some reticular neurons in mice lacking Cx36.

166 Our results show that reticular neurons in the cat operate over discrete spati

167 ence revealed spinal-projecting Galphat-S-ir reticular neurons in the caudal hindbrain.

168 Characteristically, the largest reticular neurons of the lamprey brainstem (Muller cells

169 rents within postsynaptic thalamic relay and reticular neurons.

170 AA+ ATPases that localize to the endoplasmic reticular/nuclear envelope (ER/NE) lumen.

171 ase resident in the lumen of the endoplasmic reticular/nuclear envelope.

172 r cortex (M1) strongly innervates brain stem reticular nuclei containing whisker premotor neurons, wh

173 interpeduncular nucleus, superior and middle reticular nuclei, magnocellular vestibular nucleus, soli

174 ZI has strong connections between brainstem reticular nuclei, sensory nuclei, and nonspecific thalam

175 by cholinergic neurons in several motor and reticular nuclei.

176 d the rostral parvicellular and intermediate reticular nuclei.

177 us (mRN, rostral hindbrain) and the inferior reticular nucleus (iRN, caudal hindbrain).

178 re input system originating from the lateral reticular nucleus (LRN) can represent sensory event timi

179 LR sends bilateral projections to the middle reticular nucleus (mRN, rostral hindbrain) and the infer

180 d responses to GABA uncaging in the thalamic reticular nucleus (nRT) that is absent in both nm1054 mi

181 y GABA released from neurons of the thalamic reticular nucleus (nRT), acting predominantly via synapt

182 ffect on synaptic inhibition in the thalamic reticular nucleus (nRT).

183 restricted prefrontal areas to the thalamic reticular nucleus (RE), consistent with recent anatomica

184 down-regulation of GABA inhibition from the reticular nucleus (RTN).

185 y two major inhibitory systems: the thalamic reticular nucleus (TRN) and extrathalamic inhibitory (ET

186 tiple single-unit recordings in the thalamic reticular nucleus (TRN) and medial prefrontal cortex (mP

187 s for a specific involvement of the thalamic reticular nucleus (TRN) come from its unique neuronal ch

188 nerally thought that neurons in the thalamic reticular nucleus (TRN) form GABAergic synapses with oth

189 It is known that the thalamic reticular nucleus (TRN) gates sensory information en rou

190 The inhibitory thalamic reticular nucleus (TRN) is a hub of the attentional syst

191 The thalamic reticular nucleus (TRN) is a unique brain structure at t

192 The thalamic reticular nucleus (TRN) is hypothesized to regulate thal

193 agation in thalamocortical (TC) and thalamic reticular nucleus (TRN) neurons remains unknown.

194 liminates rebound bursting in model thalamic reticular nucleus (TRN) neurons.

195 show that GABAergic neurons in the thalamic reticular nucleus (TRN) of mice and rats form two types

196 Neurons within the mature thalamic reticular nucleus (TRN) powerfully inhibit ventrobasal (

197 show that local tonic activation of thalamic reticular nucleus (TRN) rapidly induces slow wave activi

198 olinergic transmission in the mouse thalamic reticular nucleus (TRN), a brain structure essential for

199 nergic synaptic transmission in the thalamic reticular nucleus (TRN), a brain structure intimately in

200 hd1 is selectively expressed in the thalamic reticular nucleus (TRN), a group of GABAergic neurons th

201 s (VB) and GABAergic neurons in the thalamic reticular nucleus (TRN).

202 ion, we found neurons of the visual thalamic reticular nucleus (visTRN) to exhibit PFC-dependent chan

203 icity at electrical synapses in the thalamic reticular nucleus - paired burst spiking in coupled neur

204 on evoked responses from inhibitory thalamic reticular nucleus and excitatory tectothalamic terminals

205 which was associated with decreased thalamic reticular nucleus and primary somatosensory cortex activ

206 unreported retinotopic maps in the thalamic reticular nucleus and the substantia nigra.

207 s between inhibitory neurons of the thalamic reticular nucleus are bidirectionally modulated by chang

208 s been proposed that neurons in the thalamic reticular nucleus are interconnected through GABAergic s

209 The thalamic reticular nucleus has a critical role in modulating info

210 address the question of whether cells in the reticular nucleus have receptive fields small enough to

211 ection from the ipsilesional gigantocellular reticular nucleus in response to the injury.

212 The thalamic reticular nucleus is an important structure governing th

213 alamocortical neurons and GABAergic thalamic reticular nucleus neurons and that these properties are

214 major synaptic input from GABAergic thalamic reticular nucleus neurons, as well as neurons and astroc

215 In thalamocortical and thalamic reticular nucleus neurons, the site of AP generation and

216 the first 2 weeks after birth, the thalamic reticular nucleus of the mouse lacks intrinsic GABAergic

217 Because the reticular nucleus of the thalamus (RtN) provides tonic f

218 The visual sector of the overlying thalamic reticular nucleus receives input from relay cells and su

219 hlight hypothesis proposes that the thalamic reticular nucleus regulates thalamic relay activity thro

220 tions from the visual sector of the thalamic reticular nucleus to the lateral geniculate nucleus comp

221 al nucleus, somatosensory thalamus, thalamic reticular nucleus, and primary somatosensory cortex.

222 ers of the superior colliculus, the thalamic reticular nucleus, and the caudate nucleus.

223 the cochlear nucleus, and via caudal pontine reticular nucleus, pontine central gray, and MS, reached

224 her FEF connections were with the claustrum, reticular nucleus, zona incerta, lateral posterior and m

225 uence thalamic relay nuclei via the thalamic reticular nucleus.

226 by driving inhibitory cells of the thalamic reticular nucleus.

227 the GABAergic neurons of the rodent thalamic reticular nucleus.

228 ons and precerebellar neurons of the lateral reticular nucleus.

229 ypotheses about the function of the thalamic reticular nucleus.

230 ules (n = 14), followed by cysts (n = 6) and reticular opacities (n = 4).

231 Consecutive patients with reticular pattern dystrophy of the retina underwent a co

232 In this study on reticular pattern dystrophy of the retina, SDOCT provide

233 along the interlobular septa, causing a fine reticular pattern on CT images.

234 bands of yellowish-white material forming a reticular pattern, called ribbon pseudodrusen, which wer

235 ethods of detection and only 1 that formed a reticular pattern.

236 yloid deposits were usually distributed in a reticular/pericellular pattern, whereas transthyretin am

237 Senile reticular pigmentary change was the predominant peripher

238 tary changes, reticular pseudodrusen, senile reticular pigmentary changes, cobblestone degeneration,

239 ctaval nuclei; the viscerosensory lobes; and reticular populations distributed from trigeminal to vag

240 f the ARMS2 risk allele (P = .4) between the reticular pseudodrusen (homozygous = 20.0%; heterozygous

241 of the CHF risk allele (P = .7) between the reticular pseudodrusen (homozygous = 26.7%; heterozygous

242 en (n = 30) or (2) large soft drusen without reticular pseudodrusen (n = 43).

243 Reticular pseudodrusen (RPD) are frequently associated w

244 s were graded for the presence or absence of reticular pseudodrusen (RPD) using a multimodal approach

245 rrelation to visual acuity (VA) in eyes with reticular pseudodrusen (RPD) vs those with drusen withou

246 Presence of reticular pseudodrusen (RPD) was assessed by masked grad

247 zed as soft drusen, cuticular drusen, and/or reticular pseudodrusen (RPD).

248 The ability to detect reticular pseudodrusen (RPD)/subretinal drusenoid deposi

249 Reticular pseudodrusen appeared as yellowish round to ov

250 Reticular pseudodrusen are a frequent finding in patient

251 Reticular pseudodrusen are associated more strongly with

252 Reticular pseudodrusen are highly concurrent with AMD an

253 The reticular pseudodrusen group was older (median age 87 vs

254 The reticular pseudodrusen phenotype was associated with inc

255 Reticular pseudodrusen represent subretinal deposits tha

256 Reticular pseudodrusen seem to confer an increased risk

257 The presence of reticular pseudodrusen was an independent risk factor fo

258 Reticular pseudodrusen was found in 43 participants (41%

259 Reticular pseudodrusen were deposits juxtaposed to photo

260 Reticular pseudodrusen were hyporeflective on NIR reflec

261 Reticular pseudodrusen were identified frequently in SFD

262 Peripheral reticular pseudodrusen were seen in 15%.

263 subjects (n = 73) with the phenotypes of (1) reticular pseudodrusen without large soft drusen (n = 30

264 ation levels (C3d:C3 ratio), the presence of reticular pseudodrusen, and AMD phenotype.

265 sen, hypopigmentary/hyperpigmentary changes, reticular pseudodrusen, senile reticular pigmentary chan

266 SupV) and the parvicellular and intermediate reticular regions dorsal to the facial motor nucleus.

267 essive excitatory cortical drive to thalamic reticular (RT) neurons or heightened inhibition of thala

268 Grem1 expression also identifies intestinal reticular stem cells (iRSCs) that are cells of origin fo

269 r), local caspase-3/7, and local endoplasmic reticular stress-related genes.

270 - common progenitors can give rise to marrow reticular stromal cells and perivascular mesenchymal pro

271 al motif that likely drives the formation of reticular structures in living cells.

272 equired to form three-way junctions found in reticular subdomains of the endoplasmic reticulum.

273 selection, in which the PFC biases thalamic reticular subnetworks to control thalamic sensory gain,

274 Recently, significant progress was made by reticular synthesis of related organic solid-state mater

275 applying the 'node and strut' principles of reticular synthesis to molecular crystals.

276 allization, and there are fewer examples of 'reticular synthesis', in which multiple building blocks

277 etal-organic frameworks (MOFs) are formed by reticular synthesis, which creates strong bonds between

278 gely cortical in origin and suggest that the reticular system contributed, at least in part, to these

279 are largely cortical in origin and that the reticular system contributed, at least in part, to these

280 An acoustic startle cue, which engages the reticular system, suppressed MEP size during power grip

281 tic startle cue, a stimulus that engages the reticular system, suppressed MEP size during power grip

282 hibition we assessed the contribution of the reticular system, which projects to cortical neurons, an

283 naptic linkage between anterogradely labeled reticular terminals and retrogradely labeled medial rect

284 thalamocortical cells when the presynaptic, reticular thalamic (nRT) neurons fired in tonic mode.

285 ortant regulators of [Cl(-)]i Neurons of the reticular thalamic (RT) nucleus express reduced levels o

286 r KCC2 is an important Cl(-) extruder in the reticular thalamic (RT) nucleus, despite this nucleus ha

287 activity of reciprocally coupled inhibitory reticular thalamic cells (nRTs) and quantified cycle-by-

288 Gd) of the medial geniculate body (MGB), the reticular thalamic nucleus and dorsal nucleus of the lat

289 eniculate body, suprageniculate nucleus, and reticular thalamic nucleus, as well as of the inferior c

290 ets them apart from GABAergic neurons of the reticular thalamic nucleus.

291 ons in BGMT and CbMT and with neurons in the reticular thalamic nucleus.

292 men, aged 18 to 69 years, who had at least 1 reticular vein with a minimum length of 10 cm in 1 of th

293 Reticular veins are subdermal veins located in the lower

294 ective than with 75% HG alone in eliminating reticular veins from the treated area (95.17% vs 85.40%;

295 sclerotherapy is the treatment of choice for reticular veins in the lower limbs, no consensus has bee

296 The reticular veins were measured on images obtained before

297 icacy end point was the disappearance of the reticular veins within 60 days after treatment with scle

298 G was superior to 75% HG alone in sclerosing reticular veins, with no statistical difference for comp

299 nol plus 70% HG or 75% HG alone to eliminate reticular veins.

300 cy and safety of 2 sclerosants used to treat reticular veins: 0.2% polidocanol diluted in 70% hyperto