ライフサイエンスコーパス: saclofen

コーパス検索結果 (１語後でソート)

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1 influx was reduced by the GABA(B) antagonist saclofen.

2 lline, but not by the GABAB receptor blocker saclofen.

3 ptors with the selective GABA(B) antagonist, saclofen.

4 of bicuculline (0, 7.5, 75, 150, 300 ng) or saclofen (0, 0.5, 1.5, 3, 5 mug) into the other site wit

5 ABA(A) (bicuculline, 75-150 ng) and GABA(B) (saclofen, 1.5-3 microg) antagonists administered into th

6 , whereas GABAB receptor antagonists [10 mum saclofen; 10-50 mum CGP55845 (p-3-aminopropyl-p-diethoxy

7 a combination of picrotoxin (50 microM) and saclofen (100 microM), and thus appeared to be mediated

8 nduced responses were sensitive to 2-hydroxy-saclofen (100-200 microM) and CGP55845 (200 nM).

9 The GABAB receptor antagonist saclofen (200 mum) occluded effects of CTOP but the GABA

10 GABAB agonist baclofen decreased, while 2-OH-saclofen (a GABAB antagonist) increased DA release in th

11 induced feeding in that both bicuculline and saclofen administered into the nucleus accumbens shell,

12 aptoacetate-induced intake was eliminated by saclofen and significantly reduced by bicuculline in the

13 Antagonists of GABAB (saclofen) and GABAC (picrotoxin) receptors partially inh

14 l was dose-dependently decreased by GABA(B) (saclofen) antagonism.

15 locked by GABA(A) (bicuculline) and GABA(B) (saclofen) antagonists.

16 The GABAB receptor antagonist 2-hydroxy-saclofen appeared to enhance the gamma activity by incre

17 m the nucleus accumbens shell was reduced by saclofen, but not by bicuculline or naltrexone.

18 d feeding was blocked by coadministration of saclofen, but was not affected by bicuculline.

19 bicuculline or the GABA(B) receptor blocker saclofen did not significantly affect food intake.

20 Correspondingly, NAC shell saclofen dose-dependently and significantly blocked feed

21 VTA saclofen dose-dependently and significantly blocked feed

22 cted to the rostral lateral hypothalamus and saclofen (GABA-B receptor antagonist), biccuculine (GABA

23 Neither bicuculline nor saclofen in the nucleus accumbens shell altered baseline

24 shell and eliminated by both bicuculline and saclofen in the ventral tegmental area.

25 These saclofen-induced cycles were, however, less well synchro

26 blocked by NAC shell pretreatment of either saclofen or bicuculline in rats.

27 treatment with either the GABA-B antagonist, saclofen, or the GABA-A antagonist, bicuculline, into th

28 duced by bicuculline and naltrexone, but not saclofen pretreatment, baclofen-induced feeding elicited

29 vated by microinjections of baclofen or 2-OH-saclofen, systemic injections of muscimol caused an inhi

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