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1 ants die at the transition from the first to second instar.
2 le (otd) are expressed throughout the entire second instar eye-antennal disc, conferring a default fa
3 e lymph glands of third-instar, but never of second-instar hosts, are almost always accompanied by di
4  instar, reduced MMP was already apparent at second instar in the cell bodies, axons and neuromuscula
5 ism of insect ecdysis between the first- and second-instar larva, while enclosed in the bag.
6 ozygous for the insert arrest development as second instar larvae and die without pupating.
7 emizygous for our emb alleles can develop to second instar larvae but persist at this stage and consi
8                 Remarkably, some E75A mutant second instar larvae display a heterochronic phenotype i
9 netic screen utilizing transgenic Drosophila second instar larvae expressing an actin, green fluoresc
10                            The transition to second instar larvae was rescued with importin alpha1, a
11 in gene targeting, die as small, immobilized second instar larvae with severely deformed musculature.
12         Hemizygous dm(4)/Y mutants arrest as second instar larvae, and fat body nuclei of dm(4)/Y mut
13 lies die around the transition from first to second instar larvae, and homozygous importin alpha3 mut
14                                   First- and second-instar larvae become infected when they ingest fo
15 ity of irradiated combination formulation on second-instar larvae Ephestia Kuehniella was 68.89%, whi
16 arval growth, and arrest during the first to second instar larval molt.
17 hila germline cystocytes generated either in second instar larval ovaries or in adults over-producing
18 loping larvae leaves the timing of first and second instar molts largely unchanged, but triples durat
19  to layers that are already present early in second instar nymphs.
20  and L mutually antagonize each other during second instar of larval development to restrict their fu
21                      dSREBP(-) larvae die at second instar owing to loss of dSREBP-mediated transcrip
22 (ex) driven by a heat pulse during the early second instar resulted in a severe phenotype that includ
23 e its expression is specifically elevated in second-instar wing discs during wing margin formation.
24                                       In the second instar, Wingless activates genes involved in prox

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