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1 ients, increased 18F-FDG uptake identified a second primary malignancy.
2 ears of follow-up and to explore the risk of second primary malignancies.
3        We investigated the long-term risk of second primary malignancy after chemotherapy for Hodgkin
4                                              Second primary malignancies and premature death are a co
5  associated with a small increase in risk of second primary malignancies and with increased risk of p
6 ce of all second primary malignancies, solid second primary malignancies, and haematological second p
7 ond primary malignancies, and haematological second primary malignancies, and were analysed by a one-
8                 Cumulative incidences of all second primary malignancies at 5 years were 6.9% (95% CI
9                  The cumulative incidence of second primary malignancies at 60 months after trial ent
10           The cumulative incidence rate of a second primary malignancy before disease progression was
11 second primary malignancy diagnosis, type of second primary malignancy, date of death or last contact
12 ce treatment, date of first relapse, date of second primary malignancy diagnosis, type of second prim
13  increased risk of developing haematological second primary malignancies, driven mainly by treatment
14       There is no emerging safety signal for second primary malignancies following VMP.
15 sion and overall survival and an increase in second primary malignancies for lenalidomide at a median
16 ene may be associated with increased risk of second primary malignancies in glioma patients.
17              Lenalidomide has been linked to second primary malignancies in myeloma.
18 dicating premalignant lesions and preventing second primary malignancies in patients cured of squamou
19 smani et al provide important information on second primary malignancies in patients treated with tha
20 e available data to compare the incidence of second primary malignancies in patients with and without
21 an increased awareness of the possibility of second primary malignancies in patients with thymoma.
22   Three haematological and five solid tumour second primary malignancies in the placebo group were in
23 %) haematological and nine (4%) solid tumour second primary malignancies in the placebo group.
24 ficantly associated with the occurrence of a second primary malignancy in the same patient.
25 ncrease in haematological adverse events and second primary malignancies, lenalidomide maintenance th
26 l cancer (n = 7), lack of follow-up (n = 4), second primary malignancy (n = 3), or chemotherapy befor
27 halan significantly increased haematological second primary malignancy risk versus melphalan alone (H
28 24]; p=0.76) did not increase haematological second primary malignancy risk versus melphalan alone.
29 of interest were cumulative incidence of all second primary malignancies, solid second primary malign
30                                              Second primary malignancies (SPM) were observed in 8 pat
31 ll carcinoma (HNSCC) are at elevated risk of second primary malignancies (SPM), most commonly of the
32 e rate (IR, events per 100 patient-years) of second primary malignancies (SPMs) was 3.62.
33            Evaluation for the development of second primary malignancies (SPMs) was conducted, and no
34 apy is associated with a higher incidence of second primary malignancies (SPMs), including both hemat
35 I 0.62-2.00]; p=0.72), and of haematological second primary malignancies were 3.1% (95% CI 1.9-4.3) a
36        Cumulative 5-year incidences of solid second primary malignancies were 3.8% (95% CI 2.7-4.9) i
37                                      Data on second primary malignancies were collected by individual
38 (8%) haematological and 14 (6%) solid tumour second primary malignancies were diagnosed after randomi
39                                           15 second primary malignancies were reported in 12 patients
40                  Eight patients developed 11 second primary malignancies with an excess frequency of

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