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1 y CD8+ T cells that are robustly recalled by secondary immunization.
2 mory, but has no effect when injected during secondary immunization.
3 nto effector-memory CD8+ T cells following a secondary immunization.
4 her tissues that was strongly enhanced after secondary immunization.
5 majority of human donors, particularly after secondary immunization.
6 zation but enhanced antibody responses after secondary immunization.
7 e to make alphaGal-specific antibodies after secondary immunization.
8  were analyzed by ELISA following primary or secondary immunization.
9 A response when injected only at the time of secondary immunization.
10 e isotype switching and those resulting from secondary immunization.
11  T lymphocytes isolated following primary or secondary immunization.
12          This population expanded further on secondary immunization.
13 -dependent PPS14-specific IgG response after secondary immunization.
14 B7-1 and B7-2 were observed upon primary and secondary immunizations.
15 e response was not observed after primary or secondary immunizations.
16 with MVA-Ag85A IMX313 after both primary and secondary immunizations.
17  Blockade of B7 costimulation at the time of secondary immunization also completely abrogated the est
18 nteers were rechallenged 23-42 weeks after a secondary immunization, and 4 were protected.
19 - and B-cell responses; there is no need for secondary immunization (boosting).
20 mulation was required only briefly after the secondary immunization compared with after the primary i
21  that was dependent on CD4(+) T cells during secondary immunization, indicating that Pn14 primes for
22      Pretreatment with anti-CD137 before the secondary immunization inhibited memory Ab responses.
23 (AFC) in the primary response, shortly after secondary immunization its memory cell progeny produce a
24 dy titers is apparent after both primary and secondary immunization of Aiolos(-)(/)(-) mice with a ra
25 y B cells and PCs failed to expand following secondary immunization of IL-21R.KO mice, and consequent
26     When weak boosting vectors were used for secondary immunization, pre-established CD8+ T cells wer
27  were lower, but they did show boosting upon secondary immunization to the levels achieved in the old
28 rimary kinetics and was highly boosted after secondary immunization, whereas the IgG anti-MCPS respon
29 ar and humoral responses were uncoupled upon secondary immunization, which was dramatically affected
30 Foxp3(+)CD4(+) T cells into mice followed by secondary immunization with collagen accelerated the ons
31                                     Further, secondary immunization with conjugate and S. agalactiae,
32 accine demonstrated a booster response after secondary immunization with either the MCPS or the conju
33  GBS-III, depletion of CD4(+) T cells during secondary immunization with MenC or another Gram-negativ
34                                    Following secondary immunization with PPS14-C3d, there was a marke
35 3d were nearly identical to those induced by secondary immunization with PPS14-OVA.
36 on of flow-sorted basophils into mice before secondary immunization with PspA significantly protected
37 i-TNF-alpha MAb injected only at the time of secondary immunization with R36A failed to alter the boo
38  regulatory events and Ag presentation after secondary immunization with strong and weak boosting vec
39 92F strain of L. monocytogenes followed by a secondary immunization with wild-type L. monocytogenes r
40 18F strain of L. monocytogenes followed by a secondary immunization with wild-type L. monocytogenes r
41 tibacteriophage antibodies after primary and secondary immunizations with evidence of amplification a
42 educed IgG responses after either primary or secondary immunizations with sheep red blood cells (SRBC

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