ライフサイエンスコーパス: secondary somatosensory cortex

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1 deactivation of sensory processing regions (secondary somatosensory cortex).

2 l operculum (i.e. the anatomical site of the secondary somatosensory cortex).

3 the bilateral insula, frontal operculum, and secondary somatosensory cortex.

4 s in the parietal operculum, the location of secondary somatosensory cortex.

5 roup 2) showed greater bilateral primary and secondary somatosensory cortex activation with greater d

6 e further demonstrate the specificity of the secondary somatosensory cortex and dorsal posterior insu

7 (P1) was recorded in parallel in the primary/secondary somatosensory cortex and posterior insula (app

8 e major targets are granular insular cortex, secondary somatosensory cortex and several cortical area

9 cupuncture induced greater activation at the secondary somatosensory cortex and stronger functional c

10 ion of ERP amplitude was source localized to secondary somatosensory cortex, and attributed to feedfo

11 many regions including primary motor cortex, secondary somatosensory cortex, anterior insula, and the

12 ral to stimulation by PND 6, followed by the secondary somatosensory cortex at PND 7.

13 or cingulate cortex (ACC), left insula, left secondary somatosensory cortex, bilateral thalamus, and

14 o feedforward processing between primary and secondary somatosensory cortex by means of dynamic causa

15 in fMRI activity in the premotor cortex and secondary somatosensory cortex contralateral to the affe

16 L2 neurons projecting to motor and secondary somatosensory cortex differed in whisker tunin

17 ort the sensory components of physical pain (secondary somatosensory cortex; dorsal posterior insula)

18 In controls, a region of secondary somatosensory cortex exhibited attenuated acti

19 primary somatosensory cortex; in frontal and secondary somatosensory cortex, Fos expression was lower

20 the medial insula, medial cingulate cortex, secondary somatosensory cortex, frontal areas, and cereb

21 f the feedforward connection from primary to secondary somatosensory cortex (>80 ms).

22 trate a double dissociation in the patient's secondary somatosensory cortex (increased responses to a

23 g induces robust bilateral activation of the secondary somatosensory cortex, insular cortex, prefront

24 recorded responses of single neurons in the secondary somatosensory cortex of monkeys suggest how th

25 Single-unit recordings were made in secondary somatosensory cortex of three non-human primat

26 There was no effect of cTBS over the secondary somatosensory cortex on STDT, although it redu

27 Additional brain regions in bilateral secondary somatosensory cortex, premotor cortex, primary

28 ations that are significantly delayed across secondary somatosensory cortex, premotor, and motor area

29 Furthermore, failure to attenuate secondary somatosensory cortex processing was predicted

30 lternative sites of stimulation, such as the secondary somatosensory cortex (rather than primary moto

31 The primary and secondary somatosensory cortex (S1 and S2), anterior cin

32 details of the location and organization of secondary somatosensory cortex (S2) are reported, and ev

33 ions of biocytin into head and limb areas of secondary somatosensory cortex (S2) produced heavy label

34 Here we report that higher-order area 1 and secondary somatosensory cortex (S2) underwent similar sp

35 n area 3b; feedback connections from area 1, secondary somatosensory cortex (S2), parietal ventral ar

36 om POm to all layers of S1 and to layer 4 of secondary somatosensory cortex (S2).

37 ing to primary somatosensory cortex (S1) and secondary somatosensory cortex (S2).

38 ect corticocortical pathway was interrupted, secondary somatosensory cortex showed robust activity in

39 ysiological studies suggest that primary and secondary somatosensory cortex (SI and SII, respectively

40 ions, i.e., primary somatosensory cortex SI, secondary somatosensory cortex SII, posterior parietal c

41 ynchronous firing of pairs of neurons in the secondary somatosensory cortex (SII) of three monkeys tr

42 splayed significant increases in ipsilateral secondary somatosensory cortex (SII), although the magni

43 the white matter of the ipsilesional SI and secondary somatosensory cortex (SII), and in the contral

44 bilateral activation was demonstrated in the secondary somatosensory cortex (SII), indicating a bilat

45 mponent, produced little or no activation in secondary somatosensory cortex (SII), whereas light brus

46 mus, the posterior and anterior insulae, the secondary somatosensory cortex, the anterior cingulate c

47 Activity in the posterior insula/secondary somatosensory cortex, the sensorimotor cortex

48 cally inhibiting the thalamus, activation in secondary somatosensory cortex was eliminated, with a su

49 roelectrodes from the hand region of macaque secondary somatosensory cortex while vibrotactile stimul

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