ライフサイエンスコーパス: serum immunoglobulin

1 s of peripheral mature B cells and decreased serum immunoglobulin.

2 east or by immobilized protein A/G to remove serum immunoglobulin.

3 me activated in the presence of neutralizing serum immunoglobulin.

4 mbers showed progressive loss of B cells and serum immunoglobulins.

5 nd central memory T cells and an increase in serum immunoglobulins.

6 vestigated the prevalence of abnormally high serum immunoglobulin A (IgA) and IgG anti-gliadin antibo

7 nses; all volunteers who received H10407 had serum immunoglobulin A (IgA) and IgG responses, and all

8 Serum immunoglobulin A (IgA) and immunoglobulin G (IgG),

9 for RV1 and RV5 to correlate anti-rotavirus serum immunoglobulin A (IgA) antibody titers vs efficacy

10 Half of the volunteers mounted a positive serum immunoglobulin A (IgA) response to S. flexneri lip

11 fetuin, bovine submaxillary gland mucin, and serum immunoglobulin A (IgA).

12 Elevated serum immunoglobulin A and immunoglobulin G titers were

13 s female family members showed only elevated serum immunoglobulin A levels.

14 H. pylori serum immunoglobulin A, IgG, and IgG subclass responses

15 trifluoroacetylated protein adduct-specific serum immunoglobulin and hepatotoxicity were reduced.

16 oved B-cell function with increases in total serum immunoglobulin and increased splenic mitogen respo

17 inflammation and hyperresponsiveness (AHR), serum immunoglobulin and splenic T cells were assessed.

18 erved as lesser allergic phenotypes, reduced serum immunoglobulins and allergic mediators, lower mast

19 methods included flow cytometry, analysis of serum immunoglobulins and autoantibodies, lymphocyte sti

20 Ptpn6(f/f);CD19-cre mice exhibited elevated serum immunoglobulins and impaired antibody responses to

21 tive IgE, which is the least abundant of the serum immunoglobulins and occurs at subnanomolar levels,

22 scores during the last 8 weeks of treatment, serum immunoglobulins and safety assessments.

23 Allergen-specific serum immunoglobulins and total immunoglobulin E in diff

24 lasma cells within the bone marrow, elevated serum immunoglobulin, and osteolytic bone disease.

25 in early B-cell development, near absence of serum immunoglobulin, and recurrent bacterial infections

26 Eosinophil recruitment, cytokine production, serum immunoglobulins, and airway histology were analyze

27 sponse (ASR), anaphylaxis, body temperature, serum immunoglobulins, and mouse mast cell protease-1 (m

28 ignificant reductions in lymphocyte count or serum immunoglobulin, anticardiolipin antibody, or rubel

29 In 10% to 20% of patients, the serum immunoglobulins are higher than expected or the on

30 d activation parameters are normal; however, serum immunoglobulins are increased and kidney function

31 iency associated with partial persistence of serum immunoglobulin arising from a missense mutation in

32 nificantly reduced titers of IgG2a and IgG2b serum immunoglobulins as well as autoantibodies, but mai

33 solated using the device was consistent with serum immunoglobulin assays that are commonly used in MM

34 rrow transplant (BMT) make normal amounts of serum immunoglobulin but are deficient in generating spe

35 Ku70-deficient mice lacked mature B cells or serum immunoglobulin but, unexpectedly, reproducibly dev

36 als produced significantly less IgG1 and IgE serum immunoglobulins, but maintained comparable levels

37 than did those of their corresponding total serum immunoglobulin classes.

38 d a normal immune cell repertoire, unchanged serum immunoglobulin concentrations and an intact immune

39 While total serum immunoglobulin concentrations decline following CT

40 ations depended on DCs, in contrast to total serum immunoglobulin concentrations, suggesting an effec

41 B and T cell zones and resulted in elevated serum immunoglobulin concentrations.

42 sistent with eradication of B-lineage cells, serum immunoglobulins decreased to very low levels after

43 ogeneous and miniscule fraction of the total serum immunoglobulin displaying identical properties oth

44 sponses, characterized by increases in total serum immunoglobulin E (IgE) and specific serum IgG1 lev

45 While serum immunoglobulin E (IgE) concentration has been show

46 Elevated serum Immunoglobulin E (IgE) levels and increased airway

47 This study examined the association between serum immunoglobulin E (IgE) levels for a panel of commo

48 Total serum immunoglobulin E (IgE) levels were significantly l

49 al hyperresponsiveness (BHR), elevated total serum immunoglobulin E (IgE) levels, and skin tests posi

50 dies of intermediate phenotypes, one each on serum immunoglobulin E (IgE) levels, blood eosinophil co

51 Total serum immunoglobulin E (IgE) levels, for example, show s

52 ent of CD4-CD8- peripheral T cells, elevated serum immunoglobulin E (IgE), and possible pulmonary inf

53 cts on markers of Th2 inflammation, reducing serum immunoglobulin E (IgE), chemokine ligands 13 and 1

54 However, the level of a systemic Th2 marker, serum immunoglobulin E (IgE), correlated significantly w

55 ypersensitivity (DTH) to SEA; high levels of serum immunoglobulin E (IgE); a strong T2 cytokine pheno

56 Serum immunoglobulin E against eight common inhalant and

57 d with asthma phenotypes, such as high total serum immunoglobulin E and bronchial hyperresponsiveness

58 Total serum immunoglobulin E and FEV1 predicted levels were no

59 ti-TNF-alpha-treated mice exhibited elevated serum immunoglobulin E and inhibition of the anticryptoc

60 levels at year 1 and repeated assessments of serum immunoglobulin E antibodies (year 1, 4.5, 6), atop

61 the allergen specificity, allergen-specific serum immunoglobulin E concentration, or individual labo

62 Allergen-specific serum immunoglobulin E concentrations ranged from 0.1 to

63 Allergen-specific serum immunoglobulin E detection and quantification have

64 Allergen-specific serum immunoglobulin E determination with the fluoroimmu

65 nome-scan data set and incorporate the total serum immunoglobulin E level in the analysis.

66 Serum immunoglobulin E levels were increased in IL-13Ral

67 eukin-4 production by NKT cells, to increase serum immunoglobulin E levels, and to promote the genera

68 cytokine production, eosinophil influx, and serum immunoglobulin E levels.

69 so had reduced T2 cytokine production and no serum immunoglobulin E production.

70 he accumulation of eosinophils and levels of serum immunoglobulin E were increased in NFAT1 -/- mice.

71 ormed with asthma, AHR, lung function, total serum immunoglobulin E, and blood eosinophil levels.

72 es on swIg(+) cells until they disappear and serum immunoglobulin falls to a low level, in which case

73 Levels of total serum immunoglobulins fell, although the mean values eac

74 The serum immunoglobulin FLC ratio is an important additiona

75 An abnormal serum immunoglobulin free light chain (FLC) ratio at dia

76 one marrow plasmacytosis, extremely abnormal serum immunoglobulin free light chain ratio, and multipl

77 C-reactive protein, human interleukin-6, and serum immunoglobulin free light chains decreased with th

78 Natural serum immunoglobulin from mice was able to bind to the b

79 n ovarian cancer phage display library using serum immunoglobulins from an ovarian cancer patient as

80 s resulted in the induction of FimA-specific serum (immunoglobulin G [IgG] and IgA) and salivary (IgA

81 There was a slight increase in serum immunoglobulin G (IgG) against the MS11 strain and

82 Serologic markers such as serum immunoglobulin G (IgG) and antinuclear antibody le

83 although FspA1 induced the highest levels of serum immunoglobulin G (IgG) and fecal IgA.

84 ity to S. typhimurium severely depressed the serum immunoglobulin G (IgG) and IgA anti-FrgC response

85 -CAT construct induced significant levels of serum immunoglobulin G (IgG) and IgA antibody to both pa

86 Recall responses were seen in serum immunoglobulin G (IgG) and IgA following boosting

87 with Invaplex was found to enhance anti-OVA serum immunoglobulin G (IgG) and IgA responses and induc

88 oximately 2-week intervals and evaluated for serum immunoglobulin G (IgG) and IgG subclass and for sa

89 Despite production of serum immunoglobulin G (IgG) and IgM (median titers of 1

90 throughout the experiment and evaluated for serum immunoglobulin G (IgG) and IgM and salivary IgA an

91 In mice i.n. challenged with H10407, serum immunoglobulin G (IgG) and IgM antibodies were mea

92 or no bacteria in the liver and lungs, high serum immunoglobulin G (IgG) and IgM antibody titers, an

93 lpha stimulated strong antigen (Ag)-specific serum immunoglobulin G (IgG) and IgM responses, while MI

94 ion of mice with CVLPs induced gp41-specific serum immunoglobulin G (IgG) and intestinal secretory Ig

95 luenza virus showed significant increases in serum immunoglobulin G (IgG) and mucosal IgA antibodies

96 lones were found to stimulate high levels of serum immunoglobulin G (IgG) and mucosal IgA antibodies

97 oxin (CT) A2 and B subunits (CTA2/B) develop serum immunoglobulin G (IgG) and mucosal IgA antibody re

98 hesizing either PspA fusion protein elicited serum immunoglobulin G (IgG) and mucosal IgA responses a

99 The levels of antigen-specific serum immunoglobulin G (IgG) and mucosal IgA were higher

100 cific gamma interferon-producing T cells and serum immunoglobulin G (IgG) and mucosal IgA.

101 hK63 or LThR72 exhibited high titers of both serum immunoglobulin G (IgG) and mucosal secretory IgA a

102 ed animals demonstrated significantly higher serum immunoglobulin G (IgG) and salivary IgA antibody l

103 Both peptides readily induced serum immunoglobulin G (IgG) and salivary IgA antipeptid

104 astric tissue, histology, and measurement of serum immunoglobulin G (IgG) and salivary IgA.

105 The specificity of anti-P1 serum immunoglobulin G (IgG) and secretory IgA antibodie

106 enuation elicited higher Salmonella-specific serum immunoglobulin G (IgG) and/or mucosal secretory-Ig

107 later, significant reductions were seen for serum immunoglobulin G (IgG) antibodies at week 14 and f

108 Serum immunoglobulin G (IgG) antibodies have been implic

109 recombinant proteins were reacted with human serum immunoglobulin G (IgG) antibodies in Western immun

110 We investigated the ability of serum immunoglobulin G (IgG) antibodies to periodontal b

111 Low maternally derived serum immunoglobulin G (IgG) antibodies to Streptococcus

112 ta provide evidence that a critical level of serum immunoglobulin G (IgG) antibodies to the surface p

113 were found to be immunogenic since elevated serum immunoglobulin G (IgG) antibodies without a specif

114 Oral and i.p. vaccines elicited O11-specific serum immunoglobulin G (IgG) antibodies, but IgA was obs

115 isms are found and the induction of specific serum immunoglobulin G (IgG) antibodies, we examined the

116 ay was used to measure the seroprevalence of serum immunoglobulin G (IgG) antibody among NHANES parti

117 We determined pneumococcal serum immunoglobulin G (IgG) antibody concentrations aga

118 ria during the study interval, and increased serum immunoglobulin G (IgG) antibody levels substantiat

119 nce of P. gingivalis in dental plaque and of serum immunoglobulin G (IgG) antibody levels to P. gingi

120 GFPuv-specific serum immunoglobulin G (IgG) antibody responses were ana

121 oPn produced greater titers of MoPn-specific serum immunoglobulin G (IgG) antibody than mice that rec

122 zed by enzyme-linked immunosorbent assay for serum immunoglobulin G (IgG) antibody to Aggregatibacter

123 Levels of serum immunoglobulin G (IgG) antibody to GTF or CAT in t

124 Serum immunoglobulin G (IgG) antibody, induced by subcut

125 rticular) demonstrated significantly reduced serum immunoglobulin G (IgG) antibody, salivary IgA anti

126 train RB51 (SRB51) produced small amounts of serum immunoglobulin G (IgG) but no IgM antibody to smoo

127 Ribi had the highest levels of PorB-specific serum immunoglobulin G (IgG) by enzyme-linked immunosorb

128 New serum immunoglobulin G (IgG) detected by whole-cell enzy

129 mean titer (GMTs) and 4-fold rise of anti-Vi serum immunoglobulin G (IgG) enzyme-linked immunosorbent

130 ntigenic MDI-albumin products, as defined by serum immunoglobulin G (IgG) from MDI-exposed workers, w

131 Aberrant serum immunoglobulin G (IgG) glycosylation and its immun

132 immunosorbent assay to measure beta-specific serum immunoglobulin G (IgG) levels and used it to compa

133 Significant anti-GLU serum immunoglobulin G (IgG) levels were seen in groups

134 Smoking is also known to reduce serum immunoglobulin G (IgG) levels.

135 haride (PS) conjugate vaccines may prime for serum immunoglobulin G (IgG) memory responses to meningo

136 transplantations indicate that low levels of serum immunoglobulin G (IgG) negatively impact outcomes.

137 Overall, 23% of patients developed either a serum immunoglobulin G (IgG) response (9%) or sputum IgA

138 . denticola induced a significant (400-fold) serum immunoglobulin G (IgG) response compared to that i

139 Anamnestic anti-SEB serum immunoglobulin G (IgG) responses were elicited in

140 ized with replication-defective HSV, durable serum immunoglobulin G (IgG) responses were elicited.

141 e the long-term course of H. pylori-specific serum immunoglobulin G (IgG) responses with respect to s

142 were effective in eliciting antigen-specific serum immunoglobulin G (IgG) responses, and these respon

143 s evokes large increases in antigen-specific serum immunoglobulin G (IgG) responses.

144 fold increase over the prechallenge anti-SMV serum immunoglobulin G (IgG) titer, mostly subclass IgG1

145 were consistent with significantly declined serum immunoglobulin G (IgG) titers for HHV-6 of MS pati

146 ckerboard analyses of eight plaque bacteria, serum immunoglobulin G (IgG) titers to 17 bacteria, and

147 Serum immunoglobulin G (IgG) titers to 28 pneumococcal p

148 B) toxoid in saline elicited higher anti-SEB serum immunoglobulin G (IgG) titers when the toxoid was

149 Binding activity of patient serum immunoglobulin G (IgG) to HupB did not correlate w

150 colonization, 9 of 40 patients developed new serum immunoglobulin G (IgG) to OMP CD, as measured by e

151 ollowing infection and who had developed new serum immunoglobulin G (IgG) to their infecting strain w

152 eleased from bovine fetuin, polyclonal human serum immunoglobulin G (IgG), and human alpha1-acid glyc

153 he most effective means of immunization: the serum immunoglobulin G (IgG), IgA, and IgM anti-fragment

154 We measured serum immunoglobulin G (IgG), IgM, and IgA antibodies to

155 increased class switched memory B cells and serum immunoglobulin G (IgG).

156 adermal comparative tuberculin skin test and serum immunoglobulin G [IgG] responses) against a range

157 vated during primary infections (acute-phase serum immunoglobulin G [IgG] titers, <25), compared with

158 nses (HI, antibody-secreting cell [ASC], and serum immunoglobulin G [IgG]) in 15 LRs and 25 control H

159 minent specific anti-CtxB responses in vivo (serum immunoglobulin G [IgG], P </= 0.05; serum IgA, P <

160 ly correlated with the level of prechallenge serum immunoglobulin G against the O127 lipopolysacchari

161 l aplasia, several vaccine/pathogen-specific serum immunoglobulin G and A (IgG and IgA) titers remain

162 Neutralizing antibody responses, serum immunoglobulin G and A, and nasal wash specimen im

163 or, CVD 915(pTETlpp) elicited high titers of serum immunoglobulin G anti-fragment C.

164 Only conjugates with O acetyls elicited serum immunoglobulin G anti-LPS with bactericidal activi

165 ith the level of protection correlating with serum immunoglobulin G anti-protective antigen titers.

166 l immunoglobulin A antiamebic antibodies and serum immunoglobulin G antiamebic antibodies.

167 VRP resulted in high levels of DENV-specific serum immunoglobulin G antibodies and significant titers

168 Although serum immunoglobulin G antibodies are significantly high

169 ll-length block 2 antigens showed that human serum immunoglobulin G antibodies induced by infection c

170 orbent assays (ELISAs) for quantification of serum immunoglobulin G antibodies specific for N. mening

171 uced specific secretory immunoglobulin A and serum immunoglobulin G antibodies that persisted at subs

172 concentrations and neutralizing activity of serum immunoglobulin G antibodies to the RSV prefusion (

173 tion, as well as the specificity, of anti-P1 serum immunoglobulin G antibodies were demonstrated in g

174 It was shown that serum immunoglobulin G antibodies were produced against

175 d formation, an impaired ability to generate serum immunoglobulin G antibodies, and significant inhib

176 treated patients with LA have high levels of serum immunoglobulin G antibodies, and sometimes low lev

177 cator of inflammation), or edentulism and 2) serum immunoglobulin G antibody response to Aggregatibac

178 e also developed sustained anti-H. hepaticus serum immunoglobulin G antibody responses and elevated a

179 Less marked, but significant, anti-SREHP serum immunoglobulin G antibody responses were also indu

180 d animalls developed sustained anti-H. felis serum immunoglobulin G antibody responses.

181 PI) owing to the presence of highly elevated serum immunoglobulin G antibody titers with a high avidi

182 cant levels of salivary immunoglobulin A and serum immunoglobulin G antibody to the respective inject

183 A serum immunoglobulin G autoantibody (NMO-IgG) serves as

184 All conjugates elicited high levels of serum immunoglobulin G both to the polysaccharides and t

185 ortal vein diameter, splenomegaly, increased serum immunoglobulin G level, and increasing number of a

186 tly improved liver histology (P = 0.005) and serum immunoglobulin G levels (P = 0.0002).

187 We compared serum immunoglobulin G levels and toxin-neutralizing ant

188 The serum immunoglobulin G levels correlated with protection

189 P. gingivalis-specific serum immunoglobulin G levels were significantly elevate

190 , with a 44% reduction in intracardiomyocyte serum immunoglobulin G localization in het-treated-8 mic

191 cination geometric mean titre of LT-specific serum immunoglobulin G of 3400.29, compared with 315.41

192 Serum immunoglobulin G profiling by proteome microarray

193 Serum immunoglobulin G reactivity to Hsp10 and Hsp60 ant

194 the naturally infected SCID/NCr mice had no serum immunoglobulin G response against H. hepaticus.

195 vaccine doses elicited a strong PA-specific serum immunoglobulin G response with a geometric mean ti

196 Only LAM-reactive serum immunoglobulin G responses were significantly incr

197 ufficient to decrease P. gingivalis-specific serum immunoglobulin G responses, but lower antibody tit

198 osted 4 weeks later induced higher levels of serum immunoglobulin G specific for PspA and for outer m

199 Serum immunoglobulin G subclass levels and antibody tite

200 The serum immunoglobulin G subclass profiles were indicative

201 he vhs(-)/ICP8(-) mutant showed prechallenge serum immunoglobulin G titers comparable to those immuni

202 a clinical oral-health examination, and had serum immunoglobulin G titers measured against 19 period

203 of variants with ChoP correlates with higher serum immunoglobulin G titers to LPS containing this str

204 Vi stimulates serum immunoglobulin G Vi antibodies, whereas Ty21a, whi

205 A low serum immunoglobulin G was associated with number of inf

206 Subgingival P. endodontalis levels and serum immunoglobulin G were associated with a higher EL

207 e deficiency, characterized by low levels of serum immunoglobulin G, A, and/or M with loss of antibod

208 Antral gastritis, anti-H. pylori serum immunoglobulin G, and atrophy all increased, but w

209 ut adjuvant stimulated induction of specific serum immunoglobulin G, whereas antigen alone or admixed

210 proliferation in the presence of respective serum immunoglobulin G.

211 asma could not be duplicated by pooled human serum, immunoglobulin G, or fibrinogen, whether used sep

212 s well as the production of antigen-specific serum immunoglobulin G1 (IgG1) and IgG2a antibodies.

213 wever, there was an increase in the level of serum immunoglobulin G1 (IgG1) and IgG2b as well as a mi

214 fected with wild-type H. hepaticus developed serum immunoglobulin G1 (IgG1) and IgG2c responses again

215 Also, virus-specific serum immunoglobulin G1 (IgG1) levels were greatly reduc

216 i antibody levels in serum showed a dominant serum immunoglobulin G1 (IgG1) response in immunized C57

217 Oral F1-V mice had higher prechallenge serum immunoglobulin G1 (IgG1) titers than s.c. F1-V mic

218 sponse, including the levels of RSV-specific serum immunoglobulin G1 (IgG1), IgG2a, IgA, and total Ig

219 onstrated an increased level of CVB3-binding serum immunoglobulin G1 in mice inoculated with CVB3-PL2

220 le, VirB9, VirB10, and CTP are recognized by serum immunoglobulin G2 (IgG2) and stimulate memory T-ly

221 Patients with LJP have elevated levels of serum immunoglobulin G2 (IgG2), and this is most strikin

222 e demonstrated between the levels of anti-P1 serum immunoglobulin G2a (IgG2a) and IgG2b, but not of I

223 e vector and passenger antigen, resulting in serum immunoglobulin G2a (IgG2a) and modest mucosal IgA

224 mRNAs; high levels of antibody, particularly serum immunoglobulin G2a (IgG2a) and respiratory IgA; an

225 hat oral reovirus infection elicits specific serum immunoglobulin G2a (IgG2a), while parenteral reovi

226 by the production of high titers of specific serum immunoglobulin G2a antibody and the production of

227 n the dermis and subcutaneous fat, increased serum immunoglobulin G2a levels, and lymphadenopathy ass

228 al immunization with LT-R72 induced a potent serum immunoglobulin G2a response, indicating that this

229 he cecum, as well as elevated Th1-associated serum immunoglobulin G2a(b) compared to infection of B6

230 Elevated serum immunoglobulin G4 (sIgG4) is a feature of autoimmu

231 ry sclerosing cholangitis but with increased serum immunoglobulin G4 and infiltrating immunoglobulin

232 r predilection and is associated with normal serum immunoglobulin G4 levels.

233 o rotavirus infection, we analyzed levels of serum immunoglobulin (Ig) A and IgG antibodies to variou

234 Significant serum immunoglobulin (Ig) A and IgG antibody responses t

235 ek 6 (predicted peak of pollen) to determine serum immunoglobulin (Ig) E concentrations and Treg perc

236 Serum immunoglobulin (Ig) E concentrations associated wi

237 red orally without adjuvant, and they elicit serum immunoglobulin (Ig) G and intestinal IgA responses

238 pithelial antigen that reacted strongly with serum immunoglobulin (Ig) G from the mouse model of BA w

239 Notably, serum immunoglobulin (Ig) isotypes and kidney Ig/C3 depo

240 ever, in the absence of constant reexposure, serum immunoglobulin (Ig) levels rapidly decline and ful

241 Consistent with B-lymphocyte increases, serum immunoglobulin (Ig) M, IgG, and IgE were significa

242 fective B-cell function characterized by low serum immunoglobulin (Ig) M, low IgM antibody production

243 We evaluated serum immunoglobulin (Ig) proteins as predictors of NHL

244 ) cell cytotoxic activity and were devoid of serum immunoglobulin (Ig) throughout a 37-week lifespan.

245 f antibody effector functions by retargeting serum immunoglobulin (Ig).

246 ymic stromal lymphopoietin, IL-5 and IL-13), serum immunoglobulin (Ig)E and airway hyper-responsivene

247 In both cases, total serum immunoglobulin (Ig)E level was >1000 IU/mL and fun

248 re examined pulmonary pathophysiological and serum immunoglobulin (Ig)E responses in mice of 12 inbre

249 We hypothesized that serum immunoglobulin (Ig)G antibody levels against BspA

250 No baseline serum immunoglobulin (Ig)G antibody or splenic CD4+ T-ce

251 ing checkerboard DNA-DNA hybridizations, and serum immunoglobulin (Ig)G levels were measured against

252 ity to carriage did not correlate with total serum immunoglobulin (Ig)G to the homotypic capsular pol

253 pression, mutant mice had elevated levels of serum immunoglobulin (Ig)G1, IgG2b, and IgE and produced

254 Deficiency of serum immunoglobulin (Ig)M is associated with the develo

255 CAR bacillus-specific serum immunoglobulins (immunoglobulin M [IgM], IgG1, IgG

256 eficient in complement components C3, C4, or serum immunoglobulin in a hindlimb model of ischemia.

257 long-lived plasma cells, which produce most serum immunoglobulin, is central to humoral immunity.

258 re was widespread variation in the levels of serum immunoglobulin isotypes as well as in the percenta

259 o, and baseline levels of the Th2-controlled serum immunoglobulin isotypes, IgE and IgG1, were also s

260 ith baseline levels, we noted an increase in serum immunoglobulin levels across all classes, and a re

261 Serum immunoglobulin levels and lymphocyte phenotypes an

262 Despite this, patients had normal serum immunoglobulin levels and normal antigen-specific

263 Despite normal B-lymphocyte numbers, serum immunoglobulin levels decreased with age.

264 Previous studies examined the serum immunoglobulin levels in relation to coronary arte

265 Serum immunoglobulin levels were also elevated in HIV-in

266 Serum immunoglobulin levels were also markedly reduced i

267 ; CD5(+) peritoneal B cells were absent, and serum immunoglobulin levels were markedly reduced.

268 Serum immunoglobulin levels were measured by enzyme-link

269 ) is characterized by variable reductions in serum immunoglobulin levels which cause most ICF patient

270 rently normal lymphocyte development, normal serum immunoglobulin levels, and the capacity to respond

271 lts here confirm that smoking has effects on serum immunoglobulin levels, but the effects were both r

272 in viremic individuals, and correlated with serum immunoglobulin levels, particularly IgG.

273 not associated with significant decreases in serum immunoglobulin levels.

274 ma cells in tonsils appeared normal, as were serum immunoglobulin levels.

275 flow cytometry of blood B-cell subsets, and serum immunoglobulin levels.

276 cell numbers; immune complex, complement, or serum immunoglobulin levels; delayed type hypersensitivi

277 was associated with higher brain CFU, lower serum immunoglobulin M (IgM) and IgG responses to glucur

278 cryptocococcal pulmonary infection elicited serum immunoglobulin M (IgM) and IgG to the capsular pol

279 of peritoneal B1 B cells, and correction of serum immunoglobulin M (IgM) and IgG(3) levels.

280 Interestingly, anti-Bordetella serum immunoglobulin M (IgM) levels were significantly l

281 (+)CD4(-) T cells, and decreases in elevated serum immunoglobulin M and inflammatory markers includin

282 production, comprising approximately 90% of serum immunoglobulin M in ATA micro kappa mice.

283 se with nonsense CXCR4 mutations have higher serum immunoglobulin M levels and incidence of symptomat

284 88 show lower bone marrow disease burden and serum immunoglobulin M levels but show an increased risk

285 vement declined from 55% to 10% (P = .0004), serum immunoglobulin M levels declined from 4,830 to 1,1

286 ysts had no detectable mature T and B cells, serum immunoglobulin M, or even Thy-1(+) and B220(+) pre

287 Passive transfer of normal mouse serum, immunoglobulin M (IgM) from normal mouse serum, o

288 higher bone marrow (BM) disease involvement, serum immunoglobulin-M levels, and symptomatic disease r

289 CXCR4(WILDTYPE (WT)) had intermediate BM and serum immunoglobulin-M levels; those with MYD88(WT)CXCR4

290 luble and membrane-bound TGF-beta as well as serum immunoglobulin production are high in these mice.

291 ural killer cells, lymphocyte phenotype, and serum immunoglobulin subclass levels were evaluated.

292 scue of mature B cells with normal levels of serum immunoglobulins, together with complete rescue of

293 enzyme-linked immunosorbent assay, specific serum immunoglobulin was detected as early as 134 days p

294 Serum immunoglobulin was purified by protein A/G chromat

295 Furthermore, mice deficient in serum immunoglobulin were equally protected and this pro

296 ically by inadequate quantity and quality of serum immunoglobulins with increased susceptibility to i