ライフサイエンスコーパス: serum triglyceride

1 or metolazone increased the concentration of serum triglyceride.

2 correlated with a postprandial elevation of serum triglycerides.

3 correlated with a postprandial elevation in serum triglycerides.

4 tabolizing enzymes and resultant decrease of serum triglycerides.

5 rance, which resulted in decreased levels of serum triglycerides.

6 atitis even in the absence of a reduction in serum triglycerides.

7 hanged by adjustment for body mass index and serum triglycerides.

8 inefficiency, and improved fetal weights and serum triglycerides.

9 g from rheumatologic conditions and reducing serum triglycerides.

10 Os) have anti-inflammatory effects and lower serum triglycerides.

11 tein, fasting glucose-insulin metabolism, or serum triglycerides.

12 f glucokinase regulatory protein (GCKR) with serum triglycerides.

13 gh blood pressure (1.24; 1.04-1.48) and high serum triglycerides (1.18; 1.00-1.39), with a trend of i

14 into 5 groups according to strata of fasting serum triglycerides: (1) low-normal triglycerides (<100

15 Significant reductions were seen in mean serum triglycerides (1206-->226 mg/dL, P = .002), glucos

16 ed by dyslipidemic (>30% elevated, P < 0.05) serum triglycerides (139 mg/dl), very-LDLs (27.8 mg/dl),

17 Changes in serum triglycerides (+2.1 +/- 63.0 vs. +38.9 +/- 37.5 mg

18 elihood ratio test suggested interactions on serum triglycerides (4 SNP - SNP pairs), LDL cholesterol

19 detected by magnetic resonance imaging), and serum triglycerides (-51%), improved glucose tolerance,

20 r small VLDL), which produced an increase in serum triglycerides; a decrease in LDL size as a result

21 MA-IR, body mass index, waist circumference, serum triglycerides, aminotransferase level, and histolo

22 nuated triglyceride secretion, and decreased serum triglyceride and alanine aminotransaminase levels.

23 Serum SLs correlated significantly with serum triglyceride and cholesterol levels as well as wit

24 Argatroban reduced serum triglyceride and cholesterol levels in mice fed a

25 ding induced hepatic steatosis and increased serum triglyceride and cholesterol levels in the KO mice

26 abolic parameters, UDCA-LPE reduced elevated serum triglyceride and cholesterol values in HFD mice.

27 causes insulin-resistant diabetes, elevated serum triglyceride and fatty acid levels, and massive tr

28 oses of WY14,643 that were tested normalized serum triglyceride and fatty acid levels.

29 ation gene expression in liver and increased serum triglyceride and FFA levels.

30 The fish diet resulted in decreased serum triglyceride and increased HDL-cholesterol concent

31 Serum triglyceride and LDL-cholesterol concentrations we

32 Elevated serum triglyceride and low HDL-cholesterol concentration

33 Guggulipid treatment in rats lowered serum triglyceride and raised serum high density lipopro

34 as associated with a dramatic improvement in serum triglyceride and VLDL concentrations, a significan

35 We concurrently tested serum triglycerides and aminotransferases and estimation

36 was observed between postprandial changes in serum triglycerides and FMD (r = -0.47, p < 0.05).

37 diabetes and FCHL, both predisposing to high serum triglycerides and glucose intolerance, we tested t

38 rol in type 2 diabetics results in increased serum triglycerides and has a negative influence on all

39 Elevated serum triglycerides and low high-density lipoprotein (HD

40 n-3 (omega-3) fatty acid supplementation on serum triglycerides and markers of insulin sensitivity w

41 SB-induced effects were mainly on serum triglycerides and very-low-density lipoprotein (VL

42 ped a complication other than an increase in serum triglycerides and/or cholesterol.

43 erences in the concentrations of hemoglobin, serum triglyceride, and serum cholesterol were found bet

44 ight ratio, alanine transaminase, uric acid, serum triglycerides, and blood pressure.

45 ificant after adjustment for exercise level, serum triglycerides, and BMI (P = 0.02) but was no longe

46 These mice display elevated fat mass, serum triglycerides, and free fatty acids, but blood glu

47 y, obesity, type 2 diabetes mellitus, raised serum triglycerides, and metabolic syndrome.

48 are frequently used strategies for reducing serum triglycerides, and, yet, there is no information r

49 ive correlation of antibody levels and total serum triglycerides, apolipoprotein B, and apolipoprotei

50 serum HDL-cholesterol and a 71% reduction in serum triglycerides at the highest dose administered (10

51 glyceride increases manyfold but circulating serum triglyceride barely fluctuates.

52 2.03]; I(2)=78%), and when the difference in serum triglycerides between the two interventions at fol

53 e to Wy-14,643 effects on beta-oxidation and serum triglycerides but resistant to hepatocellular prol

54 iation between serum PCSK9 concentration and serum triglyceride, but care has to be taken in interpre

55 All five meals significantly raised serum triglycerides, but did not change other lipoprotei

56 Serum triglycerides, but not blood glucose were lower in

57 l/liter (0.258, 0.355) [mean (95% C.I.)] and serum triglyceride by 0.164 mmol/liter (0.12, 0.209) alt

58 Concentration of serum triglycerides, cholesterol, insulin and leptin wer

59 ion of hepatic apolipoprotein C-III mRNA and serum triglycerides compared with untreated controls.

60 tudies have reported on associations between serum triglyceride concentrations and the risk of corona

61 sterol concentrations increased slightly and serum triglyceride concentrations decreased slightly in

62 eated men had significantly higher follow-up serum triglyceride concentrations over baseline than did

63 Liver injury markers were measured in serum, triglyceride content and endocytosis (binding and

64 There was a significant decrease in serum triglycerides (decrease of 19%) in the OI group.

65 ty acid supplementation dramatically reduced serum triglycerides, decreased arachidonic acid in the p

66 entrations (four patients), and increases in serum triglycerides (eight patients) and aspartate amino

67 in clozapine-treated patients; screening for serum triglyceride elevations may be warranted before tr

68 CSWD was associated with improved serum triglycerides (evaluated by mean and median change

69 dominal adiposity; hepatosteatosis; elevated serum triglycerides, FFAs, and LDL-cholesterol; and dimi

70 n dosed chronically in DIO mice and depleted serum triglycerides following a lipid challenge in a dos

71 A significant decrease in serum triglycerides (from 103 to 75, 69 and 72 mg/dL), t

72 en); fasting blood glucose > or = 100 mg/dL; serum triglycerides > or = 150 mg/dL; blood pressure > o

73 7; 95% confidence interval [CI]: 1.15-9.89), serum triglycerides >/=150 mg/day (OR 4.35; 95% CI: 1.70

74 glucose level) and insulin resistance (e.g., serum triglycerides, high density lipoprotein cholestero

75 ncarriers had lower fasting and postprandial serum triglycerides, higher levels of HDL-cholesterol an

76 n assembly and allows homeostatic control of serum triglyceride in a fasted state.

77 that regulate lipid metabolism, and reduced serum triglycerides in a PPARalpha-dependent mechanism.

78 APOC3/APOA5 constitutes a major locus for serum triglycerides in Amerindians, especially the Pimas

79 The decrease in serum triglycerides in aP2/DTA mice was due to a marked

80 ound a statistically significant decrease in serum triglycerides in germ-free rats fed a high sugar d

81 ar role for PPARbeta in regulating levels of serum triglycerides in mice on a high fat Western diet b

82 CK sensitivity best correlated with elevated serum triglycerides in normal-weight participants and wi

83 proportion to the increase in liver fat and serum triglycerides in subjects with PNPLA3-148IIbut not

84 teraction to be statistically significant on serum triglycerides in the 1958BC.

85 ms in cell-based assays and strikingly lower serum triglycerides in vivo.

86 Serum leptin was doubled and serum triglycerides increased by 44% after LPS administr

87 ing the SBe+MD treatment, VLDL fractions and serum triglycerides increased.

88 those of a pharmaceutical dose (3.4 g/d) on serum triglycerides, inflammatory markers, and endotheli

89 are positively correlated with FEV1, whereas serum triglycerides, LDL cholesterol, and apoB are assoc

90 21, P = .048), IMCL (r = 0.27, P = .02), and serum triglyceride level (r = 0.33, P = .001), independe

91 High one-year posttransplant serum triglyceride level and pretransplant body mass ind

92 Female patients experienced serum triglyceride level elevations regardless of antips

93 ssue in preventive cardiology is whether the serum triglyceride level is an independent risk factor f

94 analysis in nonobese patients, only elevated serum triglyceride level was independently associated wi

95 antipsychotic dose, total cholesterol level, serum triglyceride level, and concurrent medications wer

96 patic lipids (IHL), serum cholesterol level, serum triglyceride level, and measures of insulin resist

97 lood pressure, platelet sensitivity, and the serum triglyceride level.

98 um cholesterol and a significant increase in serum triglycerides level.

99 w-carbohydrate diet had greater decreases in serum triglyceride levels (change, -0.84 mmol/L vs. -0.3

100 I (OR: 0.62 per 1 SD; 95% CI: 0.40 to 0.94), serum triglyceride levels (OR: 0.66 per 1 SD; 95% CI: 0.

101 at feeding resulted in a greater increase in serum triglyceride levels and an accelerated appearance

102 A good correlation was noted between serum triglyceride levels and blood viscosity (r=0.82).

103 that carry this T-87C polymorphism had lower serum triglyceride levels and significantly reduced risk

104 At week 16, serum triglyceride levels and total cholesterol levels w

105 Serum triglyceride levels at 6 and 12 months were 280 +/

106 eing glucose intolerant, and having elevated serum triglyceride levels compared to any other genotype

107 In atorvastatin treatment groups, total serum triglyceride levels decreased in a dose-dependent

108 During active weight loss, serum triglyceride levels decreased more and high-densit

109 roved adipose tissue lipid storage and lower serum triglyceride levels in the fed state, but do not c

110 h the intestinal epithelium and elevation of serum triglyceride levels in the IAP-deficient mice comp

111 Serum triglyceride levels increase as a result of accumu

112 Geometric mean serum triglyceride levels increased from 118 (95% CI, 11

113 An increase in serum triglyceride levels occurred in clozapine-treated

114 In our study, serum triglyceride levels of P-407 induced mice were ele

115 had increased hepatic lipid accumulation and serum triglyceride levels possibly due to the activation

116 ded otherwise healthy patients with elevated serum triglyceride levels to patients presenting with ac

117 n Old Order Amish participants whose fasting serum triglyceride levels were at the extremes of the di

118 Reductions in insulin resistance and serum triglyceride levels were greater in the combined-i

119 Serum triglyceride levels were increased by incremental

120 sis model assessment values were highest and serum triglyceride levels were lowest among African-Amer

121 iple blood samples were grossly lipemic, and serum triglyceride levels were markedly increased.

122 remental reduction in insulin resistance and serum triglyceride levels when combined with CPAP.

123 Black race, lower BMI, lower serum triglyceride levels, and longer QRS duration were

124 creased body-mass index, waist-to-hip ratio, serum triglyceride levels, and systolic blood pressure a

125 cardial blood volume changed with increasing serum triglyceride levels, indicating lack of vasomotion

126 which 1.2% energy as n-3 fatty acids lowers serum triglyceride levels.

127 Fish oils rich in n-3 fatty acids reduce serum triglyceride levels.

128 crease in both apolipoprotein expression and serum triglyceride levels.

129 diabetic rats results in a rapid increase in serum triglyceride levels.

130 tions in CRP levels, insulin resistance, and serum triglyceride levels.

131 wo (TRG and 4-Me-UAB30) strikingly increased serum triglyceride levels.

132 lterations in metabolic traits, most notably serum triglyceride levels.

133 roved glucose as well as positively affected serum triglyceride levels.

134 ficant association with GFR (P = 0.0006) and serum triglycerides levels (P = 0.003), after accounting

135 and body fat and increased hepatic, but not serum, triglyceride levels compared to control rats that

136 hepatic apolipoprotein A-I, C-III mRNA, and serum triglycerides observed in wild-type mice is mediat

137 squares mean=2.9 kg) and a net reduction of serum triglycerides of 32.7 mg/dl.

138 l did not statistically significantly change serum triglycerides or very-low-density lipoprotein conc

139 Although serum triglycerides originate from the liver, the effect

140 emic cardiovascular benefits of exercise, as serum triglyceride, oxidized low density lipoprotein-cho

141 Insulin resistance (P = .01), serum triglycerides (P = .01), C-reactive protein (P = .

142 ion was negatively correlated with change of serum triglycerides (P = 0.016).

143 diastolic BP (P = 0.0462, beta = 0.0206) and serum triglycerides (P = 0.0206, beta = 0.1090).

144 y mass index (p = 0.01) and higher levels of serum triglycerides (p = 0.05), and they were inversely

145 and obesity in mice, have elevated levels of serum triglycerides primarily associated with very low d

146 ations between small LDL particle number and serum triglycerides (r=0.61, P<0.0001) and HDL-C (r=-0.5

147 of 1-3 grams of nicotinic acid per day lower serum triglycerides, raise high density lipoprotein chol

148 P=0.03), cell volume (rho(G)=-0.73, P=0.04), serum triglycerides (rho(G)=-0.67, P=0.03), and between

149 dex, resting heart rate, C-reactive protein, serum triglycerides, serum creatinine, and measures of d

150 t exhibited significantly lower postprandial serum triglycerides, suggestive of a role for TM6SF2 in

151 low-density lipoprotein (LDL) cholesterols, serum triglycerides, systolic and diastolic blood pressu

152 h fasting insulin, insulin sensitivity (Si), serum triglyceride (TG) concentration, or serum HDL chol

153 Serum triglyceride (TG) level is a well-known risk facto

154 t to assess the association, if any, between serum triglyceride (TG) levels and gemfibrozil consumpti

155 Biochemical parameters measured included serum triglyceride (TG), total cholesterol (TC), low-den

156 57; 95%CI 1.45-1.69, P = 3.84 x 10(-31)) and serum triglycerides (TG) (beta = 0.067, P = 4.5 x 10(-21

157 Serum triglycerides (TG) and cholesterol (CHOL) were ass

158 terol value (CHO) was more than 240 mg/dL or serum triglycerides (TG) were more than 200 mg/dL.

159 Sat expression correlates with steatosis and serum triglycerides (TGs) in humans.

160 l (TC), low-density lipoproteins (LDLs), and serum triglycerides (TGs) were significantly higher in p

161 o the identification and characterization of serum triglycerides (TGs), was assessed using extracted

162 stasis model assessment-insulin resistance), serum-triglyceride-to-serum-high-density lipoprotein-cho

163 Serum triglyceride, total cholesterol, LDL, and VLDL con

164 nsplant patients were found to have elevated serum triglycerides, total cholesterol/ high-density lip

165 ciations between endotoxin/LPS and levels of serum triglycerides, troponin, and HDL.

166 e in HDL-cholesterol and an 84% reduction in serum triglycerides under the same treatment conditions.

167 gpat2 deficiency reports marked reduction in serum triglyceride upon feeding a fat-free diet, which s

168 Serum triglyceride values increased after starting indin

169 Serum triglyceride, very-low-density lipoprotein, and ap

170 and high density lipoprotein cholesterol and serum triglyceride were measured after a 14-hour fast.

171 Serum triglycerides were also lower, but the difference