ライフサイエンスコーパス: sinus node dysfunction

1 , atrial standstill, conduction disease, and sinus node dysfunction.

2 r (DDDR) versus ventricular (VVIR) pacing in sinus node dysfunction.

3 of dual-chamber versus ventricular pacing in sinus node dysfunction.

4 ich reduced Na+ channel function might cause sinus node dysfunction.

5 vent diseases such as atrial fibrillation or sinus node dysfunction.

6 closely resemble those observed in clinical sinus node dysfunction.

7 d risk of death during pacemaker therapy for sinus node dysfunction.

8 with subsequent stroke in patients paced for sinus node dysfunction.

9 and exhibited ventricular preexcitation and sinus node dysfunction.

10 nts (92%) to determine the late incidence of sinus node dysfunction.

11 4 years after the Fontan operation, 44% had sinus node dysfunction.

12 principally in the subgroup of patients with sinus-node dysfunction.

13 At follow-up, 7 (6%) of 108 patients had sinus node dysfunction, a permanent pacemaker, or both,

14 iminished P-wave amplitude characteristic of sinus node dysfunction, an AF risk factor in human patie

15 of pacemaker activity, reminiscent of human sinus node dysfunction and "tachy-brady" syndrome.

16 All 11 patients presented with sinus node dysfunction and 10 had atrial arrhythmias.

17 parasympathetic influence has been linked to sinus node dysfunction and arrhythmia.

18 than the broad categories and indications of sinus node dysfunction and atrioventricular block.

19 cribes an arrhythmia phenotype attributed to sinus node dysfunction and diagnosed by electrocardiogra

20 Following that operation she developed sinus node dysfunction and had a permanent epicardial du

21 We sought to eliminate sinus node dysfunction and postoperative bradyarrhythmia

22 rmal SAN function and the pathophysiology of sinus node dysfunction and suggest new potential targets

23 more likely to occur in patients with early sinus node dysfunction and those with longer follow-up.

24 s more major adverse events, major bleeding, sinus node dysfunction, and pacemaker implantation.

25 Bradycardia and sinus node dysfunction are common causes of early postop

26 pacing on subsequent stroke in patients with sinus node dysfunction are not known.

27 cardiac conduction disorder associated with sinus node dysfunction, arrhythmia, and right and occasi

28 conduction, and human SCN5A mutations cause sinus node dysfunction, atrial fibrillation, conductiona

29 hamber pacing were observed in patients with sinus-node dysfunction, but not in those with atrioventr

30 Patients with sinus-node dysfunction, but not those with atrioventricu

31 ant and persistent atrioventricular block or sinus node dysfunction can occur and indicate a need for

32 nd at 6 months, decreased R wave amplitudes, sinus node dysfunction, cardiac hypertrophy, interstitia

33 cing (DDDR) and ventricular pacing (VVIR) in sinus node dysfunction, demonstrated no difference in de

34 In sinus-node dysfunction, dual-chamber pacing does not imp

35 iciency in mice may cause the stress-induced sinus node dysfunction found in many aged individuals an

36 Sinus node dysfunction has been previously reported to o

37 I (hazard ratio, 4.0; P=0.04), and previous sinus node dysfunction (hazard ratio, 8.0; 95% confidenc

38 ofosbuvir and daclatasvir, he had an extreme sinus node dysfunction (heart rate of 27beats/min).

39 To determine the early incidence of sinus node dysfunction, hospital records and perioperati

40 stroke in a population of patients paced for sinus node dysfunction in a large prospective clinical t

41 Unlike sinus node dysfunction in nontransplanted patients, whic

42 o determine the early and late incidences of sinus node dysfunction in patients systematically and un

43 node function between the 2 stages, 23% had sinus node dysfunction in the early postoperative period

44 r its blood supply is a significant cause of sinus node dysfunction in the orthotopic heart transplan

45 Because sinus node dysfunction in the transplanted heart does no

46 patients, which typically worsens with time, sinus node dysfunction in the transplanted heart usually

47 uld be a therapeutic target for pathological sinus node dysfunction in veteran athletes.

48 Perioperative sinus node dysfunction is common after both the hemi-Fon

49 nus node function between the 2 stages, late sinus node dysfunction is common and more likely to occu

50 Sinus node dysfunction is increasingly common with longe

51 tained atrial tachyarrhythmia, implying that sinus node dysfunction is unlikely to be the dominant me

52 ficant clinical manifestation of progressive sinus node dysfunction, is the most frequent indication

53 ity, spontaneous type I ECG, and presence of sinus node dysfunction might be considered as risk facto

54 Sinus node dysfunction occurred in 12 patients in the GP

55 Sinus node dysfunction occurs commonly after orthotopic

56 Bradycardia, due to sinus node dysfunction or atrioventricular (AV) block, w

57 nical trials in patients with pacemakers for sinus node dysfunction or atrioventricular block (AVB) a

58 eatment for patients with bradycardia due to sinus node dysfunction or atrioventricular block.

59 and 7 of these patients also had evidence of sinus node dysfunction (P < .005).

60 ulmonary connection may increase the risk of sinus node dysfunction, previous studies have not report

61 Pacing-induced chronic AF induces sinus node dysfunction, prolongs intra-atrial conduction

62 of atrial fibrillation and in patients with sinus node dysfunction, reduces heart failure symptoms w

63 Ten patients with sinus node dysfunction scheduled for dual-chamber pacema

64 Although sinus node dysfunction (SND) and atrial arrhythmias freq

65 ration family (n=25) with autosomal dominant sinus node dysfunction (SND) and atrioventricular block

66 ut genetic overlap has not been reported for sinus node dysfunction (SND) and noncompaction cardiomyo

67 ildren experienced more frequent episodes of sinus node dysfunction (SND) compared with older subject

68 Sinus node dysfunction (SND) is a major clinically relev

69 Sinus node dysfunction (SND) is a major public health pr

70 ) compared with ventricular (VVIR) pacing in sinus node dysfunction (SND).

71 ndomized trial of DDDR versus VVIR pacing in sinus node dysfunction (SND).

72 we studied a family with DCM associated with sinus node dysfunction, supraventricular tachyarrhythmia

73 cardiac arrhythmia syndrome associated with sinus node dysfunction that is distinct from long QT syn

74 ing are alternative treatment approaches for sinus-node dysfunction that causes clinically significan

75 Even after appropriate pacing for sinus node dysfunction, the sinus node may recover and p

76 domly assigned a total of 2010 patients with sinus-node dysfunction to dual-chamber pacing (1014 pati

77 tment of pacemaker syndrome in patients with sinus node dysfunction treated with ventricular-based (V

78 ain containing 1 (Popdc1) or Popdc2 leads to sinus node dysfunction under stressed conditions in aged

79 , whereas observed survival of patients with sinus node dysfunction was not significantly different f

80 Sinus node dysfunction was present in 7% of the patients

81 A total of 2,010 patients with sinus node dysfunction were randomized to ventricular or

82 ECG analysis revealed severe sinus node dysfunction when freely roaming mutant animal

83 stro-esophageal reflux, retinal disease, and sinus-node dysfunction, whereas related heterozygotes ha

84 art failure hospitalization in patients with sinus node dysfunction who require pacemaker therapy is