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1  they have been marketed as a cheaper, safer smokeless alternative to traditional cigarettes and a po
2 l prevalence of current tobacco use (smoked, smokeless, and any tobacco use) among 6729 HIV-positive
3 rs, such as alcohol and tobacco (smoking and smokeless), but dietary factors and the existence of gen
4                                          The smokeless cigarette, Eclipse, has comparable ROS with ci
5 , respectively), followed by smoky coal, and smokeless coal (GM:148 and 115 mug/m(3), respectively).
6  6 times higher when burning smoky coal than smokeless coal and varied by up to a factor of 3 between
7 or users of smoky coal, compared to users of smokeless coal and wood.
8 nwei and Fuyuan females who used smoky coal, smokeless coal, or wood and 10 local controls who used e
9 in health evaluations is usually compared to smokeless coal--an anthracite coal available in some par
10  solid sorbent traps from the headspace of a smokeless gunpowder sample.
11 itive, and rapid method for the detection of smokeless powder components, from five different types o
12 ts resulted in perfect classification of the smokeless powder with respect to manufacturer.
13               ESI FTICR mass spectrometry of smokeless powder, TNT, and Powermite resolves and identi
14 rotoluene (TNT), and low explosives (several smokeless powders) resulting in the detection of 26-35 n
15 31-2.59], respectively), attempt to purchase smokeless tobacco (adjusted OR, 2.16 [95% CI, 1.90-2.45]
16 rrent prevalence, 15.7% vs 3.9%; P<.001) and smokeless tobacco (current prevalence, 8.7% vs 0.4%; P<.
17 s to determine whether an aqueous extract of smokeless tobacco (moist snuff) increases clearance of m
18 the validity of self-reported information on smokeless tobacco (SLT) use is uncertain.
19 on (EU) legislation bans the sale of snus, a smokeless tobacco (SLT) which is considerably less harmf
20              Despite the reported effects of smokeless tobacco (ST) on the periodontium and high prev
21 mucosal lesions frequently occur at sites of smokeless tobacco (ST) placement.
22  favoured by most smokers (82%) overall, but smokeless tobacco and bidis were commonly used in India
23                 Furthermore, combined use of smokeless tobacco and cigarettes did not increase overal
24  cancer mortality rate among combined users (smokeless tobacco and cigarettes), based on the rates fo
25  al. describe the association between use of smokeless tobacco and head and neck cancer in 11 US case
26  Few or no associations between each type of smokeless tobacco and HNC were observed among ever cigar
27 e exposed organotypic cultures for 3 days to smokeless tobacco aqueous extracts and determined the ch
28   The FDA thus concluded that cigarettes and smokeless tobacco are subject to FDA jurisdiction becaus
29                               Current use of smokeless tobacco at baseline was associated with 1.27-f
30 correlated with number of tins or pouches of smokeless tobacco consumed.
31                         We hypothesized that smokeless tobacco could modulate the growth of keratinoc
32 A determined that nicotine in cigarettes and smokeless tobacco does "affect the structure or any func
33             Overall, these data suggest that smokeless tobacco elicits plasma exudation in the oral m
34 lth questionnaire revealed that he was using smokeless tobacco every day.
35 effects of cigarette smoke extract (CSE) and smokeless tobacco extract (STE) on cell survival and mot
36                                  Exposure to smokeless tobacco extract was associated with a signific
37                                              Smokeless tobacco extract-induced leaky site formation a
38   Indomethacin had no significant effects on smokeless tobacco extract-induced responses.
39 , therefore, were differentially affected by smokeless tobacco extracts in an organotypic tissue mode
40                                 In contrast, smokeless tobacco extracts promoted fibroblast growth at
41 shown that most consumers use cigarettes and smokeless tobacco for pharmacological purposes, includin
42                                       Use of smokeless tobacco in the United States has been relative
43 d of accelerating the decline in smoking and smokeless tobacco initiation.
44 dical school curricula, specific training in smokeless tobacco intervention, tobacco intervention tra
45                                              Smokeless tobacco is a powerful autonomic and hemodynami
46                                              Smokeless tobacco is associated with pathologic alterati
47  factor for periodontal disease, the role of smokeless tobacco is unclear.
48                         The presence of oral smokeless tobacco lesions among adolescents may be an ea
49                                   Preventing smokeless tobacco lesions and their possible malignant t
50                                              Smokeless tobacco lesions were detected in 1.5% of stude
51 tronger risk factor than chewing tobacco for smokeless tobacco lesions, but the use of either of thes
52 cohol or cigarettes may increase the risk of smokeless tobacco lesions.
53                  These results indicate that smokeless tobacco may also be an important risk factor f
54 arction (MI) in people who use snus, a moist smokeless tobacco product, we hypothesized that disconti
55  with regular and heavy nicotine intake from smokeless tobacco rather than from smoking.
56                             Current users of smokeless tobacco should be informed of its harm and adv
57 A) asserted jurisdiction over cigarettes and smokeless tobacco under the Federal Food, Drug, and Cosm
58 co smoking (1.90 [1.38-2.62]; p<0.0001), and smokeless tobacco use (1.32 [1.03-1.69]; p=0.030) than i
59 ment for confounders, no association between smokeless tobacco use and all-cause (hazard ratio = 1.1,
60 ter understand the cancer risks of exclusive smokeless tobacco use and dual use of smokeless tobacco
61                          Previous studies on smokeless tobacco use and head and neck cancer (HNC) hav
62 rvey was to evaluate the association between smokeless tobacco use and severe active periodontal dise
63 udy was to characterize the relation between smokeless tobacco use and the risk of all-cause and dise
64                                              Smokeless tobacco use appears to be associated with HNC,
65 e smoking, nicotine replacement therapy, and smokeless tobacco use during pregnancy are associated wi
66             The pooled prevalence of current smokeless tobacco use in pregnant women was lowest in th
67                        The health effects of smokeless tobacco use need further documentation.
68                            The difference in smokeless tobacco use prevalence between HIV-positive an
69 7.8) for tobacco smoking, 3.4% (1.8-5.6) for smokeless tobacco use, and 27.1% (22.8-31.7) for any tob
70 1.9) for tobacco smoking, 2.1% (1.1-3.4) for smokeless tobacco use, and 3.6% (95% CI 2.3-5.2) for any
71 ative prevalence ratios for tobacco smoking, smokeless tobacco use, and any tobacco use separately fo
72 luc), in urine and plasma after cessation of smokeless tobacco use, in which NNK is administered p.o.
73 d at intervals 2-126 days after cessation of smokeless tobacco use.
74 lity of borderline significance among female smokeless tobacco users (hazard ratio = 1.7, 95% CI: 1.0
75 eline (1971-1975) were categorized as either smokeless tobacco users (n = 1,068) or non-smokeless tob
76 r smokeless tobacco users (n = 1,068) or non-smokeless tobacco users (n = 5,737).
77 igarettes), based on the rates for exclusive smokeless tobacco users and exclusive smokers, was highe
78 ays after cessation than at baseline in both smokeless tobacco users and smokers, indicating stereose
79 Gluc (26.1 +/- 15.1 versus 39.6 +/- 26.0) in smokeless tobacco users and smokers.
80 3.89 +/- 2.43) were significantly shorter in smokeless tobacco users than in smokers.
81                  The mortality experience of smokeless tobacco users was not significantly greater th
82 assessed the 20-year mortality experience of smokeless tobacco users.
83  the authors examined whether current use of smokeless tobacco was associated with increased incidenc
84                In conclusion, current use of smokeless tobacco was associated with increased risk of
85                                  Past use of smokeless tobacco was not associated with CVD incidence.
86  adults and never-smokers who currently used smokeless tobacco were twice as likely to have severe ac
87 lusive smokeless tobacco use and dual use of smokeless tobacco with other tobacco products, including
88 ion on bidis (small hand-rolled cigarettes), smokeless tobacco, and locally brewed alcohols.
89 s, pipe tobacco, hookah, snus pouches, other smokeless tobacco, dissolvable tobacco, bidis, and krete
90 te smoke may have more profound effects than smokeless tobacco.
91 f quitting which do not include switching to smokeless tobacco.
92 e acute hemodynamic and autonomic effects of smokeless tobacco.
93 sues, less is understood about the effect of smokeless tobacco.
94 r pipe smokers, and 2% were current users of smokeless tobacco.
95                                          All smokeless tobaccos stimulated keratinocyte proliferation
96 revalence of current tobacco use (smoked and smokeless) was estimated for every country.

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