ライフサイエンスコーパス: spinal cord compression

1 c fracture, radiation or surgery to bone, or spinal cord compression).

2 c fracture, radiation or surgery to bone, or spinal cord compression).

3 lled pain, impending pathologic fracture, or spinal cord compression.

4 , bone fracture and paralysis resulting from spinal cord compression.

5 bone metastases include pain, fractures, and spinal cord compression.

6 ity caused by moderate to severe pain and by spinal cord compression.

7 oracic vertebra may give rise to symptomatic spinal cord compression.

8 They had no metastases or spinal cord compression.

9 nclude bone pain, pathological fractures and spinal cord compressions.

10 emotherapy (1.6%) or major interventions for spinal cord compression (0.9%) was uncommon.

11 Corticosteroids have a specific role in spinal cord compression and brain metastases, where impr

12 L1, detectable already 1 week after thoracic spinal cord compression and immediate vector injection,

13 vis, or femur require careful evaluation for spinal cord compression and pathologic fracture before b

14 fracture, radiation or surgery to bone, and spinal cord compression) and hypercalcemia were assessed

15 need for radiation to bone or bone surgery, spinal cord compression, and hypercalcemia (a serum calc

16 r fracture or impending fracture, radiation, spinal cord compression, and hypercalcemia.

17 tion or surgery to treat bone complications, spinal cord compression, and hypercalcemia.

18 demonstrated in models of cerebral ischemia, spinal cord compression, and sciatic nerve crush.

19 on should be sought for long-bone fractures, spinal cord compression, and vertebral column instabilit

20 pain, prevent pathological fractures and/or spinal cord compression are also considered skeletal-rel

21 Spinal metastatic disease and spinal cord compression are major causes of morbidity an

22 Data on assessment and treatment of spinal cord compression are reviewed.

23 Presence of a nerve or spinal-cord compression at diagnosis, presence or site o

24 ing, patient request for referral, delirium, spinal cord compression, brain or leptomeningeal metasta

25 ditions, and 90% of ambulatory patients with spinal cord compression can remain ambulatory.

26 This report describes a case of spinal cord compression caused by Actinomyces israelii w

27 The standard treatment for spinal cord compression caused by metastatic cancer is c

28 ed trial, we randomly assigned patients with spinal cord compression caused by metastatic cancer to e

29 nt with radiotherapy alone for patients with spinal cord compression caused by metastatic cancer.

30 Malignant spinal cord compression demands immediate spinal imaging

31 or bone pain (HR 0.67, 95% CI 0.53-0.85) and spinal cord compression (HR=0.52, 95% CI 0.29-0.93) were

32 ture, irradiation of or surgery on bone, and spinal cord compression), hypercalcemia (symptoms or a s

33 morbidity, such as pathologic fractures and spinal cord compression in men with hormone-refractory p

34 ed acutely 25 days following graded thoracic spinal cord compression in rats.

35 t functional and morphological outcome after spinal cord compression injury may occur in a discontinu

36 Two days after transient T6 spinal cord compression injury, wild-type mice developed

37 examining the effects of DHA treatment after spinal cord compression injury.

38 keletal complications: pathologic fractures, spinal cord compression, irradiation of or surgery on bo

39 When spinal cord compression is suspected, providers should t

40 lly selected MS patients who have coexistent spinal cord compression is well tolerated and may result

41 Spinal cord compression led to long lasting hypersensiti

42 tra-canal spinal pathological masses causing spinal cord compression, lung abscess, pyothorax, parave

43 ose hypermetabolism at the level of cervical spinal cord compression may predict an improved outcome

44 Metastatic epidural spinal cord compression (MESCC) occurs when cancer metas

45 therapy in patients with metastatic epidural spinal cord compression (MESCC), the impact of surgery o

46 patients who, each year, develops metastatic spinal cord compression, Ms H wishes to walk and live he

47 Malignant spinal-cord compression (MSCC) is a common complication

48 indicated that DHA administered 30 min after spinal cord compression not only greatly increased survi

49 ses can lead to emergent situations, such as spinal-cord compression or impending fracture of a weigh

50 ogic fracture, radiation or surgery to bone, spinal cord compression, or hypercalcemia), and a pilot

51 conservatively treated pathologic fracture, spinal cord compression, or hypercalcemia, were taken di

52 d events (SREs) such as pathologic fracture, spinal cord compression, or the necessity for radiation

53 vertebral or non-verterbal), or occurence of spinal cord compression, or tumour-related orthopeadic s

54 , which we defined as pathological fracture, spinal cord compression, palliative radiation to bone, o

55 These were defined as fractures, spinal cord compression, radiation or surgery to bone, a

56 event (SRE), defined as pathologic fracture, spinal cord compression, radiation therapy to bone, and

57 related event (defined as clinical fracture, spinal cord compression, radiation to bone, or surgery i

58 x patients in the treatment group (malignant spinal cord compression requiring surgery [grade 3]; mal

59 spinal epidural abscess may be the result of spinal cord compression, spinal cord arterial and/or ven

60 ned as radiation to bone, clinical fracture, spinal cord compression, surgery to bone, or death as a

61 vical stenosis with neuroradiologic signs of spinal cord compression underwent decompressive surgery.

62 he only primary therapy except in cases with spinal cord compression, where radiation therapy was all

63 01 reduced the area of damage at the site of spinal cord compression, which was corroborated by histo