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1 t antimicrobial strategies, leading to fatal staphylococcal infection.
2 and IL-22 protects against severe pulmonary staphylococcal infection.
3 andidiasis predisposes the host to secondary staphylococcal infection.
4 bers of neutrophils, the key defense against staphylococcal infection.
5 nt complication of non-Staphylococcus aureus staphylococcal infection.
6 n and thus contribute to the pathogenesis of staphylococcal infection.
7 sal carriage is a documented risk factor for staphylococcal infection.
8 mans, which in turn may diminish the risk of staphylococcal infection.
9 nd of increasing risk for coagulase-negative staphylococcal infection.
10 as been associated with an increased risk of staphylococcal infection.
11 ad spectrum of superantigens released during Staphylococcal infection.
12 multiplex PCR protocol for the diagnosis of staphylococcal infection.
13 l subset 2 and is frequently associated with staphylococcal infection.
14 major source of inflammatory cytokines after staphylococcal infection.
15 ungoides, were studied for the occurrence of staphylococcal infection.
16 nly 1 initiated CAS; 67% (8/12) of these had staphylococcal infections.
17 nd develop therapeutic approaches to control staphylococcal infections.
18 for attenuating current multidrug resistant staphylococcal infections.
19 potential treatment for multidrug-resistant staphylococcal infections.
20 he development of novel targets for treating staphylococcal infections.
21 a rise in enterococci and coagulase-negative staphylococcal infections.
22 ctams with approved clinical indications for staphylococcal infections.
23 ng into question the role of Agr in invasive staphylococcal infections.
24 rtality, and economic impact associated with staphylococcal infections.
25 omplement resistance and the pathogenesis of staphylococcal infections.
26 e agents for the management and treatment of staphylococcal infections.
27 al resistance highlights their importance in staphylococcal infections.
28 n important role in cardiovascular and other staphylococcal infections.
29 assay should improve clinical management of staphylococcal infections.
30 amamelitannin may be used as a suppressor to staphylococcal infections.
31 at interferons play a protective role during staphylococcal infections.
32 ibutions of prophages to the pathogenesis of staphylococcal infections.
33 herapeutic agents for preventing or treating staphylococcal infections.
34 sues and for the establishment of persistent staphylococcal infections.
35 itor can eliminate medical device-associated staphylococcal infections.
36 s an important aspect of the pathogenesis of staphylococcal infections.
37 gnificant clinical implications for relapsed staphylococcal infections.
38 e responsible for approximately 70% of human staphylococcal infections.
39 cts with the TLR2 Arg753Gln polymorphism had staphylococcal infections.
40 n the only uniformly effective treatment for staphylococcal infections.
41 intracellular survival in the persistence of staphylococcal infections.
42 hylococcus aureus plays an important role in staphylococcal infections.
43 simplex infections, neonatal varicella, and staphylococcal infections.
44 is an integral part of the innate defense to staphylococcal infection and comprises a well-coordinate
45 d laboratory evidence support roles for both staphylococcal infection and environmental allergens in
47 mial bacterial sepsis and coagulase negative staphylococcal infections, and thus should not be used i
51 ocytes (PMN) are the first responders during staphylococcal infection, but 15-50% of the initial inge
52 lls modulate the development of experimental staphylococcal infections by controlling polymorphonucle
53 ese findings show a novel mechanism by which staphylococcal infections can inhibit Th1 reactions and
55 intact complement pathway in an experimental staphylococcal infection, control and C3-depleted mice w
56 y, abscesses can persist or even reoccur, as staphylococcal infections fail to induce protective immu
57 s from patients with invasive or superficial staphylococcal infections for use in the Tigecycline Eva
59 ribe here a novel, spontaneous model of oral staphylococcal infection in double knockout mice, defici
60 thicillin-resistant S. aureus, many cases of staphylococcal infection in the ICU are now classified a
63 mpted a change in the approach to therapy of staphylococcal infections in both the outpatient clinic
64 ing truncated AIPs as a means of attenuating staphylococcal infections in species beyond Staphylococc
65 in a large proportion of coagulase-negative staphylococcal infections in very-low-birth-weight infan
66 ort an outbreak of botryomycosis, an unusual staphylococcal infection, in a colony of uPA-deficient m
70 such a devastating impact on chemotherapy of staphylococcal infections--is related to the properties
71 (1) the effect of PVL on the pathogenesis of staphylococcal infection may depend on the level of toxi
72 omparative studies exist in the treatment of staphylococcal infections may also offer effective and l
73 whether host factors released in response to staphylococcal infections may increase cell resistance t
80 diabetic db/db mice are more susceptible to staphylococcal infection than their nondiabetic litterma
81 unities are often associated with persistent staphylococcal infections that place a tremendous burden
83 groups, the mortality rate in patients with staphylococcal infections was significantly higher among
84 Thus, using in vitro models of intracellular staphylococcal infection, we demonstrate that EsxA inter
85 c target of the humoral immune system during staphylococcal infections, we developed a synthetic meth
86 in a pediatric hospital with a high rate of staphylococcal infections were examined for any increase
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