ライフサイエンスコーパス: stressor

1 gonist (yohimbine; used as a pharmacological stressor).

2 rfusion injury (IRI), a common type of renal stressor.

3 ting time as an additional work-related time stressor.

4 ediately following the first exposure to the stressor.

5 e also induced in wild-type mice by an acute stressor.

6 tic conditions altered vulnerability to this stressor.

7 ed as a result of direct experience with the stressor.

8 nce, dominance, and warmth) responses to the stressor.

9 Radiation is a prevalent environmental stressor.

10 ulates a conserved survival response to this stressor.

11 nd 5.5% for peak cortisol levels following a stressor.

12 ly depended upon the presence of the natural stressor.

13 ponse to an inflammatory challenge or social stressor.

14 amine can be prophylactic against subsequent stressors.

15 properties against endogenous and exogenous stressors.

16 for mental disorders arising through social stressors.

17 y occurs when mitochondria adapt to tolerate stressors.

18 organismal capacity to respond to additional stressors.

19 n exposures and integrate risk from multiple stressors.

20 istinct context, likely triggered by similar stressors.

21 T to VE to see if they differ with different stressors.

22 after a recurrent exposure to environmental stressors.

23 organism's capacity to respond to additional stressors.

24 lethal exposure to natural and anthropogenic stressors.

25 or bodily responses to internal and external stressors.

26 is association across a large range of acute stressors.

27 ays a critical role in resistance to various stressors.

28 general case in adaptation to environmental stressors.

29 ccount for increased complexity and multiple stressors.

30 ns are linked to adaptation to environmental stressors.

31 and the relative importance of anthropogenic stressors.

32 he many other coastal systems facing similar stressors.

33 at varying levels of one or more additional stressors.

34 igenetic memory in response to environmental stressors.

35 response to combined climate change-related stressors.

36 rders, as well as sensitivity to compounding stressors.

37 not lead to adequate responses to the biotic stressors.

38 er risk of adverse outcomes after even minor stressors.

39 more resources to tolerate and recover from stressors.

40 proteins to survive the sudden appearance of stressors.

41 ific biological systems impacted by external stressors.

42 te samples that are exposed to environmental stressors.

43 e to acidification as well as the additional stressors.

44 ems maintenance, is altered by anthropogenic stressors.

45 change, food availability, and human-related stressors.

46 that have the potential to act as additional stressors.

47 s been linked to reduced ability to adapt to stressors.

48 to drugs of abuse, drug-associated cues, and stressors.

49 environmental contaminants and psychosocial stressors.

50 entrally controls responses to environmental stressors.

51 oral reefs isolated from other anthropogenic stressors.

52 s overseas, and loneliness as post-migration stressors.

53 ate change when such change results in novel stressors.

54 llness) and the number of social integration stressors (1.10, 1.05-1.16 for PTSD).

55 estments to counteract land- and ocean-based stressors: (1) marine restoration should be prioritised

56 arrhythmias after exposure to QT-prolonging stressors, 10 had other LQTS pathogenic mutations, and 1

57 and were then exposed to a social-evaluative stressor (a mock job interview).

58 re often a result of different environmental stressors acting synergistically.

59 mechanisms by which different environmental stressors affect plasmodesmal permeability are not well

60 This is because environmental stressors also cause CHD.

61 e relatively inhibited in the absence of the stressor and mounted robust responses upon stressor pres

62 rossover study addressed the impact of daily stressors and a history of major depressive disorder (MD

63 eing implemented to relieve areas from local stressors and allow populations to restore to natural le

64 These data show how recent stressors and an MDD history can reverberate through met

65 ging support is needed to identify potential stressors and available social support in order to promo

66 Psychological stressors and inappropriate inflammation increase the ri

67 Psychological/physical stressors and local tissue damage increase inflammatory

68 t-migration potentially traumatic events and stressors and mental health, and assessed the moderating

69 d periphyton communities exposed to chemical stressors and show that effects on primary production un

70 he interplay of regional scale environmental stressors and the lack of long-term instrumental data of

71 ntory of Stressful Events assessed prior day stressors and the Structured Clinical Interview for DSM-

72 Interactions among global change stressors and their effects at large scales are often pr

73 explained by the genetic vulnerability, the stressors and their interactions, we fitted linear mixed

74 Increasing effects of anthropogenic stressors and those of natural origin on aquatic ecosyst

75 yloidogenic LC production as an intrinsic PC stressor, and identifies stress-responsive pathways as n

76 ement to different drugs of abuse, different stressors, and different behavioral procedures.

77 the influence of biological sex, early-life stressors, and genetic and epigenetic factors in individ

78 y vulnerable to adverse effects of perinatal stressors, and impaired cBF integrity results in lasting

79 e valence systems), reactivity to subsequent stressors, and, ultimately, risk for psychopathology.

80 ain baseline, predisposing and precipitating stressors, angiographic features, revascularization, use

81 to affect the transcriptome's response to a stressor.Animals' response to acute stress is known to b

82 rs in response to prototypical mitochondrial stressors (antimycin-A, xanthine/xanthine oxidase).

83 logical reserve and diminished resistance to stressors; approximately 10% of those in the elderly pop

84 cope with later environments where the same stressors are experienced.

85 Ubiquitous environmental stressors are often thought to alter animal susceptibili

86 cellular stress and found that the oxidative stressor arsenite and heat shock-activated stress respon

87 biological communities respond to concurrent stressors at local-to-regional scales remains largely un

88 ssive mortality to anoxia (a postulated SIDS stressor) at P5 and P8.

89 they did not allow responding to the biotic stressors attack adequately, leading to the observed gro

90 stressors implicated in colony declines, one stressor being the exposure to pesticides.

91 and atolls are less exposed to direct human stressors but are becoming increasingly vulnerable becau

92 umulative risk assessments consider multiple stressors but it is impractical to test every chemical c

93 .SIGNIFICANCE STATEMENT Everyone experiences stressors, but how we respond to them can range from pro

94 capable of adapting to internal and external stressors by undergoing structural plasticity, and failu

95 As expected, for a woman with no prior day stressors, C-reactive protein (CRP), serum amyloid A (SA

96 iated microbiota in mice exposed to a social stressor (called social disruption), as well as non-stre

97 Natural and anthropogenic stressors can cause phase shifts from coral-dominated to

98 site-sex mates and suggest that a variety of stressors can induce social support-like behaviors.

99 ndividuals, exposure to severe or cumulative stressors can lead to an array of pathological condition

100 s into the mechanism through which emotional stressors can lead to cardiovascular disease in human be

101 y vulnerable period when chronic exposure to stressors can precipitate the onset of a broad spectrum

102 Many stressors cause an increase in ventilation in humans.

103 ecipitation (ChIP) analysis showed that this stressor caused substantial gene-dependent increases in

104 Furthermore, stressors caused the recruitment of previously inactive

105 e substantia nigra (SN) to neurodegenerative stressors causes Parkinson's disease (PD).

106 S: Mammalian cells are frequently exposed to stressors causing volume changes.

107 any single etiological agent, environmental stressors compromising coral condition might play a larg

108 mg Cl(-)/L), ethanol-vehicle control, or no-stressor control) and subsequently tested their toleranc

109 o standardized method for analyzing multiple-stressor data using dose-response regression.

110 mixture gene expression profiles from single stressor data were able to better predict effects on rep

111 tems on Earth, yet the extent to which human stressors determine species occurrences, compared with b

112 Interestingly, mixed stressors did not affect crash rates in startling events

113 increases in phenotypes relevant to anxiety/stressor disorders and alcohol addiction, including anxi

114 ence the pathologic outcome or response to a stressor due to the release of a plethora of substances,

115 difications associated with early transplant stressors (e.g., cold ischemia time) and donor aging.

116 Models developed specifically for groups of stressors (e.g., either cyanobacteria or insecticides) p

117 In IECs exposed to cell stressors, EIF2A signaling reduced expression of VIL1 an

118 In this model, neither a stressor (electric footshock) nor stress-level corticost

119 Salivary cortisol confirmed that our stressor elicited changes in endocrine activity.

120 de variety of both natural and anthropogenic stressors encountered by corals in the Anthropocene.

121 o study how this organism withstands various stressors encountered in the oral cavity.

122 reas are also hotspots of the other climatic stressors examined.

123 Multiple-stressor experiments improve our understanding of an org

124 we found that neutrophils and monocytes from stressor-exposed mice expressed higher levels of IL-1bet

125 The stressor-exposed mice had significant differences in mic

126 These data indicate that stressor exposure affects the colonic microbiota during

127 Further, developmental stressor exposure confers long-term habit biases across

128 Stressor exposure significantly affects the colonic muco

129 tes multiple neurobehavioral consequences of stressor exposure, including long-lasting biases towards

130 Our findings indicate that during stressor exposure, Lactobacillus spp. can translocate to

131 as a control, via oral gavage following each stressor exposure.

132 (BNST) mediates many consequences of chronic stressor exposure.

133 nflammation, suggesting that like many other stressors, extended sleep disruption may lead to a state

134 ted by activation of the receptor during the stressor followed by a persistent, ligand-independent co

135 ate adaptive autonomic responses to physical stressors (for example, hypotension, hemorrhage and pres

136 ure to physical (centrifugation) and thermal stressors (freeze, thaw cycling).

137 bitat patches within a landscape to modulate stressors from extreme climate events, and animal distri

138 The aggregate exposure pathway (AEP) tracks stressors from sources to the exposures and serves as a

139 ations between cortisol response following a stressor, functional brain activity to reward, and depre

140 Mice exposed to the stressor had different microbial community composition r

141 Different emotional or psychological stressors have been identified to precede the onset of T

142 Diverse cellular stressors have been observed to trigger site-specific ub

143 -related differences disappeared-because the stressors heightened CRP, SAA, sICAM-1 and sVCAM-1 respo

144 for extrapolation of laboratory findings of stressor impacts on specific components of the axis.

145 ng genetic factors, environmental toxins and stressors, impaired immune responses, mitochondrial dysf

146 ely studied, showing that there are multiple stressors implicated in colony declines, one stressor be

147 led the pleotrophic acts of an environmental stressor in a complex stress system relevant to public h

148 predicted from combining the effect of each stressor in isolation.

149 etamine should be administered relative to a stressor in order to maximize its antidepressant and/or

150 , first shown with an intermittent footshock stressor in rats trained to self-administer heroin, gene

151 tressors were reported in 48.3% and physical stressors in 28.1%.

152 eful as a model for increasing resilience to stressors in at-risk populations.

153 ssed the moderating effect of post-migration stressors in humanitarian migrants in Australia.

154 d be predicted based on the effects of these stressors in isolation, suggesting that OA increases the

155 rier that protects the cell from a number of stressors in the environment.

156 it enhances adaptive responses to ecological stressors in the shared environment.

157 ith better adaptive fitness to environmental stressors in this endangered tree species.

158 , H2S elicits cytoprotective effects against stressors in various cellular models of injury.

159 Plants respond to multiple stressors in ways not directly predictable from single s

160 red in a prophylactic manner 1 week before a stressor, in order to protect against heightened fear re

161 uced by arsenite, a thiol-directed oxidative stressor, in the absence of eIF2alpha phosphorylation su

162 vulnerable this species is to anthropogenic stressors including exploitation.

163 ), a conserved eukaryotic response to myriad stressors including hypoxia, may play a role in hypoxia-

164 ytic phenotype when evaluated using multiple stressors including open-field, elevated plus maze, hole

165 nditions in addition to multiple, additional stressors including thermal stress, food-limitation, and

166 This review investigates the impact of stressors including: cold, heat, hypoxia, pain and panic

167 onfounding factors were resettlement related stressors, including loneliness (odds ratio 1.17, 95% CI

168 enhanced stability against extreme external stressors, including organic solvents, proteases, and et

169 Anthropogenic stressors, including pollutants, are key evolutionary dr

170 Freshwater ecosystems are exposed to many stressors, including toxic chemicals and global warming,

171 but are threatened by multiple anthropogenic stressors, including warming.

172 These stressors increase turf and macroalgal cover, destabiliz

173 esearch showing: (1) that exposure to social stressors increases proinflammatory activity, (2) that i

174 l confinement (SC), a moderate environmental stressor, increases tic-like responses and elicits TS-li

175 Acute stressors induce brain activation, increasing bioavailab

176 ting host social and communicative behavior, stressor-induced behavior, and performance in learning a

177 The potent HN analogue HNG protects from stressor-induced cell death in fibroblasts, cardiomyobla

178 Stressor-induced energetic trade-offs in physiology and

179 nitude of individual CRH cells covaried with stressor intensity.

180 How the duration and timing of exposure to stressors interact to shape critical windows and influen

181 ms have evolved in response to environmental stressors, interactions dictated by physical forces that

182 However, duration of exposure to a stressor is likely critical, because while chronic stres

183 However, an overlooked stressor is the change in ocean circulation in response

184 ntinental scale; likewise the effect of each stressor is variable at regional scales.

185 uced cell viability and elevated response to stressors is noted in T150 mutant cells.

186 rupt reorganizations in response to changing stressors, known as regime shifts or critical transition

187 ER stressors lead to a PERK-dependent activation and recrui

188 lnerability of mitochondria to mitochondrial stressors, leading to elevated superoxide release and re

189 We demonstrate that this early life stressor leads to enhanced excitatory synaptic transmiss

190 es have shown that exposure to psychological stressors leads to inflammation throughout the body.

191 Moreover, as interactions with natural stressors may cause underestimates of the impact of pest

192 Hematopoietic stressors may contribute to leukemic transformation by i

193 se results suggest that repeated exposure to stressors may differentially alter the neural circuits u

194 known about the influence that environmental stressors may have on genome-wide methylation patterns,

195 designed experiments to test how exposure to stressors may induce resource trade-offs that shape crit

196 s support that deficiencies in recovery from stressors may result in attenuated tolerance to stress t

197 s where rivers are already degraded by other stressors may undermine efforts to conserve biodiversity

198 Analyses targeted two stressors (narrow, broad) and two depression outcomes (c

199 are increasingly threatened by a variety of stressors, no comprehensive global analysis of change in

200 esponse to projected multiple climate change stressors (ocean acidification and warming) on Antarctic

201 lation should be considered as an additional stressor of marine ecosystems in a similar way to ocean

202 Here, we ask (i) does the intensity of stressors of an ECE vary across a landscape?

203 them adjust to the associated environmental stressors of this time.

204 Stressors often contribute to difficulties in maintainin

205 In the case of cognitive or emotional stressors, often a smaller range of lane departures was

206 tigated the effect of a single-episode acute stressor on generalization of aversive learning when str

207 ique to assess for the effects of additional stressors on an LCx estimate in a consistent framework,

208 and chemical (elevated metal concentrations) stressors on benthic macroinvertebrate communities.

209 ognitive, emotional, sensorimotor, and mixed stressors on driver arousal and performance with respect

210 model to quantify the roles of anthropogenic stressors on hydrologic alteration and biodiversity in U

211 ences (such as famine and exposures to other stressors) on the long-term persistence of specific patt

212 dent following the six-cycle exposure to the stressor, on Day 6 post-C. rodentium challenge, and pers

213 ans are threatened by numerous global change stressors, one of which is ocean acidification.

214 Mice were exposed to the social stressor or home cage control conditions for six consecu

215 e and depends on the context and type of the stressor or pathology.

216 duration and susceptibility of organisms to stressors or disease.

217 To minimize the deleterious effects of stressors organisms adopt various strategies.

218 we propose as an additional, fourth putative stressor (pathogens), which may adversely impact the end

219 ible growth arrest triggered by a variety of stressors, plays important roles in normal physiology an

220 For biotic stressors, post-stress reductions in ID are consistent w

221 us) to one of seven natural or anthropogenic stressors (predator cue (Anax spp.), 0.5 or 1.0 mg carba

222 e stressor and mounted robust responses upon stressor presentation.

223 Twenty-four hours following the last stressor, rats were bilaterally infused into the BNST wi

224 entially traumatic events and post-migration stressors related to asylum process and resettlement wer

225 athogenetic role by transmitting trauma- and stressor-related disorders (TSRD) across generations.

226 s of a major pharmacotherapy for trauma- and stressor-related disorders and anxiety disorders.

227 cellular stress responses elicited by these stressors represents a key step in understanding potenti

228 , have historically been considered negative stressors, requiring responses that are strictly defensi

229 tosynthetic efficiency, tolerance to abiotic stressors, resistance to fungal pathogens and grain qual

230 sticide tolerance results from a generalized stressor response in organisms, and would be induced fol

231 Rather, distinct hematopoietic stressors result in the selective expansion of HSPCs car

232 do not themselves harm cells through complex stressors resulting from their design or operational con

233 For the stressors reviewed, as the stress becomes extreme VE gen

234 In contrast to the biological stressors, rotation sequence had a greater effect on rhi

235 ecosystem components under a combination of stressor scenarios.

236 ptom Scale-Interview Version (PSS-I) and the stressor-specific Posttraumatic Stress Disorder Checklis

237 Exposure to the SDR stressor specifically increased Lactobacillus RNA in the

238 ophy as well as decreased basal, diurnal and stressor-stimulated plasma corticosterone secretion and

239 t, our findings for the main effects of life stressors (strong risk factor) and 5-HTTLPR genotype (no

240 elated traits and when challenged by certain stressors, succumb to enhanced tumour development and an

241 ment resistance and upregulated by apoptotic stressors such as chemotherapy.

242 HT-29 cells were incubated with cell stressors such as DTT, IFN, and adherent-invasive E coli

243 l circuits, and from environmental energetic stressors such as food deprivation and physical exertion

244 survive and proliferate when challenged with stressors such as growth factor insufficiency.

245 In this polyphenism, environmental stressors such as high aphid density cause asexual, vivi

246 could worsen glycemic control by activating stressors such as higher neighborhood crime, lower housi

247 ay aggravate harmful effects of ROS-inducing stressors such as ionising radiation.

248 n fail to account for ongoing changes due to stressors such as land use change and climate change and

249 ween thermal effects and other environmental stressors such as organic pollution and hypoxia.

250 ers that stabilize proteins to environmental stressors such as refrigeration and elevated temperature

251 were or were not (controls) exposed to cell stressors such as tumor necrosis factor and adherent-inv

252 al vulnerability who experience an exogenous stressor, such as prone/side sleeping or soft bedding, d

253 Time-related stressors, such as long working hours, are recognized as

254 Environmental stressors, such as pollutants, can increase disease risk

255 e can play a role in mitigating the negative stressors, such as the symptoms of HS, on patients' ment

256 mbination of environmental and psychological stressors superimposed on genetic vulnerability, the neu

257 lenge, and persisted for up to 19 days after stressor termination.

258 rage may be less vulnerable to global change stressors than ancient counterparts.

259 crophage ER stress responses triggered by ER-stressors thapsigargin or tunicamycin.

260 died using social disruption (SDR), a social stressor that involves repeated social defeat in subordi

261 The local stressor that most undermines energy acquisition is sedi

262 Repeated social defeat (RSD) is a murine stressor that recapitulates key physiological, immunolog

263 hough this endosomal pathway is activated by stressors that also activate mitochondrial autophagy, en

264 and exponential functional relationships to stressors that could cause a nonlinear decrease in seagr

265 d on the spill were exposed to toxicants and stressors that could lead to adverse effects.

266 We discuss multiple stressors that could reinforce and prolong the effect of

267 Organisms are repeatedly exposed to stressors that disrupt protein homeostasis (proteostasis

268 pret this as evidence that the environmental stressors that limit tree height also act as ecological

269 important response of animal microbiomes to stressors that reduce the ability of the host or its mic

270 ty and may be a compensatory response to the stressors that this projection is experiencing.

271 amined cortisol responses to an experimental stressor, the Trier Social Stress Test (TSST), in 208 of

272 tragenerational purging, whereas for abiotic stressors, there appeared to be an interaction between p

273 But if a woman had prior day stressors, these meal-related differences disappeared-be

274 ightening their responsiveness to subsequent stressors, this enhanced sensitivity probably increases

275 n particular the ability to quickly quantify stressor thresholds in conjunction with dynamic compensa

276 Targeting resettlement-related stressors through augmenting psychosocial care programme

277 While the epigenome remains sensitive to stressors throughout life, we propose two key principles

278 regarding the power of chronic environmental stressors to accelerate biological aging across multiple

279 makes it possible for childhood psychosocial stressors to affect immune system development, which in

280 tivity are widely considered to be the major stressors to ocean ecosystems induced by emissions of CO

281 g the partial contributions of each of these stressors to programming of cardiovascular dysfunction.

282 host cells links a chemotherapeutic agent-a stressor-to immune modulation and cancer metastasis.

283 Exposure to environmental stressors, toxicants, and nutrient deficiencies can affe

284 eas extra-hypothalamic CRH has a key role in stressor-triggered behaviors.

285 Catalase was induced by the canonical ER stressor, tunicamycin, and by I/R in cardiac myocytes fr

286 in ways not directly predictable from single stressors, underscoring the complex and trait-specific m

287 Higher total cortisol output following a stressor was associated with increased depression severi

288 In the greenhouse, a stressor was introduced by soil infestation with western

289 fects development by acting as an early life stressor, we examined birthweight, litter size, maternal

290 s of cortisol responses to a prolonged acute stressor, we identified three distinct trajectories of c

291 Here, in mice more vulnerable to stressors, we demonstrate structural plasticity within t

292 lls treated with four types of mitochondrial stressors, we identify activating transcription factor 4

293 in phytoplankton in response to a variety of stressors were also evaluated.

294 entially traumatic events and post-migration stressors were positively associated with PTSD and sever

295 Both mortality and extraneous stressors were potentially contributing to this inconsis

296 Precipitating emotional stressors were reported in 48.3% and physical stressors

297 est that post-migration resettlement-related stressors were the most important correlates of mental h

298 neuroendocrine-immune responses to prenatal stressors, which adversely impact neurodevelopment.

299 lished protocols, one that combines a social stressor with physical alcohol cue exposure, and one tha

300 tem is continuously exposed to environmental stressors with non-linear interrelationships between abi