ライフサイエンスコーパス: subcapsular

1 lassified as nuclear, cortical, or posterior subcapsular.

2 eneously, 109 had a smooth surface, 101 were subcapsular, 89 had ill-defined margins, and 62 had a ce

3 As opposed to the clear DNA-free subcapsular and cortical areas of young adult mouse lens

4 In aged mice, most subcapsular and cortical cataracts colocalize with accum

5 nset of heart failure and the development of subcapsular and cortical cataracts is observed in Mbnl3(

6 vital dyes, to determine whether age-related subcapsular and cortical cataracts were linked to the fa

7 oped that initially appeared to be posterior subcapsular and gradually matured to involve the entire

8 lipopolysaccharide, were very visible in the subcapsular and medullary sinuses but were largely exclu

9 a recta in the renal medulla, the lymph node subcapsular and medullary sinuses, and some capillaries

10 ly associated with the severity of posterior subcapsular and nuclear cataracts, which was the only fe

11 immature nuclear sclerotic, mature posterior subcapsular, and mature nuclear.

12 s; nanoparticles were found in the cortical, subcapsular, and medullary sinuses.

13 NK cells located in the subcapsular area exhibited reduced motility and were fou

14 sults in the accumulation of NK cells in the subcapsular area of the draining lymph node and their ac

15 gions in the medullary, interfollicular, and subcapsular areas where viral infection was initially co

16 itating reticulum cells of T zones, cells in subcapsular areas, and cells of the reticular network we

17 rotic diseases in the lens, such as anterior subcapsular cataract (ASC) formation.

18 ciated with a higher prevalence of posterior subcapsular cataract (PSC) (OR, 1.29; 95% CI, 1.07-1.55)

19 te a possible relationship between posterior subcapsular cataract (PSC) formation and expression of t

20 cities formed during recovery from posterior subcapsular cataract (PSC) in Royal College of Surgeons

21 presence of cortical, nuclear, or posterior subcapsular cataract (PSC) opacification in at least one

22 were correlated to the severity of posterior subcapsular cataract (r = 0.4, P = .0006).

23 ty, except for variable mild local posterior subcapsular cataract and local retinal toxicity with hig

24 lation was evident between risk of posterior subcapsular cataract and size at birth.

25 95% CI, 1.27-2.87; P = 0.002); and posterior subcapsular cataract increase of 5% or more versus less

26 aract, and any role in cortical or posterior subcapsular cataract is scarcely measurable.

27 ers (OR, 1.28; 95% CI, 0.79-2.08); posterior subcapsular cataract occurred in 3.0% of statin users an

28 I 0.96-1.63) cataracts, but not to posterior subcapsular cataract or cataract surgery.

29 act, 1.95 (95% CI: 0.48, 7.95) for posterior subcapsular cataract, 1.82 (95% CI: 0.91, 3.66) for soft

30 95% CI, 0.78-1.65; P = 0.519); and posterior subcapsular cataract, 3.05 (95% CI, 1.79-5.19; P < 0.001

31 The associations of lens features (posterior subcapsular cataract, nuclear color, nuclear white scatt

32 .82, 95% CI: 0.68, 0.97; primarily posterior subcapsular cataract, RR = 0.90, 95% CI: 0.71, 1.13).

33 ear white scatter, cortical spokes, anterior subcapsular cataract, vacuoles, waterclefts, coronary fl

34 iations were found between PXS and posterior subcapsular cataract.

35 n 50% of NF2 patients also develop posterior subcapsular cataracts (PSCs).

36 es regarding the pathophysiology of anterior subcapsular cataracts secondary to posterior chamber pIO

37 Posterior subcapsular cataracts were present in 97.5% of patients.

38 ar incident nuclear, cortical, and posterior subcapsular cataracts, but was related to incident catar

39 .23-2.27), but not to cortical and posterior subcapsular cataracts.

40 t laxity, skin hyperelasticity and bilateral subcapsular cataracts.

41 ce from the four families exhibited anterior subcapsular cataracts.

42 raphs were graded for cortical and posterior subcapsular cataracts.

43 resembled the plaques seen in human anterior subcapsular cataracts.

44 al dominant "progressive childhood posterior subcapsular" cataracts segregating in a white family to

45 We observed that mice with adrenal subcapsular cell hyperplasia (SCH), a common histologica

46 is triggered by the interaction of bacterial subcapsular components and bone marrow-derived DC (likel

47 fr2(UB-/-) kidneys have abnormally thickened subcapsular cortical stromal mesenchyme.

48 th hydrocortisone induced both medullary and subcapsular cortical TE cells to express CK6, a differen

49 gitidis and that in each case, the bacterial subcapsular domain markedly influences the Ig response t

50 osition and/or architecture of the bacterial subcapsular domain.

51 mains unresolved whether different bacterial subcapsular domains can exert differential effects on PS

52 sues, high endothelial venules and basal and subcapsular epithelia are CD164 class II-positive, while

53 le negative thymoctyes and laminin 5 made by subcapsular epithelial cells is required for the surviva

54 to the cell surface of thymic medullary and subcapsular epithelium.

55 differentiating fibers of the bow region and subcapsular fibers of the central zone, whereas the lens

56 oneal implants, intratumoral hemorrhage, and subcapsular fluid, showed a significant association with

57 When grown as a subcapsular graft, the Gli2(-/-) UGS exhibited prostatic

58 icities of these two important streptococcal subcapsular group polysaccharides to fully understand th

59 CT showed a voluminous subcapsular hematoma compressing the hepatic parenchyma,

60 s, ureteropelvic junction obstruction, renal subcapsular hematoma, cholelithiasis, medullary calcinos

61 acute liver failure occurs in patients with subcapsular hematoma.

62 including abruptio placentae, renal failure, subcapsular hematomas, and hepatic rupture.

63 Gross liver lesions, characterized by subcapsular hemorrhages or enlargement of the right inte

64 l vascular abnormalities, including aberrant subcapsular hepatic veins, enlarged glomeruli, intestina

65 murine model of chronic kidney disease, with subcapsular hydrogel injections acting as a delivery dep

66 aft model of kidney cancer, characterized by subcapsular implantation of Caki-1 clear cell human kidn

67 Moreover, renal restricted subcapsular infusion of Snx5-specific siRNA (vs. mock si

68 ix pancreatic cancer cell lines and a spleen subcapsular inoculation nude mouse model were also used.

69 ion although they survived longer than renal subcapsular islet allografts.

70 The renal subcapsular islet graft was easily detectable on T2*-wei

71 ll to glucose challenge, comparable to renal subcapsular islet grafts, despite a marginal islet dose,

72 4 to prevent recurrence of diabetes in renal subcapsular islet isografts in DR-BB (RT1uu) rats with e

73 awbacks of the conventional method of kidney subcapsular islet transplantation.

74 normoglycemia faster than animals with renal subcapsular islet transplants.

75 tolerance of intratesticular, but not renal subcapsular, islet allografts.

76 onsive (>120 days) Lewis recipients of renal subcapsular islets underwent nephrectomy of the islet be

77 ed reading center for cortical and posterior subcapsular lens opacities and for AMD severity.

78 age-related nuclear, cortical, and posterior subcapsular lens opacities.

79 ted with an increased risk of mild posterior subcapsular lens opacity development.

80 receptor CCR7 for their transition from the subcapsular lymph node sinus into the parenchyma, a migr

81 Depletion of subcapsular macrophages (SCMsmall ef, Cyrillic) or abrog

82 affected by the loss of F4/80(+) or CD169(+) subcapsular macrophages.

83 d.HGF-transduced NHP islets in vivo, a renal subcapsular marginal mass islet transplant model was dev

84 CD169(+) SIGNR1(+) subcapsular medullary macrophages are the primary cells

85 (2) Subcapsular necrotic areas in the liver suggestive of pr

86 ocal inflammation, and large grossly visible subcapsular necrotic foci.

87 anticipated from earlier studies, posterior subcapsular, nuclear, and cortical cataracts were associ

88 d to UVB exhibited accelerated anterior lens subcapsular opacification, which was more pronounced in

89 ngle-strand breaks, as well as lens anterior subcapsular opacification.

90 aract (RR = 1.39), and preexisting posterior subcapsular opacities (RR = 6.67).

91 d cortical opacities as grade > or = 0.5 and subcapsular opacities as grade > or =0.3 of the Lens Opa

92 Cortical and posterior subcapsular opacities increased with age, but scores for

93 tations, 2 eyes had progression of posterior subcapsular opacities, although neither required surgery

94 graded for nuclear, cortical, and posterior subcapsular opacities, and the amount of liquid vitreous

95 iring nearwork, nuclear opacities, posterior subcapsular opacities, glaucoma, and ocular hypertension

96 tical opacity as grade >/=1.0, and posterior subcapsular opacity as grade >/=0.5 according to the Len

97 OL showed either stage 5 (complete posterior subcapsular opacity) or stage 6 (mature) cataracts, wher

98 ryl CoA reductase inhibitor, developed frank subcapsular opacity.

99 (OR, 1.57; 95% CI, 1.13-2.20) and posterior subcapsular (OR, 1.73; 95% CI, 1.10-2.72) cataract, but

100 stant cell renewal, involving recruitment of subcapsular progenitors to ZG fate and subsequent lineag

101 graded for nuclear, cortical, and posterior subcapsular (PSC) cataract, following the Wisconsin Cata

102 presence of nuclear, cortical, or posterior subcapsular (PSC) cataract, from standardized grading of

103 , including nuclear, cortical, and posterior subcapsular (PSC) cataract.

104 cipants had nuclear, cortical, and posterior subcapsular (PSC) cataracts, respectively.

105 the odds of developing cortical or posterior subcapsular (PSC) cataracts.

106 entage involvement of cortical and posterior subcapsular (PSC) lens opacities within the central 5 mm

107 viduals with nuclear, cortical, or posterior subcapsular (PSC) opacities and individuals with no cata

108 ens for presence of nuclear (NSC), posterior subcapsular (PSC), and cortical cataract (CC), using the

109 tes assemble in clusters in the cords of the subcapsular red pulp and are distinct from macrophages a

110 lomeruli identified located in the immediate subcapsular region (P<0.001).

111 nsplanted, but most biopsies sample only the subcapsular region and may not accurately represent the

112 revealed laminin 5 expression mostly in the subcapsular region of the adult thymus.

113 Gp96 accesses the subcapsular region of the draining lymph node, and it is

114 ghout the cortex rather than confined to the subcapsular region of the thymus.

115 butes to pre-T cell development, as does the subcapsular region of the thymus.

116 onized by newly produced antibodies from the subcapsular region to the germinal center, and affinity

117 Naive T lymphocyte locomotion in the subcapsular region was 38% slower and had higher turning

118 a predominance of sclerosis in the kidney's subcapsular region, the area predominantly sampled by th

119 ry junction in lymphoid follicles and in the subcapsular region.

120 MBs were larger and more numerous in the subcapsular region.

121 /delta TCR expression especially high in the subcapsular region.

122 elop cataracts in the anterior and posterior subcapsular regions as well as punctate opacities in the

123 n DCs from the early stage in the lymph node subcapsular regions, and COX-2 inhibition markedly suppr

124 lant survival and blood glucose control in a subcapsular renal graft model in immuno-incompetent diab

125 The tissue recombinants were grown as subcapsular renal grafts and treated from the time of gr

126 tain innate-like lymphocytes that survey the subcapsular sinus (SCS) and associated macrophages for p

127 CD169+MHCII+ macrophages on the floor of the subcapsular sinus (SCS) and in the medulla of lymph node

128 The layer of macrophages at the subcapsular sinus (SCS) captures pathogens entering the

129 inate via lymphatics and preferentially bind subcapsular sinus (SCS) CD169(+) macrophages in tumor-dr

130 ) alpha1beta2, which maintained a protective subcapsular sinus (SCS) macrophage phenotype within viru

131 were found associated with CD169(+)-positive subcapsular sinus (SCS) macrophages and collagen fibers.

132 Subcapsular sinus (SCS) macrophages capture antigens fro

133 y than naive T cells, relocalized toward the subcapsular sinus (SCS) near invaded macrophages, and en

134 b(+)CD169(+) macrophages, which populate the subcapsular sinus (SCS) of LNs, are critical for the cle

135 in-water emulsion adjuvant MF59 localizes in subcapsular sinus and medullary macrophage compartments

136 Our findings identify macrophages lining the subcapsular sinus as an important site of B cell encount

137 , small numbers of beads were present in the subcapsular sinus as early as 6 h after inhalation.

138 CD1d-dependent manner in close proximity to subcapsular sinus CD169(+) macrophages.

139 Depletion of subcapsular sinus CD169-positive macrophages by clodrona

140 ng by lymphatic endothelial cells lining the subcapsular sinus ceiling stabilizes interfollicular CCL

141 borne antigens and chemoattractants from the subcapsular sinus directly to the B cell follicles.

142 ure that macrophages of the mouse lymph node subcapsular sinus facilitate B cell activation in vivo b

143 broblastic reticular cells that connects the subcapsular sinus floor and the HEVs by intertwining wit

144 In contrast, subcapsular sinus macrophages (SSMs) exposed to lymph-bo

145 t contain the unprocessed Ag are captured by subcapsular sinus macrophages and are transferred onto f

146 resulting from S. flexneri interactions with subcapsular sinus macrophages and dendritic cells, and r

147 By contrast, large antigens were bound by subcapsular sinus macrophages and subsequently transferr

148 culate antigens relies on antigen capture by subcapsular sinus macrophages of the lymph node.

149 cate that NK-cell recruitment is mediated by subcapsular sinus macrophages, IFN-gamma, and CXCR3 duri

150 acquisition did not require dendritic cells, subcapsular sinus macrophages, or B cell movement to the

151 nate-loaded liposomes, indicating a role for subcapsular sinus macrophages.

152 cles must be translocated into follicles via subcapsular sinus macrophages.

153 rticulate antigens and large IC are bound by subcapsular sinus macrophages.

154 s (Mo) and B cell areas in the spleen and to subcapsular sinus Mo in lymph nodes of naive mice (CR-Fc

155 ymph node, exosomes were not retained in the subcapsular sinus of CD169(-/-) mice but penetrated deep

156 cells were identified in the paracortex and subcapsular sinus of the draining internal iliac lymph n

157 n the marginal zone of the spleen and in the subcapsular sinus of the lymph node.

158 ith the removal of macrophages that line the subcapsular sinus of the lymph node.

159 in the DLN, most of the LPS was found in the subcapsular sinus or medulla, near or within lymphatic e

160 antigens diffuse directly from lymph in the subcapsular sinus to be acquired by antigen-specific B c

161 rt of small antigens and chemokines from the subcapsular sinus to follicular B cells.

162 ymph-borne soluble molecules travel from the subcapsular sinus to the HEVs is unclear.

163 luding chemokines, traveled rapidly from the subcapsular sinus to the HEVs using the reticular networ

164 njection, first in the region closest to the subcapsular sinus where lymph enters the lymph node.

165 endothelial cells lining the ceiling of the subcapsular sinus, but not those lining the floor, expre

166 luorophore-labeled molecules highlighted the subcapsular sinus, the reticular fibers, and the ablumin

167 h the lymph to macrophages in the lymph node subcapsular sinus.

168 tion of lymphoid stromal cells lining the LN subcapsular sinus.

169 vessels at the junction with the lymph node subcapsular sinus.

170 ion into the LN parenchyma from lymph in the subcapsular sinus.

171 sinus macrophages, or B cell movement to the subcapsular sinus.

172 spite the microspheres being confined to the subcapsular sinus.

173 and infected cells residing just beneath the subcapsular sinus.

174 tor 1 (S1P1)-expressing CD68+ macrophages in subcapsular sinuses of FTY-P-treated MLNs.

175 cessed islets were transplanted at the renal subcapsular site in rats.

176 In addition, the spatially limited renal subcapsular site restricts the mass of islet tissue that

177 r allografts were transplanted to the kidney subcapsular site.

178 st, porcine islets transplanted to the liver subcapsular space do not survive, although autologous is

179 2500, were transplanted into the left renal subcapsular space of a syngeneic adult mouse made diabet

180 -2d) islets were transplanted into the renal subcapsular space of diabetic c-Rel-/- C57BL/6 (H-2b) mi

181 ncreatic islets of Langerhans into the renal subcapsular space of immunodeficient BALB/c.rag2(-/-).cg

182 regeneration of prostatic structures in the subcapsular space of the kidney was observed within 4-8

183 ically, its expression was restricted to the subcapsular space of the LN during early inflammation, w

184 mokidney by thymic autografting to the renal subcapsular space results in normal thymic growth and fu

185 Control dogs received liver subcapsular space transplants of porcine islets and auto

186 n as few as 10 islets implanted in the renal subcapsular space, intrahepatic, intraabdominal, and sub

187 re recovered and transplanted into the renal subcapsular space.

188 cells flowed from medullary sinuses into the subcapsular space.

189 sorganized TEBs characterized by exaggerated subcapsular spaces, breaks in basal lamina, dissociated

190 These results suggest the presence of common subcapsular surface antigens, such as outer membrane pro

191 m-immunosuppressed TMX recipients with renal subcapsular syngeneic thymic grafts.

192 cified schedule starting 2 days before renal subcapsular transplantation of an islet isograft.

193 Twelve (75%) of 16 hemangiomas were subcapsular, two (12%) of 16 demonstrated exophytic grow

194 The nuclear, cortical, and posterior subcapsular types of cataracts did not show different re

195 fat planes obscured, retroperitoneal fluid (subcapsular vs extracapsular), ascites beyond the cul-de

196 tes downregulate CCR9 and migrate toward the subcapsular zone where they recombine their TCR beta-cha

197 across the cortex before accumulation in the subcapsular zone.