ライフサイエンスコーパス: sulfite

1 ancement increased with the concentration of sulfite.

2 s showed sensitivity and overaccumulation of sulfite.

3 mproved by 29 times in the presence of 20 mM sulfite.

4 kely metabolite of thiosulfate assimilation, sulfite.

5 reflecting a decrease in the ability to bind sulfite.

6 f ammonium with o-phthaldialdehyde (OPA) and sulfite.

7 ains that function together to reduce APS to sulfite.

8 ty to form complexes with glycine betaine or sulfite.

9 donor for the reduction of APS to 5'-AMP and sulfite.

10 for methanogenesis and a possible target for sulfite.

11 nversion of taurine to aminoacetaldehyde and sulfite.

12 rapidly oxidised sulfide to thiosulfate and sulfite.

13 ts dibenzothiophene to 2-hydroxybiphenyl and sulfite.

14 ) during the free radical chain oxidation of sulfite.

15 fide and unexpectedly, when it is exposed to sulfite.

16 t rate by reaction with sulfide but not with sulfite.

17 which is then partitioned to thiosulfate or sulfite.

18 chloride/base, which initially forms cyclic sulfite 10.

19 reacts with OPA and forms in the presence of sulfite a product, which can be detected by spectrophoto

20 y displacement of the endogenous ligand with sulfite, a less tightly bound competing ligand.

21 nderstood, particularly for the reduction of sulfite-a key intermediate in the pathway.

22 e reductase was enhanced, leading to further sulfite accumulation in SIR Ri plants.

23 in protecting leaves against the toxicity of sulfite accumulation.

24 These sulfite addition reactions, which are slowly reversible

25 ic red wines produced without sulfur dioxide/sulfites addition are comparable to conventional red win

26 ic red wines produced without sulfur dioxide/sulfites addition in comparison to those of eight conven

27 udies (stabilizations of the covalent FAD-N5-sulfite adduct and p-quinonoid form of 8-mercapto-FAD),

28 model wine was present in free and hydrogen sulfite adduct forms and the measured total, free and pe

29 reactivity, PutA(Hh) forms a reversible FAD-sulfite adduct.

30 The presence of a prFMN sulfite-adduct in one of the UbiD crystal structures con

31 nd Mn(II) caused damage to DNA while neither sulfite alone nor metal ions alone did have the same eff

32 lly, the effect of the reducing agent sodium sulfite also was evaluated to characterize the nature of

33 Sulfite, an inhibitor of oxygen reduction, is bound at t

34 and (v) sequential elimination via a cyclic sulfite and a cyclobutyl triflate.

35 the two-electron reduction of APS and forms sulfite and adenosine 5'-monophospahate (AMP).

36 her reactive sulfur species (RSS), including sulfite and bisulfite, as well as sulfane sulfur species

37 vasculature system, where it is reduced into sulfite and finally sulfide within the subcellular organ

38 transfer in the direction of thiosulfate to sulfite and glutathione persulfide; sulfur transfer in t

39 everely impaired both in the ability to bind sulfite and in catalysis, with a second-order rate const

40 ctivity of human rhodanese to cyanide versus sulfite and might be important in differences in suscept

41 Other sulfite compounds, namely sodium sulfite and potassium metabisulfite, also promote the li

42 sulfite-sensitive mutants accumulate applied sulfite and show a decline in glutathione levels.

43 suggests an efficient oxidation pathway via sulfite and sulfate radical anions on droplets possibly

44 oxidize sulfide (H2 S, HS(-) and S(2-) ) to sulfite and thiosulfate via polysulfide.

45 ection for simultaneous analysis of sulfate, sulfite, and chloride in human urine, plasma, and sweat

46 ple thiophilic acceptors, including sulfide, sulfite, and glutathione, to form as products, hydrodisu

47 n and to 2.4 A resolution in the presence of sulfite, and the C185A variant to 2.8 A resolution.

48 gradation rate of MO was greatly enhanced by sulfite, and the enhancement increased with the concentr

49 ent dissociation constants for NAD(+), NADH, sulfite, and the sulfite-NAD(+) adduct.

50 spontaneously desulfinated to succinate and sulfite; and (iii) whereas succinate enters the central

51 icals generated by the reactions of sulfite (sulfite anions or bisulfite anions) with holes or hydrox

52 ts in maintaining sulfite homeostasis, where sulfite appears to act as an orchestrating signal molecu

53 and activity levels are likewise promoted by sulfite application as compared with water injection con

54 Exogenous sulfite application induced up-regulation of the sulfite

55 Resistance to high sulfite application is manifested by fast sulfite disapp

56 -stressed wild-type plants were resistant to sulfite applications, but SO RNA interference plants sho

57 containing compounds, augmented by exogenous sulfite applications, underlines the role of SO and othe

58 r of their ability to induce DNA damage with sulfite as follows: Fe(III) > Co(II) > Cu(II) > Cr(VI) >

59 hat it is essential for growth on sulfate or sulfite as the sole sulfur source and, further, that the

60 . jannaschii grows and produces methane with sulfite as the sole sulfur source.

61 Oxidation of H(2)S by SQOR with sulfite as the sulfane sulfur acceptor is rapid and high

62 caftaric acid were found in wines containing sulfites as a preservative.

63 linear in the range 0.032-0.320 mg L(-1) of sulfite (as SO2), with a correlation coefficient of 0.99

64 In addition to the BiOBr/MO system, the sulfite-assisted photocatalysis approach has been succes

65 vities of the cell are incapable of reducing sulfite at a rate sufficient to allow growth.

66 Reduction of At-SO with sulfite at high pH generates the well-known high-pH (hpH

67 another low pH form, whereas reduction with sulfite at higher pH values gives a mixture of Species 1

68 Sulfite at the lowest experimentally feasible concentrat

69 NA damage induced by radicals generated from sulfite autoxidation using cyclic voltammetry (CV) and e

70 R is consistent with glutathione rather than sulfite being the predominant acceptor at physiologicall

71 An apparent sulfite-binding pocket at the protein-protein interface

72 An amperometric sulfite biosensor was fabricated using SO(X)/Fe(3)O(4)@G

73 idation and insight into the impact of these sulfite bound carbonyls on antimicrobial and antioxidant

74 ithout increasing concentrations of free and sulfite-bound acetaldehyde.

75 ration from components of the medium (mainly sulfite) but not from sulfate.

76 iderable photocurrent for photo-oxidation of sulfite, but generated significantly reduced photocurren

77 d many bacteria, this compound is reduced to sulfite by APS reductase (APR); in fungi and some cyanob

78 ate (PAPS), is necessary before reduction to sulfite by PAPS reductase (PAPR).

79 The 1,2-eliminations in cyclic carbonate and sulfite by regioselective abstraction of methine protons

80 ytoplasmic membrane for further oxidation to sulfite by the dissimilatory reductase DsrAB is incomple

81 labile enzyme-intermediate before release of sulfite by the protein cofactor thioredoxin.

82 of this species prepared with (33)S-labeled sulfite clearly show the presence of coordinated sulfate

83 wever, dechlorination of reacted N-CNTs with sulfite completely suppresses N-CNT toxicity.

84 rom dissolution at lower temperatures to TMI-sulfite complex formation at higher temperatures.

85 Other sulfite compounds, namely sodium sulfite and potassium m

86 However, under pathological conditions when sulfite concentrations are high, sulfite could compete w

87 on, did not correlate with total phenolic or sulfite content.

88 itions when sulfite concentrations are high, sulfite could compete with sulfide for addition to the a

89 e (SO) is found in animals and plants, while sulfite dehydrogenase (SDH) is found in bacteria.

90 ively charged substitution R55K in bacterial sulfite dehydrogenase (SDH).

91 ana plant sulfite oxidase, and the bacterial sulfite dehydrogenase from Starkeya novella.

92 AB is a protein-based trisulfide, in which a sulfite-derived sulfur is bridging two conserved cystein

93 ant SO (PSO) also plays an important role in sulfite detoxification and in addition serves as an inte

94 sulfur dioxide was hydrated and bound as the sulfite dianion in the Zn5 L6 structure.

95 Subsequent in situ oxidation of the sulfite dianion resulted in a sulfate dianion bound with

96 ased sulfate uptake but sulfate reduction to sulfite did not seem to be regulated.

97 Due to the chemical reactivity of sulfite, dilute formaldehyde is used as a reagent to for

98 gh sulfite application is manifested by fast sulfite disappearance and an increase in glutathione lev

99 Herein, we have developed a sulfite-enhanced visible-light-driven photodegradation p

100 o-glycosylation of lithium azide by a cyclic sulfite ester.

101 At pH 6, reduction by sulfite followed by partial reoxidation with ferricyanid

102 ted by sulfide, a precursor to mitochondrial sulfite formation.

103 sulfur from glutathione persulfide (GSSH) to sulfite generating thiosulfate and from thiosulfate to c

104 tially catalyze sulfur transfer from GSSH to sulfite, generating thiosulfate and glutathione.

105 We demonstrate that, in addition to sulfite, glutathione functions as a persulfide acceptor

106 on being: H2S --> glutathione persulfide --> sulfite --> sulfate, than with a more convoluted route t

107 er sulfite network components in maintaining sulfite homeostasis, where sulfite appears to act as an

108 ssible to observe and sometimes quantify the sulfite, hydrate, and acetal forms of the carbonyl compo

109 s PRF is poised to metabolize thiosulfate to sulfite in a sulfur assimilation pathway rather than in

110 ate the disadvantageous presence of residual sulfite in crystal sugar.

111 was evaluated with 96.46% recovery of added sulfite in red wine and 1.7% and 3.3% within and between

112 ults in a covalent adduct between NAD(+) and sulfite in the active site of the enzyme that binds very

113 action method (HS-SDME) for determination of sulfite in the form of sulfur dioxide was developed.

114 It was found that sulfite in the presence of Co(II), Cu(II), Cr(VI), Fe(II

115 oxidation of alkanesulfonate to aldehyde and sulfite in the presence of O2 and FMNH2.

116 in the overall reaction process whereby the sulfite, in the presence of transition metals, may cause

117 imum concentration of a transition metal for sulfite induced DNA damage revealed that electrochemical

118 is also formed when sulfide is added to the sulfite-induced CT intermediate, representing a new mech

119 SiR transcript and activity within 30 min of sulfite injection into Arabidopsis and tomato leaves.

120 rn about food preservation, the reduction of sulfite input plays a major role in the wine industry.

121 te oxidation to sulfate and incorporation of sulfite into sulfoquinovosyl diacylglycerols were not su

122 stance to ectopically applied sulfur dioxide/sulfite is a function of SiR expression levels and that

123 Sulfite is a strong inhibitor of PTDH that is competitiv

124 ts for the removal of water pollutants since sulfite is a waste from flue gas desulfurization process

125 In biological systems, the detoxification of sulfite is catalyzed by the sulfite-oxidizing enzymes (S

126 eas succinate enters the central metabolism, sulfite is detoxified by the previously identified putat

127 Sulfite is inhibitory to the methanogens.

128 The higher sulfite is likely the main reason for the initiation of

129 The results suggest that sulfite is rapidly autoxidized in the presence of Co(II)

130 s a role in the protection of plants against sulfite is supported by the rapid up-regulation of SiR t

131 We propose that sulfite is the physiological acceptor of the sulfane sul

132 1A mutation causing a 5-fold increase in the sulfite K(m) value, perhaps reflecting a decrease in the

133 sis thaliana has been reduced at pH = 6 with sulfite labeled with 33S (nuclear spin I = 3/2), followe

134 Biosensor measured sulfite level in red and white wines.

135 conditions, the sulfite network can control sulfite levels in the absence of SO activity.

136 Little is known about the homeostasis of sulfite levels, a cytotoxic by-product of plant sulfur t

137 ls were not sufficient to maintain low basal sulfite levels, resulting in accumulative leaf damage in

138 is necessary to cope with rising endogenous sulfite levels.

139 at it has excellent electrocatalysis towards sulfite, lower detection limit, higher storage stability

140 Among these, sulfites may induce adverse effects after ingestion.

141 re found for specific Kraft, mechanical, and sulfite mills, suggesting yet unidentified causative age

142 constants for NAD(+), NADH, sulfite, and the sulfite-NAD(+) adduct.

143 However, under nonstressed conditions, the sulfite network can control sulfite levels in the absenc

144 cations, underlines the role of SO and other sulfite network components in maintaining sulfite homeos

145 opersicum) wild-type leaves, while the other sulfite network components were down-regulated.

146 ic pathways, we followed key elements of the sulfite network enzymes that include adenosine-5'-phosph

147 f nitrite, which need not be the case in the sulfite/nitrite reductase family.

148 ell-known stimulatory effect of the oxyanion sulfite on MgATP hydrolysis.

149 Sulfite, on the other hand, has a significantly less neg

150 ependent oxidation of S degrees , sulfide or sulfite or H(2)oxidation.

151 Cyanide, sulfite, or sulfide can act as the sulfane sulfur accept

152 nter of the pathogenic R160Q mutant of human sulfite oxidase (hSO) confirms the presence of three dis

153 dies on the pathogenic R160Q mutant of human sulfite oxidase (HSO) have shown that Mo-heme intramolec

154 hotosystem I, cytochrome c (cyt c) and human sulfite oxidase (hSOX).

155 A sulfite oxidase (SO(X)) (EC 1.8.3.1) purified from Syzyg

156 Sulfite oxidase (SO) catalyzes the physiologically criti

157 8D were identified in patients with isolated sulfite oxidase (SO) deficiency, and the equivalent amin

158 Dimethylsulfoxide reductase (DMSOR) and sulfite oxidase (SO) families were the most widespread m

159 Sulfite oxidase (SO) is a vitally important molybdenum e

160 Sulfite oxidase (SO) is found in animals and plants, whi

161 The Mo(V) state of the molybdoenzyme sulfite oxidase (SO) is paramagnetic and can be studied

162 sulfate reductase and the sulfite scavengers sulfite oxidase (SO), sulfite reductase, UDP-sulfoquinov

163 relate to the reduced and oxidized forms of sulfite oxidase (SO).

164 Sulfite oxidase (SUOX) expression and the drug-transport

165 o either the xanthine dehydrogenase (XDH) or sulfite oxidase (SUOX) families, and these have pyranopt

166 ing partial misfolding and monomerization of sulfite oxidase and attenuating both substrate binding a

167 persulfide dioxygenase (PDO), rhodanese, and sulfite oxidase and converts H2S to thiosulfate and sulf

168 Saccharomyces cerevisiae lacking sulfite oxidase and deleted of flavohemoglobin showed an

169 ed the characteristic absorption spectrum of sulfite oxidase and exhibited steady state and rapid kin

170 The physiological implications of plant sulfite oxidase as a copious generator of superoxide are

171 onance Raman spectra of oxidized A. thaliana sulfite oxidase catalytically cycled in both H2(16)O and

172 We synthesized the gene for chicken sulfite oxidase de novo, working backward from the amino

173 Isolated sulfite oxidase deficiency (ISOD) causes severe intellec

174 In humans, sulfite oxidase deficiency is an inherited recessive dis

175 tations identified in patients with isolated sulfite oxidase deficiency, the G473D variant is of part

176 for confirmatory testing of cystic fibrosis, sulfite oxidase deficiency, urolithiasis, and other diso

177 crystal structures of the wild type and the sulfite oxidase deficiency-causing R138Q (R160Q in human

178 affecting the type of reactions catalyzed by sulfite oxidase family enzymes.

179 Plant sulfite oxidase from Arabidopsis thaliana has been chara

180 Sulfite oxidase from Arabidopsis thaliana has been reduc

181 Several point mutations in the sulfite oxidase gene have been identified from patients

182 xidases and the failure to clone the chicken sulfite oxidase gene.

183 ent response of the catalytic cycle of human sulfite oxidase immobilized on an electrode.

184 0Q in humans) variant of recombinant chicken sulfite oxidase in the resting and sulfate-bound forms.

185 Analysis of recombinant G473D sulfite oxidase indicated that it is severely impaired b

186 The crystal structure of chicken liver sulfite oxidase indicated that this residue, Cys185 in c

187 of the wild-type dimeric state of mammalian sulfite oxidase is not yet well understood.

188 te forms of the molybdenum-containing enzyme sulfite oxidase possess a b-type cytochrome prosthetic g

189 Peroxisomal sulfite oxidase transcripts and activity levels are like

190 sulfite oxidase, Arabidopsis thaliana plant sulfite oxidase, and the bacterial sulfite dehydrogenase

191 remarkably similar to that found in chicken sulfite oxidase, Arabidopsis thaliana plant sulfite oxid

192 f this residue in the catalytic mechanism of sulfite oxidase, serine and alanine variants at position

193 subsequent reconstitution of MoCo-free human sulfite oxidase-molybdenum domain yielding a fully activ

194 rsulfide dioxygenase (ETHE1), rhodanese, and sulfite oxidase.

195 is remarkably similar to the low-pH form of sulfite oxidase.

196 calculations are extended to oxo transfer by sulfite oxidase.

197 s in crystallizing recombinant human and rat sulfite oxidases and the failure to clone the chicken su

198 Sulfite oxidases are metalloenzymes that oxidize sulfite

199 Sulfite oxidases have been wired to electrode surfaces,

200 s the reason for the decrease in activity of sulfite oxidases upon immobilization.

201 In vertebrate sulfite oxidases, the electrons generated at the Mo cent

202 We found that sulfite oxidation mediated by EPO/H(2)O(2) induced the f

203 Increased sulfite oxidation to sulfate and incorporation of sulfit

204 indicate that neither variant is capable of sulfite oxidation.

205 Sulfite oxidizing enzymes (SOEs) are molybdenum cofactor

206 ell-known high-pH (hpH) signal common to all sulfite oxidizing enzymes.

207 etoxification of sulfite is catalyzed by the sulfite-oxidizing enzymes (SOEs), which interact with an

208 similar results were observed for samples of sulfite-oxidizing enzymes from other organisms that were

209 All reported sulfite-oxidizing enzymes have a conserved arginine in t

210 sis of the possible mechanistic pathways for sulfite-oxidizing enzymes is presented and related to av

211 The DNA damage induced by sulfite plus Co(II), Cr(VI), and Fe(III) was inhibited b

212 ons were measured by HPLC using a stable OPA/sulfite precolumn derivatization and an electrochemical

213 ed wild-type plants, while expression of the sulfite producer, adenosine-5'-phosphosulfate reductase,

214 subsequent steps by thioredoxin to yield the sulfite product.

215 n rapidly oxidise sulfide to thiosulfate and sulfite, providing the foundation for using heterotrophi

216 Studies using hole scavengers suggest that sulfite radicals generated by the reactions of sulfite (

217 It is suggested that at low pH the sulfite-reduced At-SO has coordinated sulfate and is in

218 s the presence of coordinated sulfate in the sulfite-reduced low-pH form of the plant enzyme.

219 f infected oat tissue homogenate with sodium sulfite reduces transmission of the purified virus by ap

220 The combination of sulfite reducing Clostridium spp. with SRB may improve g

221 lic processes, and profiles of dissimilatory sulfite reductase (dsr) transcripts are consistent with

222 CR targeting the 16S rRNA, dissimilatory (bi)sulfite reductase (dsrAB), and dissimilatory arsenate re

223 and gamma subunits of reverse dissimilatory sulfite reductase (rdsr).

224 Assimilatory NADPH-sulfite reductase (SiR) from Escherichia coli is a struc

225 Sulfite reductase (SiR) is an essential enzyme of the su

226 Plant sulfite reductase (SiR; Enzyme Commission 1.8.7.1) catal

227 Deletion of sulfite reductase (sirA, originally misannotated nirA) r

228 The consequences of reduced sulfite reductase activity in particular are exacerbated

229 s to grow slowly on metabolites that require sulfite reductase activity.

230 ductase), as well as cytosolic Fe-S enzymes (sulfite reductase and isopropylmalate isomerase).

231 oad similarity to the hemoprotein subunit of sulfite reductase but has many significant differences i

232 Sulfite reductase catalyzes the six-electron reduction o

233 flavin oxidoreductase component of the CysJI sulfite reductase complex (CysJ(8)I(4)), we show that th

234 sion with sequence similarity to the nitrite/sulfite reductase family.

235 The initial rate parameters for the purified sulfite reductase from M. tuberculosis were determined u

236 t may interact with HdrABC and dissimilatory sulfite reductase gamma subunit (DsrC) to perform novel

237 hic patterns of the functional dissimilatory sulfite reductase gene (dsrA) and the 16S rRNA gene in s

238 ce a sulfate transporter mutant strain and a sulfite reductase mutant strain are fully virulent.

239 ences or could arise from alterations of the sulfite reductase structure that arise from the isolatio

240 dentification of virus-encoded dissimilatory sulfite reductase suggests SUP05 viruses reprogram their

241 ase), or by Escherichia coli cysJI (encoding sulfite reductase).

242 e and of dsrC, associated with dissimilatory sulfite reductase).

243 dehydrogenase, a methanogenesis enzyme, and sulfite reductase, a detoxification enzyme.

244 -terminal half a dissimilatory-type siroheme sulfite reductase, and Fsr catalyzes the corresponding p

245 s coding for DsrAB, the enzyme dissimilatory sulfite reductase, inevitably also contain the gene codi

246 ing the nitrite reductases and dissimilatory sulfite reductase, respectively.

247 the sulfite scavengers sulfite oxidase (SO), sulfite reductase, UDP-sulfoquinovose synthase, and beta

248 cation is unique and independent of CysI and sulfite reductase.

249 first report of a coenzyme F(420)-dependent sulfite reductase.

250 ain late evolving archaea, and dissimilatory sulfite reductases of bacteria and archaea.

251 Like their nitrite reductase counterparts, sulfite reductases require a siroheme cofactor for catal

252 Previously described sulfite reductases use nicotinamides and cytochromes as

253 anaerobic S(0) reduction, anaerobic sulfate/sulfite reduction and anaerobic respiration of organic s

254 acterial system, we show that the product of sulfite reduction by DsrAB is a protein-based trisulfide

255 C biosynthesis and that pseudomonads utilize sulfite reduction enzymology distinct from that of E. co

256 Sulfite reduction in Mycobacterium smegmatis was investi

257 Sulfite reduction is one such mycobacterium-specific ste

258 ic acid cycle, NAD biosynthesis, nitrate and sulfite reduction, and CO2/CO fixation.

259 was due to deficient expression of fprA and sulfite reduction.

260 rence plants lacking SO activity accumulated sulfite, resulting in leaf damage and mortality.

261 enzyme with NAD(+) and low concentrations of sulfite results in a covalent adduct between NAD(+) and

262 ite application induced up-regulation of the sulfite scavenger activities in dark-stressed or unstres

263 denosine-5'-phosphosulfate reductase and the sulfite scavengers sulfite oxidase (SO), sulfite reducta

264 od for measuring APR activity by using novel sulfite-selective colorimetric or "off-on" fluorescent l

265 The sulfite-sensitive mutants accumulate applied sulfite and

266 tion, sulfide serves as the sulfur donor and sulfite serves as the acceptor, forming thiosulfate.

267 produce GSSH; PDO oxidises GSSH to sulfite; sulfite spontaneously reacts with polysulfides to genera

268 as a result of these mutations; however, the sulfite-stimulated activity decreased by more than 60%.

269 consequence of SiR impairment, the levels of sulfite, sulfate, and thiosulfate were higher and glutat

270 ase is capable of being regenerated from its sulfite/sulfate heat stable salt, which enables the simu

271 lfite radicals generated by the reactions of sulfite (sulfite anions or bisulfite anions) with holes

272 thione to produce GSSH; PDO oxidises GSSH to sulfite; sulfite spontaneously reacts with polysulfides

273 that in the C185S variant, in the absence of sulfite, the active site residue Tyr322 became disordere

274 Similarly, if chloride is added before sulfite, the lpH species is formed instead of the blocke

275 185S variant crystallized in the presence of sulfite, the Tyr322 residue relocalized to the active si

276 was observed in the presence of graphite and sulfite, thiosulfate, or polysulfides.

277 animals, SO catalyzes the oxidation of toxic sulfite to sulfate as the final step in the catabolism o

278 ite oxidases are metalloenzymes that oxidize sulfite to sulfate at a molybdenum active site.

279 sociated with the AMP-dependent oxidation of sulfite to sulfate), some of which occur in multiple (up

280 enzyme that catalyzes the oxidation of toxic sulfite to sulfate.

281 s the physiologically critical conversion of sulfite to sulfate.

282 mmission 1.8.7.1) catalyzes the reduction of sulfite to sulfide in the reductive sulfate assimilation

283 ve pathway, which catalyzes the reduction of sulfite to sulfide.

284 tase catalyzes the six-electron reduction of sulfite to sulfide.

285 that catalyzes the six-electron reduction of sulfite to sulfide.

286 fonyl derivatives by spontaneous addition of sulfite to UMP and to OMP.

287 The method is based on the conversion of sulfite to volatile sulfur dioxide by acidification of t

288 ts that overexpress SiR are more tolerant to sulfite toxicity, exhibiting little or no damage.

289 chii an anabolic ability and protection from sulfite toxicity.

290 are not mutually exclusive: (a) that sodium sulfite treatment disrupts critical virion-host protein

291 es, some of which were lost following sodium sulfite treatment.

292 CsoR is unreactive toward sulfite under the same conditions.

293 good correlation (r=0.99) between red wines sulfite value by standard DTNB (5,5'-dithio-bis-(2-nitro

294 The kcat/Km,sulfite values for the variants in the sulfur transfer r

295 at values, and the large increases in the Km(sulfite) values, rationalize the fatal impact of these m

296 in the sulfur transfer reaction from GSSH to sulfite were 1.6- (Asp-102) and 4-fold (Ala-285) lower t

297 a more convoluted route that would result if sulfite were the primary acceptor of sulfane sulfur.

298 Succinate and sulfite were verified as the final reaction products.

299 ted the effects of both conventional and low sulfite wines on ex vivo human erythrocytes under oxidat

300 r artifact-free determination of sulfate and sulfite with consistent results for chloride when compar

301 te (APS) to adenosine 5'-phosphate (AMP) and sulfite with reducing equivalents from the protein cofac