ライフサイエンスコーパス: sulfur amino acid

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1 re found for red blood cell folate and other sulfur amino acids.

2 variations associated with dietary intake of sulfur amino acids.

3 indistinguishable from sulfate derived from sulfur amino acids.

4 the terminal reaction in the degradation of sulfur amino acids.

5 ng normal food (protein, 0.8 g kg(-1) d(-1); sulfur amino acids, 20 mg kg(-1) d(-1)) at standardized

6 netics of leucine, phenylalanine, glutamine, sulfur amino acid, and threonine and their relation to w

7 hogen responses, two to proteins involved in sulfur amino acid biosynthesis, and two having significa

8 is sufficient for degradation, and specific sulfur amino acids can promote the degradation by destab

9 Thus, in addition to sulfur amino acid catabolism, ETHE1 also affects the oxi

10 , membrane transport activities of the three sulfur amino acids cysteine, cystine and methionine, and

11 al capacity according to the availability of sulfur amino acids, establishing a functional significan

12 This experiment marks the first synthesis of sulfur amino acids from spark discharge experiments desi

13 ntribution to homocysteine remethylation and sulfur amino acid homeostasis is not known.

14 ntrations were not affected by the amount of sulfur amino acids in the three diets.

15 e cytosolic disulfide-reducing power and all sulfur amino acids in TR/GR-null livers.

16 thionine is the precursor of homocysteine, a sulfur amino acid intermediate in the methylation and tr

17 Vitamin B12, folate, and sulfur amino acids may be modifiable risk factors for st

18 previously underappreciated link between the sulfur amino acid metabolic pathway and obesity and card

19 l homocystinuria (HCU) is an inborn error of sulfur amino acid metabolism caused by deficient activit

20 the physiological significance of BHMT-2 in sulfur amino acid metabolism.

21 BS) deficiency, is the most common defect of sulfur amino acid metabolism.

22 omocystinuria, the most frequent disorder of sulfur amino acid metabolism.

23 Examples of metabolomics evaluations of sulfur amino-acid metabolism in psychiatry, neurology, a

24 The first six deal with the functionality of sulfur amino acids (methionine and cysteine) and related

25 selenomethionine of mutants impaired in the sulfur amino acid pathway, we excluded a toxic effect of

26 thesis are the availability of cysteine, the sulfur amino acid precursor, and the activity of the rat

27 Understanding sulfur amino acid precursor-dependent cellular mechanism

28 the extra- and intracellular equilibrium of sulfur amino acids, resulting in a decrease of approxima

29 supplying an adequate amino acid intake or a sulfur amino acid (SAA) (methionine and cysteine) free m

30 creases in seven transcripts occurred in the sulfur amino acid (SAA) biosynthetic pathway and the iro

31 DR-mediated stress resistance, we found that sulfur amino acid (SAA) restriction increased expression

32 flux and oxidation rates were determined and sulfur amino acid (SAA, methionine plus cysteine) balanc

33 ning normal or low amounts of acid-producing sulfur amino acids (SAA) and examined how this adaption

34 w these rates compare with those when either sulfur amino acids (SAAs: methionine and cyst(e)ine) or

35 Methionine is an essential sulfur amino acid that is engaged in key cellular functi

36 Homocysteine is a sulfur amino acid whose metabolism stands at the interse

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