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1 ucleotide 3'-5' phosphodiesters proceeds via the attack of a 3'-OH on a high-energy 5' phosphoanhydri
2 s of catalysis proposed for these enzymes is the attack of a methylene radical, derived from a methyl
3 econd activity was 2',5'-branch formation by the attack of a substrate 2'-hydroxyl group on the 5'-te
4 step in this protein modification pathway is the attack of a substrate lysine residue on Ub bound thr
5 lved to protect multicellular organisms from the attack of a variety of pathogens.
6 e and the fellow eye of the same patient and the attack of ACG after CRVO.
7 It is argued that the steric interactions in the attack of alcohols (sp(3)-hybridized O) and of typic
8                                              The attack of aliphatic amines may be directed to the ar
9           To that end, we have characterized the attack of AMPs on Escherichia coli cytoplasmic membr
10 trategies to evade and in some cases exploit the attacks of an activated immune system.
11 o report the anterior chamber (AC) depth and the attack of angle-closure glaucoma (ACG) in eyes with
12  multiple sclerosis (MS) and is initiated by the attack of autoreactive T cells against self-myelin a
13 rt the role of the amide moiety in directing the attack of carboxylic acid via hydrogen bonding.
14  that the mechanism of inactivation involves the attack of Cys114 at C-2' of MCPF-CoA, resulting in r
15 neity that arises in the wild-type MurA from the attack of Cys115 on C-2 of FPEP in competition with
16 ns is how hosts integrate signals induced by the attack of different parasites.
17 e cross-linking reaction was consistent with the attack of DNA by transient Cr(III)-ascorbate complex
18        A unique reaction mechanism involving the attack of enzyme-bound acetate or the direct attack
19 e system evolved to protect the host against the attack of foreign, potentially pathogenic, microorga
20  and 24 h after infection), HdT responded to the attack of H. vastatrix with a larger number of up-re
21 raft-versus-host disease (GVHD) results from the attack of host tissues by donor allogeneic T cells a
22 s, we have documented that larvae can escape the attack of Leptopilina boulardi parasitoid wasps by r
23 roduction by placental trophoblasts prevents the attack of maternal T-cells activated in response to
24 e have used computational methods to examine the attack of methyl thiolate on each of the three elect
25 tudies indicate that the first step involves the attack of Ni(I) on the sulfur of Me-SCoM, forming a
26                               Examination of the attack of OH radicals produced in the Fenton way on
27 firstly by epoxide-ring opening initiated by the attack of one phenolic hydroxyl group and, then, by
28   A C203S substitution protects SsrB against the attack of RNS while preserving its DNA-binding capac
29 tep, concerted, S(N)2(P) mechanism involving the attack of solvent water on phosphorus assisted by in
30                                              The attack of Srx or GSH on the Prx-SO(2)PO(3)(2-) inter
31 of (S)-HPP and the epoxide ring is formed by the attack of the 2-OH group at C-1 coupled with the tra
32     It ligates two substrate RNAs, promoting the attack of the 3'-hydroxyl of one substrate upon the
33  terminal 2'-OH at the nick is important for the attack of the 3'-OH on the 5'-adenylated strand to f
34                     That step is followed by the attack of the C-terminal asparagine side chain on it
35                                     To evade the attack of the cell host's immune system, KSHV switch
36 bstract the proton from 2-OH and facilitates the attack of the deprotonated 2-oxygen on the phosphoru
37 high or even complete facial selectivity for the attack of the glycosyl acceptor.
38  fluorescence microscopy to directly observe the attack of the human antimicrobial peptide LL-37 on s
39           The mechanism by which KSHV evades the attack of the immune system and establishes latency
40  a transesterification reaction that entails the attack of the neighbouring 2'-oxygen with departure
41 and rearrangement must occur after or during the attack of the OX substrate.
42  assigned to an ester-like species formed by the attack of the product's 4-O- group on the carbonyl o
43 del-Crafts reactions occurred as a result of the attack of the proton at the carbon atom to which the
44 ilizes the enamine intermediate required for the attack of the second aldose substrate, changing the
45 e endogenous His ligand dissociates prior to the attack of the second NO molecule on the proximal hem
46      The ruffled porphyrinate ring would aid the attack of the terminal oxygen of the hydroperoxide i
47 His-265 in vaccinia topoisomerase) catalyzes the attack of the tyrosine nucleophile (Tyr-274) at the
48 lass I antigens, we do not know what signals the attack of these targets.
49                        Adaptations to divert the attacks of visually guided predators have evolved re
50 lucosamine 1-phosphate and UMP by catalyzing the attack of water at the alpha-P atom.
51                       The stereochemistry of the attack of water on each carbocation is independent o
52 rometry, demonstrating that CcLpxI catalyzes the attack of water on the beta-P atom of UDP-2,3-diacyl
53 onium species, (3) hydrolysis to 2'-AADPR by the attack of water on the carbonyl carbon, and (4) an S

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