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1 ntly in patients undergoing vascular (7.7%), thoracic (6.5%), and transplant surgery (6.3%).
2  were increased in human sporadic descending thoracic AAD (TAAD) samples.
3                           Anterior-posterior thoracic amplitude, SUVmax, and SUVpeak (SUVmean in a 1-
4 8.9+/-10.3 years), who underwent noncontrast thoracic and abdominal multidetector computed tomography
5 ility and safety of the recovery of multiple thoracic and abdominal organs with multiple vascular com
6 y of Transplantation Liver and Intestine and Thoracic and Critical Care Communities of Practice, prov
7 ly localizes vertebral body fractures in the thoracic and lumbar spine on CT images with a high sensi
8                               A total of 210 thoracic and lumbar vertebrae showed compression fractur
9 ession fractures and measure bone density of thoracic and lumbar vertebral bodies on computed tomogra
10         Overall, the patients underwent 2103 thoracic and nonthoracic MRI examinations that were deem
11 or muscle and a wide spectrum of ipsilateral thoracic and upper extremity deformities.
12 pinal cord and migrate caudally to the lower thoracic and upper lumbar regions.
13 mation and progression within the ascending, thoracic, and abdominal aorta.
14 ons (25 [17%] vs 21 [14%]), and respiratory, thoracic, and mediastinal disorders (13 [9%] vs 17 [12%]
15  LPA2, LPA4, and LPA6 In endothelium-denuded thoracic aorta (TA) and abdominal aorta (AA) segments, 1
16 alloon Occlusion of the Aorta (REBOA) at the thoracic aorta (Zone 1) can limit subdiaphragmatic blood
17                           Enlarged ascending thoracic aorta and descending thoracic aorta were not si
18 pG hypermethylation within the dilated human thoracic aorta and in SMCs cultured from these tissues,
19        Progressive disease course (P=0.017), thoracic aorta involvement (P=0.009), and retinopathy (P
20                                The ascending thoracic aorta is designed to withstand biomechanical fo
21 repair of aortic aneurysms of the descending thoracic aorta thoracic endovascular aortic repair (TEVA
22 rged ascending thoracic aorta and descending thoracic aorta were not significantly associated with CV
23 symphysis pubis to aortic zone I (descending thoracic aorta) and zone III (infrarenal aorta).
24 , including aneurysms and dissections of the thoracic aorta, are a major cause of morbidity and morta
25 cumulation of aggrecan and a dilation of the thoracic aorta, confirming that aggrecanase activity reg
26 natomically defined locations: the ascending thoracic aorta, descending thoracic aorta, the infrarena
27 siform enlargement of the root and ascending thoracic aorta, leading to ascending aortic dissections.
28 ns: the ascending thoracic aorta, descending thoracic aorta, the infrarenal abdominal aorta, and lowe
29 n the mesenteric circulation compared to the thoracic aorta.
30 ng electron microscopic images of normal rat thoracic aorta.
31 less newly synthesis of IkappaBalpha mRNA in thoracic aortas (gestational day 20, postnatal week 7 an
32 We examined by immunofluorescence microscopy thoracic aortas from 16 simian immunodeficiency virus (S
33 in-angiotensin system (RAS) over-activity in thoracic aortas, resulting in reduced blood pressure in
34 el tone of porcine coronary arteries and rat thoracic aortas.
35 issection (AoD) is a serious complication of thoracic aortic aneurysm (TAA).
36 iscusses published data on genes involved in thoracic aortic aneurysm and attempts to explain diverge
37                  A genetic predisposition to thoracic aortic aneurysm has been established, and gene
38  modern understanding of the pathogenesis of thoracic aortic aneurysm is quite limited.
39 genes known to be associated with BAV and/or thoracic aortic aneurysm was performed.
40 condition, which is commonly associated with thoracic aortic aneurysm.
41            Genetic aortopathy (GA) underlies thoracic aortic aneurysms (TAA) in younger adults.
42                                              Thoracic aortic aneurysms and acute aortic dissections (
43 al registry of GenTAC (Genetically Triggered Thoracic Aortic Aneurysms and Cardiovascular Conditions)
44 lood Institute GenTAC (Genetically Triggered Thoracic Aortic Aneurysms and Cardiovascular Conditions)
45 most common genetic alterations for familial thoracic aortic aneurysms and dissections (TAAD) are mis
46                                              Thoracic aortic aneurysms and dissections (TAAD) represe
47                                    Heritable thoracic aortic aneurysms and dissections (TAAD), includ
48  patients with conditions that predispose to thoracic aortic aneurysms and dissections, including MFS
49  SNPs at the FBN1 locus associated with both thoracic aortic aneurysms and dissections.
50 esponsible for 25% of families with familial thoracic aortic aneurysms and dissections.
51 mutation responsible for a large family with thoracic aortic aneurysms and dissections.
52 ACTA2, are the most common cause of familial thoracic aortic aneurysms that lead to dissection (TAAD)
53  smooth muscle isoform of alpha-actin, cause thoracic aortic aneurysms, acute aortic dissections, and
54 at underwent elective surgery for descending thoracic aortic aneurysms.
55 ective Aortic Arch Perfusion (SAAP) combines thoracic aortic balloon hemorrhage control with intra-ao
56 echocardiography and gated contrast-enhanced thoracic aortic computed tomography or magnetic resonanc
57            The altered genes predisposing to thoracic aortic disease either disrupt smooth muscle cel
58                             Risk factors for thoracic aortic disease include increased hemodynamic fo
59 he Diagnosis and Management of Patients With Thoracic Aortic Disease" and the "2014 AHA/ACC Guideline
60 lly defective aorta is the primary driver of thoracic aortic disease, and that TGF-beta overactivity
61 significantly associated with development of thoracic aortic disease.
62 t rare genetic variants in LOX predispose to thoracic aortic disease.
63 nal molecular pathway that leads to familial thoracic aortic disease.
64 fied potential therapeutic agents to prevent thoracic aortic disease.
65                                              Thoracic aortic diseases, including aneurysms and dissec
66 nters worldwide that specialize in heritable thoracic aortic diseases, was used to gather data on 441
67 ing specifically with non-familial, sporadic thoracic aortic dissection (STAD) and compared them to t
68 conditions in abdominal aortic aneurysms and thoracic aortic dissections to thrombosis in stenotic ar
69 ion associated with spinal cord injury after thoracic aortic ischemia-reperfusion (TAR) in mice.
70                               RATIONALE: The thoracic aortic wall can degenerate over time with catas
71 ture SMC senescence, with relevance to human thoracic aortopathy.
72 loss of specialization in both abdominal and thoracic appendages.
73                           Bilateral internal thoracic arteries (BITA) have demonstrated superior pate
74 to compare survival after bilateral internal thoracic artery (BITA) over single left internal thoraci
75 ; stenosis was quantified for 7,903 internal thoracic artery (ITA) grafts and 20,066 saphenous vein g
76 g (MAG) vs the standard use of left internal thoracic artery (LITA) supplemented by saphenous vein gr
77 score-matched recipients of a right internal thoracic artery (n=1576) or a radial artery (n=4290).
78 acic artery (BITA) over single left internal thoracic artery (SITA).
79 with similar mortality rates (right internal thoracic artery 10.3% versus radial artery 10.7% at 7 ye
80 terial conduits other than the left internal thoracic artery are seldom used in the United States.
81                             A right internal thoracic artery graft offered no benefit over that of a
82 on with radial artery grafts, right internal thoracic artery grafts were associated with similar mort
83 ed a second arterial conduit (right internal thoracic artery or radial artery, n=5866) or a venous co
84 rtery bypass grafting with the left internal thoracic artery, and who received a second arterial cond
85 ence between those receiving single internal-thoracic-artery grafts and those receiving bilateral int
86 rafts and those receiving bilateral internal-thoracic-artery grafts with regard to mortality or the r
87 s compared with the use of a single internal-thoracic-artery plus vein grafts.
88 /Japanese Respiratory Society/Latin American Thoracic Association guidelines (81.3%), usual interstit
89 metabolized less quercetin and produced less thoracic ATP, the energy source for flight muscles.
90 F) is a common side effect for patients with thoracic cancer receiving radiation therapy.
91 al information is available at: www.asco.org/thoracic-cancer-guidelines and www.asco.org/guidelineswi
92  thoracic duct of these mice could enter the thoracic cavity by retrograde flow into enlarged paraver
93 ume between 60 and 250 mL suctioned from the thoracic cavity in a period of 5 minutes) were randomize
94 se models, the path chyle takes to reach the thoracic cavity is unclear.
95 ravasates from these plexuses and enters the thoracic cavity through exfoliated regions of the pleura
96 io 1.0 or less and liver herniation into the thoracic cavity.
97 acting pulmonary embolism (PE) findings from thoracic computed tomography (CT) reports from two insti
98 4 cm, who also had a gated contrast-enhanced thoracic computed tomography or magnetic resonance angio
99 metry, chest radiograph, and high-resolution thoracic computerized tomography scans.
100                       Patients with isolated thoracic cord myelitis at the time of pain onset were si
101 ammatory disease with a predilection for the thoracic cord.
102            Persistent, caudally located (ie, thoracic) cord lesions in AQP4-Ab-positive patients asso
103 (1) global calcification (defined by aortic, thoracic, coronary, and valvular calcification); (2) adi
104                                       In all thoracic CT examinations of the chest at least parts of
105 nty eight patients with pleural effusions on thoracic CT who underwent thoracentesisis within two wee
106 is unknown how patients with smoking-related thoracic diseases or their surrogates display future-ori
107 d injections of dextran-amine were made into thoracic DRGs (T8-T12).
108 : 2 patients (8%) with traumatic leak from a thoracic duct (TD) branch, 14 patients (56%) with pulmon
109                Intranodal lymphangiogram and thoracic duct catheterization was successful in all pati
110 sults from retrograde flow of chyle from the thoracic duct into lymphatic tributaries with defective
111   Although retrograde flow of chyle from the thoracic duct is considered a potential mechanism underl
112 phatic network in the zebrafish, whereas the thoracic duct is initially dispensable for lymphatic fun
113 luorescent tracer revealed that lymph in the thoracic duct of these mice could enter the thoracic cav
114  either lymphatic embolization procedures or thoracic duct stenting with covered stents to exclude re
115 onstrated retrograde lymphatic flow from the thoracic duct toward lung parenchyma.
116 f trunk lymphatic vessels and did not form a thoracic duct.
117 as well as in the genetic diseases short-rib thoracic dysplasia, Mohr-syndrome and amyotrophic latera
118 d in mainland China between 2008 and 2015 on thoracic endovascular aortic repair (TEVAR) for type B a
119 c aneurysms of the descending thoracic aorta thoracic endovascular aortic repair (TEVAR) is relativel
120 eating type A (ascending aorta) AAS, whereas thoracic endovascular aortic repair may be optimal for t
121 tive of this randomized trial was to compare thoracic epidural analgesia (TEA) to intravenous patient
122                                              Thoracic epidural anesthesia resulted in a significant d
123 ry, was tested before and after induction of thoracic epidural anesthesia using combined pressure-con
124 sed afterload were the same before and after thoracic epidural anesthesia.
125 ignals from the brain to the spinal cord (or thoracic ganglia in insects).
126 neurons projecting to the labrum, brain, and thoracic ganglion have received less attention.
127 s who underwent inpatient general, vascular, thoracic, genitourinary, neurosurgical, orthopedic, or s
128 ed 24 adult-specific NB lineages within each thoracic hemineuromere of the larval ventral nervous sys
129  was assessed by means of serial cardiac and thoracic imaging.
130 that is possible that neuroglial loss from a thoracic inflammatory process results in anterograde and
131  examination, 39% (574 of 1461) had signs of thoracic injuries (men, 74.0% [425 of 574]; mean age, 46
132  in lung transplant recipients from the same thoracic intensive care unit (ICU).
133     We report the discovery of male-specific thoracic interneurons-the TN1A neurons-that are required
134  patients with mesothelioma after large-bore thoracic interventions is not justified.
135 leotidase CD73 in fibrosis development after thoracic irradiation.
136 s a potentially lethal late adverse event of thoracic irradiation.
137 ting side effect that occurs in up to 30% of thoracic irradiations in breast and lung cancer patients
138 n a mouse model of inducible pneumonia, high thoracic lesions that interrupt sympathetic innervation
139                                 Cervical and thoracic lesions that persisted from pain onset to 'out
140 ervation to major immune organs, but not low thoracic lesions, significantly increased bacterial load
141 ogenic effect was more pronounced after high-thoracic level (Th1) SCI disconnecting adrenal gland inn
142 adrenal gland innervation, compared with low-thoracic level (Th9) SCI.
143 bjects at the aqueduct, in 11/12 subjects at thoracic level 2, and in 4/12 subjects at thoracic level
144 ury (SCI) at high spinal levels (e.g., above thoracic level 5) causes systemic immune suppression; ho
145 at thoracic level 2, and in 4/12 subjects at thoracic level 5.
146 a transition in facet morphology at the 11th thoracic level.
147 all had zygapophyseal facets that shift from thoracic-like to lumbar-like at the penultimate rib-bear
148 Over the course of infection, granulomas and thoracic lymph nodes experienced dynamic changes in affi
149 nectomy, and group 3-a minimal abdominal and thoracic lymphadenectomy.
150    Purpose To summarize existing evidence of thoracic magnetic resonance (MR) imaging in determining
151                The use of bilateral internal thoracic (mammary) arteries for coronary-artery bypass g
152  occasional asphyxia over 10 seconds with no thoracic motion after a desaturation episode.
153 ime a thoracic surgeon ordered a nonvascular thoracic MR imaging study via radiology order entry, he
154 propriate cases, assessment with nonvascular thoracic MR imaging substantially affects the clinical d
155          A thorough visual inspection of the thoracic MR-AC map and Dixon images from which it is der
156 uated the quality of current vendor-provided thoracic MR-AC maps and further investigated the reprodu
157       Artifacts occur frequently in standard thoracic MR-AC maps, affecting the reproducibility of PE
158               During larval life most of the thoracic neuroblasts (NBs) in Drosophila undergo a secon
159  the larval hemilineage tracts for all three thoracic neuromeres through metamorphosis into the adult
160 g to 3 groups: group 1-exclusion of proximal thoracic nodes, group 2-a minimal abdominal lymphadenect
161 or the formation of the insect wing from the thoracic notum as well as the already known pleural elem
162 s), supporting their overall origin from the thoracic notum as well as the expected medial, pleural s
163 ne of wings representing an extension of the thoracic notum, the other stating that they are appendic
164 , from inside and outside of the Assembly on Thoracic Oncology.
165 ns, continues to affect a high proportion of thoracic organ transplant recipients.
166  by increased age, white race, male sex, and thoracic organ transplantation.
167 es (3%), paroxysmal atrial tachycardia (3%), thoracic pain (3%), upper gastrointestinal hemorrhage (3
168  increasingly complex abdominal injuries and thoracic, peripheral vascular, and orthopedic injuries.
169 ble non-small cell lung cancer underwent 2-5 thoracic PET/MRI scan-rescan examinations within 22 d.
170 gh-mortality inpatient general, vascular, or thoracic procedures.
171 bstructive pulmonary disease (COPD) based on thoracic quantitative computed tomographic (QCT) paramet
172 chemotherapy regimens used concurrently with thoracic radiation for patients with unresectable IIIA a
173 ts with non-small-cell lung cancer receiving thoracic radiation.
174                                            A thoracic radiologist annotated 89 semantic image feature
175                            For each tumor, a thoracic radiologist recorded 87 semantic image features
176                            Three experienced thoracic radiologists identified true locations of nodul
177          The reproducibility and accuracy of thoracic radiologists in classifying whether or not a no
178 een no previous study on the efficacy of the thoracic radiotherapy (TRT) in oligometastatic or polyme
179 ) of 24 patients who had previously received thoracic radiotherapy had any recorded pulmonary toxicit
180           Three (13%) patients with previous thoracic radiotherapy had treatment-related pulmonary to
181 therapy and 24 (25%) of 97 patients received thoracic radiotherapy.
182 sus 29 (40%) of 73 patients with no previous thoracic radiotherapy.
183                                They increase thoracic rigidity [8], which decreases speed of locomoti
184 vents were relatively common after high-dose thoracic RT and were independently associated with both
185 ithin the largest cerebral artery after high-thoracic SCI, leading to increased stiffness and possibl
186 y hominins, to date, no complete cervical or thoracic series has been recovered.
187 tored by bending specialised elements of the thoracic skeleton that are composites of the rubbery pro
188  activation of afferent nerves in the dorsal thoracic skin of the mouse.
189                                 The American Thoracic Society (ATS) created a multidisciplinary ad ho
190          The goals of this Official American Thoracic Society (ATS) Research Statement are to discuss
191  a collaborative effort between the American Thoracic Society and the American College of Chest Physi
192  a collaborative effort between the American Thoracic Society and the American College of Chest Physi
193       The Pediatric Assembly of the American Thoracic Society assembled an interdisciplinary workgrou
194                                 The American Thoracic Society committee on Proficiency Standards for
195                                 The American Thoracic Society convened a multistakeholder committee w
196 measured FE NO50 in accordance with American Thoracic Society guidelines, 2005 (off-line excluded); r
197  18 years or older in Malawi, using American Thoracic Society standard spirometry, internationally va
198       A task force supported by the American Thoracic Society, Centers for Disease Control and Preven
199  the NHLBI, in partnership with the American Thoracic Society, convened a workshop of investigators i
200 American Academy of Pediatrics, the Canadian Thoracic Society, the International Union Against Tuberc
201                                  An American Thoracic Society-sanctioned guideline development commit
202 d according to the standards of the American Thoracic Society.
203 ording to the mapping system of the American Thoracic Society.
204                                 The American Thoracic Society/American College of Chest Physicians re
205 ions between SA (as defined by 2014 American Thoracic Society/European Respiratory Society guidelines
206 ally classified as IPF according to American Thoracic Society/European Respiratory Society/Japanese R
207       NTM disease was defined by 1) American Thoracic Society/Infectious Disease Society of America c
208                      We applied the American Thoracic Society/Infectious Disease Society of America c
209 emarkable for mild cerebellar and noticeable thoracic spinal cord atrophy.
210                                          The thoracic spinal cord is intrinsically susceptible to isc
211 MNs) and Renshaw cells (RCs) is disrupted by thoracic spinal cord transection at postnatal day 5 (P5T
212 OEC transplants acutely after a complete low-thoracic spinal cord transection in adult rats.
213                 At the lesion epicenter (mid-thoracic spinal cord), the microenvironment created by C
214 l branches but sympathetically denervated by thoracic spinal pithing, cardiac chronotropic vagal tone
215 umbar and pelvic) studies, especially in the thoracic spine and thoracic wall, pelvic and shoulder gi
216 o our facility for evaluation of findings on thoracic spine radiographs obtained at a peripheral hosp
217 pondylodiscitis included: involvement of the thoracic spine, involvement of 2 or more adjacent verteb
218  (fl/fl) mice had a severely deformed curved thoracic spine, with an associated loss of trabecular bo
219                                        Thus, thoracic staging with PET/MRI bears a risk of missing sm
220 homeotic transformation of the fly head into thoracic structures.
221 uly 16, 2013, and July 13, 2015, each time a thoracic surgeon ordered a nonvascular thoracic MR imagi
222 e stroke was defined according to Society of Thoracic Surgeons (STS) criteria as any confirmed neurol
223   Clinical data were obtained and Society of Thoracic Surgeons (STS) score was calculated.
224 raphy data were recorded, and the Society of Thoracic Surgeons (STS) score was calculated.
225  (7) years; 582 (59.1%) men; mean Society of Thoracic Surgeons (STS) score, 11.4% (4.0%); and mean LV
226                               The Society of Thoracic Surgeons (STS)/American College of Cardiology T
227 US Census, US News Top Hospitals, Society of Thoracic Surgeons composite rating for coronary artery b
228 al heart disease operation in the Society of Thoracic Surgeons Congenital Heart Surgery Database betw
229 essive intervention with skilled cardiac and thoracic surgeons may improve chances of stroke-free sur
230 theter Valve Therapy Registry and Society of Thoracic Surgeons National Database linked to Medicare a
231 (mean age, 79+/-9 years; 44% men; Society of Thoracic Surgeons predicted risk mortality score, 6.7+/-
232       We hypothesized that if the Society of Thoracic Surgeons Predicted Risk of Mortality (STS PROM)
233 .1% (IQR 3.6-12.8) and the median Society of Thoracic Surgeons predicted risk of mortality for mitral
234 ched intermediate- and high-risk (Society of Thoracic Surgeons Predicted Risk of Mortality score >/=3
235 n age 82 years, 48% women, median Society of Thoracic Surgeons Predicted Risk of Mortality score 5.6%
236  considered an alternative to the Society of Thoracic Surgeons Predicted Risk of Mortality score for
237                 Compared with the Society of Thoracic Surgeons Predicted Risk of Mortality score, the
238  its predictive accuracy with the Society of Thoracic Surgeons Predicted Risk of Mortality score.
239 years, 64.9% were women, the mean Society of Thoracic Surgeons Predicted Risk of Mortality was 5.5 +/
240                               The Society of Thoracic Surgeons Predicted Risk of Mortality was 7.3 +/
241                               The Society of Thoracic Surgeons predicted risk of mortality was 9.6 +/
242 at intermediate risk for surgery (Society of Thoracic Surgeons Predicted Risk of Mortality, 4.5+/-1.6
243 , nonagenarians had a higher mean Society of Thoracic Surgeons Predicted Risk of Operative Mortality
244     Addition of gait speed to the Society of Thoracic Surgeons predicted risk resulted in a C statist
245  filtration rate, and higher mean Society of Thoracic Surgeons score (9.0% vs. 8.0%; all p < 0.001).
246         On Cox analysis, a higher Society of Thoracic Surgeons score (hazard ratio 1.55), higher base
247 nsformed BNP to a clinical model (Society of Thoracic Surgeons score and baseline right ventricular s
248                                   Society of Thoracic Surgeons score and cardiovascular death were re
249 orsening eGFR had a higher median Society of Thoracic Surgeons score and left ventricle mass.
250                                   Society of Thoracic Surgeons score and right ventricular systolic p
251 A total of 248 patients with mean Society of Thoracic Surgeons score of 8.9 +/- 6.8% underwent TMVR.
252 f % age-sex-predicted METs to the Society of Thoracic Surgeons score resulted in significant reclassi
253 nclusion of GDF-15 and CRP to the Society of Thoracic Surgeons score significantly improved C index (
254 was 78.9 +/- 10.2 years, and mean Society of Thoracic Surgeons score was 9.1 +/- 4.7%.
255                                   Society of Thoracic Surgeons score was calculated.
256                               The Society of Thoracic Surgeons score was calculated.
257 lity included alternative access, Society of Thoracic Surgeons score, and disabling stroke.
258 statistic for the clinical model (Society of Thoracic Surgeons score, degree of aortic regurgitation,
259 /=10 cm(2)/m to a clinical model (Society of Thoracic Surgeons score, inherited aortopathies, hyperte
260                               The Society of Thoracic Surgeons score, left ventricular ejection fract
261                              Mean Society of Thoracic Surgeons score, left ventricular ejection fract
262                 At baseline, mean Society of Thoracic Surgeons score, LV ejection fraction, mean aort
263 on of mortality when added to the Society of Thoracic Surgeons score.
264 ar Angiography and Interventions, Society of Thoracic Surgeons, and American Association for Thoracic
265 tion Physician Masterfile and the Society of Thoracic Surgeons-Congenital Heart Surgery Database to e
266              After adjustment for Society of Thoracic Surgeons-recognized risk factors, women had sig
267  decision making and diagnostic certainty of thoracic surgeons.
268 tient data were obtained from the Society of Thoracic Surgeons/American College of Cardiology Transca
269            National data from the Society of Thoracic Surgeons/American College of Cardiology Transca
270 ne 2014) and were included in the Society of Thoracic Surgeons/American College of Cardiology Transca
271 onal Cardiovascular Data Registry Society of Thoracic Surgeons/American College of Cardiology Transca
272 tenosis who underwent TAVR in the Society of Thoracic Surgeons/American College of Cardiology Transca
273 ODS AND We analyzed data from the Society of Thoracic Surgeons/American College of Cardiology Transca
274             METHODS AND Using the Society of Thoracic Surgeons/Transcatheter Valve Therapy Registry l
275 nary artery bypass surgery at 663 Society of Thoracic Surgery Database participating sites (January 1
276  from the Netherlands Association for Cardio-Thoracic Surgery database.
277  years (55.8% men), with a median Society of Thoracic Surgery predicted risk of mortality of 6.1% (in
278                         The mean Society for Thoracic Surgery score was 6.4 +/- 5.5%; 86% of patients
279 ed with the early development of general and thoracic surgery to which he contributed.
280 racic Surgeons, and American Association for Thoracic Surgery, along with key specialty and subspecia
281 of the anticipated need for postchemotherapy thoracic surgery.
282                     Data from the Society of Thoracic Surgery/American College of Cardiology Transcat
283 /-8.2 years; 60% women; mean STS [Society of Thoracic Surgery] score 8.1+/-5.5%).
284 s study was to examine the influence of high-thoracic (T3 spinal segment) SCI on cerebrovascular stru
285 /TSP patients followed longitudinally showed thoracic thinning followed by cervical thinning.
286 xons, leading to the temporal progression of thoracic to cervical atrophy described here.
287                 In cases of recurrent CMV in thoracic transplant patients after a disease- and drug-f
288 pedics, otolaryngologic, plastic, podiatric, thoracic, transplant, urologic, and peripheral vascular.
289 y, combining the fracture with an additional thoracic trauma.
290 ions and 19.9% (114 of 574) in patients with thoracic trauma.
291 nockout of Abd-B resulting in an animal with thoracic type legs along what would have been an abdomen
292 een clear is whether Australopithecus had 12 thoracic vertebrae as in most humans, or 13 as in most A
293 inal cord, which terminates midway along the thoracic vertebrae before giving rise to a long and exte
294 vical vertebrae and provides evidence for 12 thoracic vertebrae with a transition in facet morphology
295        The exceptions were the atlas and mid-thoracic vertebrae, which remained at the 5- to 6-year s
296 the only known complete hominin cervical and thoracic vertebral column before 60,000 years ago.
297 sites of dorsal amniotic closure and ventral thoracic wall formation.
298 tudies, especially in the thoracic spine and thoracic wall, pelvic and shoulder girdles, and peripher
299 the body of the embryo and distribute to the thoracic wall, pharyngeal arches and heart.
300  carina and the aortic valve, divided by the thoracic width, was found to be the best performing rati

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