ライフサイエンスコーパス: to escape from

1 Endobronchial valves that allow air to escape from a pulmonary lobe but not enter it can ind

2 system, a population of viruses that evolved to escape from a single antisense RNA would require a co

3 es in envelope structure, enabling Y. pestis to escape from adaptive immune responses and plague immu

4 hat fill the HAP site are not a path for HBV to escape from AEfs.

5 ells, and correlates well with the inability to escape from an extended late S-phase-G2 arrest.

6 t drug self-administration enables an addict to escape from and avoid the severe withdrawal symptoms

7 that, CCl4 (but also (CH3)3CBr) was proposed to escape from, and a molecule of solvent to enter, the

8 f human immunodeficiency virus (HIV) leading to escape from anti-HIV drugs is the greatest challenge

9 ar the receptor binding site were sufficient to escape from antibodies specific for A(H1N1)pdm09 viru

10 art by mutations in the viral capsid leading to escape from antibody neutralization.

11 nfers a survival advantage by allowing cells to escape from apoptosis, supporting a new role for aber

12 demonstrated the propensity of an RNA virus to escape from attenuation but also showed that, through

13 expansion of the V1V2 domain is one pathway to escape from autologous neutralization in subtype C En

14 grow at bird body temperature, and are able to escape from bird macrophages by vomocytosis.

15 ly, probably because they have no safe place to escape from bullying.

16 reased apoptosis but, remarkably, was unable to escape from cells as efficiently as the single mutant

17 ht that spare CCR5, which might permit HIV-1 to escape from chemokines, should be targeted for effici

18 We provide evidence that epitopes known to escape from chronic CD8 T cell responses in animals t

19 ve now obtained evidence that epitopes known to escape from chronic CD8 T cell responses in animals t

20 Many invasive species are able to escape from coevolved enemies and thus enjoy a compet

21 cohesion because it permits chromatin fibers to escape from cohesin rings.

22 Pathogens, in turn, have evolved strategies to escape from commensal-mediated resistance to coloniza

23 The ability to escape from confinement and reintegrate after mitosis

24 active-site cysteine provides PDI with a way to "escape" from covalent intermediates that do not rear

25 tical for normal and transformed human cells to escape from crisis and is implicated in oncogenesis.

26 itro system to evaluate the ability of HIV-1 to escape from CTL clones, finding that virus replicatin

27 HIV's ability to mutate to escape from CTL pressure is increasingly recognized;

28 studies have highlighted the ability of HIV to escape from cytotoxic T lymphocyte (CTL) responses th

29 sents a cardinal strategy deployed by tumors to escape from detection and elimination by the immune s

30 In one, subjects are given the option to escape from difficult trials.

31 In this environment, movement is critical to escape from disturbances and to find resources scatte

32 nts have evolved shoot elongation mechanisms to escape from diverse environmental stresses such as fl

33 ormant lymphoma cells and the host that lead to escape from dormancy.

34 ce of Chk2 allow Brca1(delta11/delta11) mice to escape from embryonic lethality.

35 2 murine macrophage-like cells, an inability to escape from endocytic vacuoles and a complete absence

36 trated the impaired capacity of inlB mutants to escape from endocytic vacuoles and to enter the cytop

37 mAM(PU.1+) and MH-S cells, adenovirus failed to escape from endosomes, colocalized exclusively with e

38 ing skin infections by allowing the bacteria to escape from endosomes, replicate intracellularly, and

39 ficantly, the transported proteins were able to escape from endosomes.

40 Thus, although much attention has been paid to escape from enemies as a factor in the establishment

41 epidoptera in temperate forests with respect to escape from enemies.

42 pathway are abrogated and allow individuals to escape from group behavior.

43 sforming events that metastases must undergo to escape from growth restriction, cannot be extracted f

44 ucleotide analogs and those with the ability to escape from HBV-neutralizing antibody have the potent

45 cytogenes Deltahly mutants, which are unable to escape from host cell vacuoles, did not express actA/

46 HLA-I-restricted peptide antigens allows it to escape from host cytotoxic T lymphocytes (CTLs).

47 osis factor (TNF), which causes cancer cells to escape from host immune defenses.

48 HIV-1 populations, thereby allowing variants to escape from host immunity or antiviral therapies.

49 alization epitopes likely allowed GII.4-2012 to escape from human herd immunity and emerge as the new

50 n contribute to the ability of a skin cancer to escape from immune attack by cytotoxic T lymphocytes,

51 anding the mechanisms that enable the cancer to escape from immune attack.

52 The ability of an AIDS virus to escape from immune containment by selective mutation

53 least in part, to the capacity of this virus to escape from immune recognition through mutation.

54 cells with selective HLA class I allele loss to escape from immune recognition.

55 of the HCV-specific antibodies, enabling HCV to escape from immune surveillance.

56 rds persisting virus a window of opportunity to escape from immune surveillance.

57 e 12 (Ad12)-transformed cells, enabling them to escape from immunosurveillant cytotoxic T lymphocytes

58 poration into the cell genome, enabling them to escape from induction of senescence.

59 h has also shown that migration allows hosts to escape from infected habitats, reduces disease levels

60 unique features of ADAMDEC1 may have evolved to escape from inhibition by endogenous metalloprotease

61 EF-5 did not appear to enable RNA polymerase to escape from intrinsic pause sites.

62 into one aphid species led to it being able to escape from its natural enemy and increase in density

63 Chemopreventive agents allow Nrf2 to escape from Keap1-mediated repression, although the m

64 This suggests that DNA is able to escape from late endosomes without traversing lysosom

65 After perceiving light, roots bend to escape from light (root light avoidance) and reduce t

66 te cellular uptake, a histidine-rich peptide to escape from lysosomes, and an Alexa Fluor 488 tag for

67 tch, enabling wild-type C. albicans and KWN8 to escape from macrophages within 6 h, whereas KWN6 was

68 that cholesterol can activate WT mouse eggs to escape from MII arrest.

69 y permitting a partially degraded fatty acid to escape from mitochondria.

70 ting Ipl1p allows cells overexpressing Mps1p to escape from mitosis and segregate their chromosomes n

71 sphorylatable Cdc20 mutant allows HeLa cells to escape from mitosis in the presence of spindle damage

72 show that it is more difficult for the flies to escape from Mono Lake water than from fresh water, du

73 al genomes to acquire substantial length and to escape from Muller's Ratchet.

74 that allows autoantigen-activated CLL cells to escape from negative selection and indicate that this

75 pidly, as shown by viral mutations that lead to escape from neutralizing antibodies.

76 ew findings indicate that Leishmania is able to escape from neutrophils and 'silently' enter macropha

77 t inactivation of TGF-beta signaling leading to escape from normal growth control occurs at an early

78 teremia, where increased CPS may be required to escape from opsonic clearance.

79 Policy diversification, leading to escape from panacea traps, can come from monitoring i

80 murine macrophage cell lines and was unable to escape from phagosome vacuoles.

81 enes plays a role in the bacterium's ability to escape from phagosomes and spread from cell to cell.

82 er of possible substitutions that could lead to escape from population immunity.

83 d invasive, suggestive of heightened ability to escape from primary tumors due to matrix-degrading ac

84 in antigenic variation that allows the virus to escape from protective herd immunity, resulting in ne

85 Control mice rapidly learned to escape from shallow water in a paddling pool, which c

86 of the ability of renin-angiotensin activity to escape from suppression during long-term treatment.

87 ates at the endosomal pH of 5.5-6.0 in order to escape from target-mediated degradation.

88 MC simulation steps required for the ligand to escape from the 'funnel of attraction' of the binding

89 acid changes in the epitope allow the virus to escape from the antibody.

90 h the previous analysis of MVMi mutants able to escape from the B7 antibody.

91 tions in selected vessels to allow liposomes to escape from the blood stream and to extravasate into

92 editing could allow some autoreactive cells to escape from the bone marrow in lupus-prone mice, thus

93 d capacity of the ST8Sia IV(-/-) progenitors to escape from the bone marrow niche.

94 Furthermore, parasites were able to escape from the brain and establish a systemic infect

95 In gene delivery the genetic material has to escape from the cellular compartments into the cytoso

96 I antigens but also allowed excess molecules to escape from the endoplasmic reticulum.

97 d structure that allow the endocytosed virus to escape from the endosome, pass through the cell cytop

98 cell, presumably reflecting their inability to escape from the endosomes.

99 membrane-bound J proteins B12, B14, and C18 to escape from the ER into the cytosol en route to succe

100 IL-2-dependent T-cell lines allows the cells to escape from the G1 arrest induced by IL-2 withdrawal.

101 his host shutoff is thought to allow viruses to escape from the host antiviral response, which restri

102 es have developed a wide range of strategies to escape from the host cells in which they replicate.

103 an ability to cross the integuments and then to escape from the host immune defenses.

104 uzi, the causative agent of Chagas' disease, to escape from the host immune system and to invade the

105 s to resist to lysozyme degradation and thus to escape from the host innate immune system but little

106 rors made during replication allow the virus to escape from the host's immune system and to develop r

107 HCV employs evasion and sabotage tactics to escape from the host's immune system.

108 e terminates virulence gene expression prior to escape from the host, but it is unknown how this ToxT

109 h ToxT and TcpP undergoing proteolysis prior to escape from the host.

110 mmunodeficiency viruses allows these viruses to escape from the immune pressure mediated by neutraliz

111 ion S(173)A, and the difficulty of the virus to escape from the immune response against the KK10 epit

112 ids, providing an important path for a virus to escape from the immune response.

113 tively high transmissibility and the ability to escape from the immune surveillance of the host.

114 We discuss why alpha males seem to escape from the immunosuppressive costs of glucocorti

115 odies to map the amino acid changes that led to escape from the initial autologous neutralizing antib

116 s latter context, we measure motile bacteria to escape from the interfaces preferentially into the is

117 ze, and hence animals were randomly assigned to escape from the left or the right arm, one of which c

118 and increases the ability of immature cells to escape from the marrow.

119 y decreased indicating that crayfish learned to escape from the maze more rapidly and efficiently.

120 attachment and enables progeny swarmer cells to escape from the monolayer biofilm.

121 ctomized ERalphaKO and WT mice rapidly learn to escape from the Morris water maze.

122 Virulent M. marinum is able to escape from the Mycobacterium-containing vacuole (MCV

123 type II receptor protein, thus allowing them to escape from the negative growth control of TGF-beta1.

124 and combinations to allow neural crest cells to escape from the neuroepithelium.

125 esize that the former may enable tumor cells to escape from the normal growth-constraining influence

126 icient time to dissociate from membranes and to escape from the outer segment.

127 use Francisella organisms have been reported to escape from the phagolysosome into the cytosol, we hy

128 low-dose intracellular pathogen that is able to escape from the phagosome and replicate in the cytoso

129 nes depends on the ability of this bacterium to escape from the phagosome of the host cells via the a

130 ith LVSDeltaiglC, a Ft LVS mutant that fails to escape from the phagosome, displayed greatly increase

131 a monocytogenes that infects cells but fails to escape from the phagosome, we demonstrate the inducti

132 uring tumor formation, allowing cancer cells to escape from the primary tumor.

133 differences in the ability of these enzymes to escape from the promoter and to recognize certain typ

134 The productive ITCs are able to escape from the promoter rapidly to produce full-leng

135 re either released or extended to allow RNAP to escape from the promoter.

136 l capabilities to target cancerous cells and to escape from the recognition and elimination by the re

137 amage their internalization vacuole, leading to escape from the Salmonella-containing vacuole (SCV) a

138 quire a significant period for magnetic flux to escape from the solid inner core and sufficiently wea

139 to result when the replication fork attempts to escape from the stall site.

140 pproximately 140 mV a polymer is more likely to escape from the vestibule against the applied voltage

141 sease form, providing an effective mechanism to escape from the vitamin D-dependent antimicrobial pat

142 o nonindigenous habitats has been attributed to escape from their native natural enemies, allowing re

143 o adapt to low-oxygen environments, and also to escape from them.

144 ck cancers, enable HPV-positive cancer cells to escape from these regulatory principles: E6/E7 is eff

145 est at G2/M. hdf1 cells, lacking Ku70p, fail to escape from this RAD9/RAD17-dependent checkpoint.

146 Among the factors contributing to escape from vacuoles are a phosphatidylcholine phosph

147 Trapped larval worms were sometimes able to escape from worm-stars by undergoing autotomy, separa