ライフサイエンスコーパス: vaginitis

1 sexually transmitted infection (STI) causing vaginitis.

2 n microscopy was associated with Trichomonas vaginitis.

3 very, low birth weight, cervical cancer, and vaginitis.

4 ntibodies are protective in a mouse model of vaginitis.

5 tory role for gamma/delta T cells in Candida vaginitis.

6 one-associated susceptibility to C. albicans vaginitis.

7 ques as an alternative model of experimental vaginitis.

8 nfirmed a nonprotective role for PMNs during vaginitis.

9 sis and immunity associated with C. albicans vaginitis.

10 ant than systemic CMI for protection against vaginitis.

11 en at the time that they were suffering from vaginitis.

12 culation does not mediate protection against vaginitis.

13 nal candidiasis suffering from acute Candida vaginitis.

14 us acidophilus in the prevention of candidal vaginitis.

15 ispose women to recurring attacks of Candida vaginitis.

16 onses previously characterized during murine vaginitis.

17 tions with various prevalences of infectious vaginitis.

18 where it may be asymptomatic or cause severe vaginitis and cervicitis.

19 bacillus species isolated per patient in the vaginitis and control groups, respectively.

20 olates obtained from patients suffering from vaginitis and four isolates from commercial products in

21 croflora in women with signs and symptoms of vaginitis and is comparable in diagnostic accuracy to th

22 rable effects by systemic CMI or PMN against vaginitis and suggest that if local T cells are importan

23 tment of Candida vulvovaginitis, Trichomonas vaginitis, and bacterial vaginosis.

24 lis and M. hominis bacterial counts, Candida vaginitis, and herpes simplex virus (HSV) were positivel

25 Resistance to Candida vaginitis appears additive in CD10 x B6F1 animals and do

26 iae is a ubiquitous, ascomycetous yeast, and vaginitis caused by this organism has been reported only

27 all three with two episodes of S. cerevisiae vaginitis, different strains were isolated during the re

28 of yeast culture plus exclusion of alternate vaginitis etiologies.

29 years of age who presented with symptoms of vaginitis, exposure to T. vaginalis, or multiple sexual

30 Armed with new criteria for defining vaginitis immunopathology, the purpose of this study was

31 extent, the Bcr1 pathways in contributing to vaginitis immunopathology.

32 based assays for determining the etiology of vaginitis in a cohort of 323 symptomatic women.

33 We compared susceptibility to Candida vaginitis in derived murine substrains differing in sens

34 ndidiasis and a murine model of experimental vaginitis indicate that systemic cell-mediated immunity

35 creased susceptibility to experimental yeast vaginitis, indicating a role for alpha(1,2)fucosylated c

36 ALS gene expression patterns in a murine vaginitis model were identical to those from the clinica

37 For Trichomonas vaginitis (n=55), HIV-1 RNA decreased from 3.67 to 3.05

38 tion, genital ulcers, Trichomonas vaginalis, vaginitis or cervicitis, and male circumcision.

39 sted odds ratio, 18.34; 95% CI, 4.95-67.96), vaginitis or vulvovaginitis (adjusted odds ratio, 5.17;

40 nce, preterm labor during current pregnancy, vaginitis or vulvovaginitis, and sepsis.

41 nts (P=.0001; positive association), Candida vaginitis (P=.007), and HSV (P=.03) were significantly a

42 It was demonstrated that (i) in each vaginitis patient, there was only one colonizing strain;

43 Women with symptoms of vaginitis presenting to one of three clinical centers we

44 This animal model of C. glabrata vaginitis provides a means to study the genetics and pat

45 Clinical disease (vaginitis), serum and vaginal washing antibody levels, a

46 VC) and from an animal model of experimental vaginitis suggest that deficiencies in immune function s

47 ows to lower titers and produces less severe vaginitis than wild-type or gC rescued virus, indicating

48 acity to cause immunopathology during murine vaginitis to this avirulent hypofilamentous strain.

49 ontaining the three common organisms causing vaginitis: Trichomonas vaginalis, Candida species, and G

50 dependent reference standard for trichomonal vaginitis (TV), a positive result in either assay was de

51 ansmitted infection (STI) that can result in vaginitis, urethritis, and preterm birth.

52 the combined search terms of diagnosis with vaginitis, vaginal discharge, candidiasis, bacterial vag

53 acterium, Candida, gastrointestinal- system, vaginitis, vaginosis-bacterial, and related terms.

54 he importance of fungal morphogenesis during vaginitis was addressed with a two-pronged approach.

55 of three patients suffering from C. glabrata vaginitis were clonally plated on agar containing CuSO(4

56 lcer disease, vaginal discharge, and Candida vaginitis were significantly associated with HIV-1 seroc

57 In females, trichomoniasis causes vaginitis, while in males, it is frequently asymptomatic

58 onents for the immunopathogenesis of Candida vaginitis, with implications for transition from benign

59 This parasite, a common cause of vaginitis worldwide, is one of the earlier branching euk