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1 ponse was defined as a 15% reduction in left ventricular end-systolic volume.
2 MSC group, because of a preservation of left ventricular end-systolic volume.
3 e brain natriuretic peptide levels, and left ventricular end-systolic volume.
4 The primary end point was left ventricular end-systolic volume.
5 ic volume (0.01 mm/mL; P<0.01), a lower left ventricular end-systolic volume (-0.01 mm/mL; P=0.01), a
7 and was associated with a reduction in left ventricular end-systolic volume (-24.8 +/- 3.0 ml vs. -8
8 unction as indicated by an increase in right ventricular end-systolic volume (54 +/- 10 to 87 +/-6 mL
9 ic volume (-49+/-16% versus -35+/-20%), left ventricular end-systolic volume (-59+/-20 versus -37+/-2
10 ignificant differences were observed in left ventricular end-systolic volumes (-6.4 mL [95% CI, -18.8
11 ctions (42% versus 69%), higher indexed left ventricular end-systolic volumes (96 versus 40 mL), and
12 stolic and end-diastolic dimensions and left ventricular end-systolic volume also decreased after 12
13 with pulmonary regurgitation, elevated right ventricular end systolic volumes and reduced right and l
14 VEF) and the noninvasive calculation of left ventricular end-systolic volume and left ventricular end
15 left ventricular end-diastolic volume, left ventricular end-systolic volume, and left ventricular ej
16 left ventricular end-diastolic volume, left ventricular end-systolic volume, and LVEF were not stati
17 S-LVRR (defined as >/=15% reduction in left ventricular end-systolic volume at 1-year of follow-up)
18 dians (quartiles 1 and 3) for change in left ventricular end-systolic volume at 6 months for the Smar
23 ft ventricular contractility increased (left ventricular end-systolic volume at a pressure of 100 mm
24 actility was assessed by the calculated left ventricular end-systolic volume at an end-systolic left
26 ic volume (beta=0.01/mL; P<0.0001), and left ventricular end-systolic volume (beta=0.01/mL; P<0.001)
27 left ventricular end-diastolic volume, left ventricular end-systolic volume, cardiac index, dP/dt ma
28 mproved in 69% of 426 patients, whereas left ventricular end-systolic volume decreased > or = 15% in
30 during the first month (120% increased left ventricular end-systolic volume [ESV; P<0.01]), but shun
32 ess of CRT was defined as a decrease in left ventricular end-systolic volume >15% at follow-up echoca
35 aced QRS duration, and smaller baseline left ventricular end systolic volume index also were also ass
36 d from 29 +/- 10.4 to 39 +/- 12.4%, and left ventricular end systolic volume index decreased from 109
37 hanges in the clinical composite score, left ventricular end systolic volume index, 6-minute walk tim
38 lume index (-26.2 versus -7.4 mL/m(2)), left ventricular end-systolic volume index (-28.7 versus -9.1
39 ) at follow-up; p = 0.0012), as did the left ventricular end-systolic volume index (48.4 +/- 19.7 ml/
42 found no significant difference in the left ventricular end-systolic volume index (LVESVI) or surviv
46 found no significant difference in the left ventricular end-systolic volume index (LVESVI), survival
47 ienced a 25.3-mL/m(2) mean reduction in left ventricular end-systolic volume index (P<0.0001), wherea
48 0.01) and was associated with increased left ventricular end-systolic volume index (r=0.62, P<0.01),
49 nt MRI predictors of death (P < 0.01): right ventricular end-systolic volume index adjusted for age a
50 In a multivariable regression model, left ventricular end-systolic volume index and left atrial vo
52 p = 0.004), but not when combined with right ventricular end-systolic volume index and strain-rate e'
54 vorably affected by VNS (p < 0.05), but left ventricular end-systolic volume index was not different
55 as also a strong predictor of change in left ventricular end-systolic volume index with monotonic inc
56 ight ventricular dilatation (increased right ventricular end-systolic volume index), high Acute Physi
57 t age, sex, ST or T changes on ECG, and left ventricular end-systolic volume index, LGE maintained a
58 MVR group compared with the CABG group: left ventricular end-systolic volume index, MR volume, and pl
59 ox regression analyses, the MRI-derived left ventricular end-systolic volume index, RV, and OMR categ
62 r (CRT-D), defined as reduction in both left ventricular end-systolic volume (LVESV) and left atrial
63 lar end-diastolic volume (LVEDV) index, left ventricular end-systolic volume (LVESV) index, and LVEF
65 entricular ejection fraction (LVEF) and left ventricular end-systolic volume (LVESV) relative to base
67 % and <25% reductions, respectively, in left ventricular end-systolic volume [LVESV] at 1 year compar
68 d for age, sex, and body surface area, right ventricular end-systolic volume (P=0.004) strongly predi
69 patients with crypts had lower indexed left ventricular end-systolic volumes (P=0.042) and higher in
70 ng (left ventricular ejection fraction, left ventricular end-systolic volume), plus clinical outcomes
73 estimate, 21.34 mL; bias, -4.93 mL) and left ventricular end-systolic volume (r=0.96; standard error
74 egments at peak stress, and an abnormal left ventricular end-systolic volume response to stress were
75 ranging from 35% to 91%; for predicting left ventricular end-systolic volume response, sensitivity ra
76 etralogy of Fallot in women had larger right ventricular end-systolic volumes (standard deviation sco
77 tricular ejection fraction, from which right ventricular end-systolic volume was derived, was measure
78 left ventricular end-diastolic volume, while ventricular end-systolic volume was reduced by 24 +/- 6%
79 owed that incremental 10% reductions in left ventricular end-systolic volume were associated with cor
80 hic variables, especially the change in left ventricular end-systolic volume with exercise and the ex
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