ライフサイエンスコーパス: visual hallucination

1 ple system atrophy, including a patient with visual hallucinations.

2 ry hallucinations, but not the subgroup with visual hallucinations.

3 al hallucinations, relative to those without visual hallucinations.

4 sential components of a model of oscillating visual hallucinations.

5 echanism underlying 5-HT2A receptor-mediated visual hallucinations.

6 of visual perceptual alterations, including visual hallucinations.

7 al feedback mechanisms can enhance patterned visual hallucinations.

8 utes to the observed regularity of geometric visual hallucinations.

9 These are experienced as geometric visual hallucinations.

10 nitive function in patients with and without visual hallucinations.

11 was made for reporting of persistent formed visual hallucinations.

12 and executive function, were associated with visual hallucinations.

13 y in visual recognition to social vision and visual hallucinations.

14 n the clinical study of whom 32% experienced visual hallucinations.

15 eelings of presence and passage) and complex visual hallucinations.

16 particularly for patients with a history of visual hallucinations.

17 the left ILF (P = .02) than patients without visual hallucinations.

18 suggest three mechanisms underlying complex visual hallucinations.

19 considering current models for mechanisms of visual hallucinations.

20 alterations in visual perception, including visual hallucinations.

21 erse events for ADS-5102 vs placebo included visual hallucinations (15 [23.8%] vs 1 [1.7%]), peripher

22 sm symptoms-71 years (66-92 years), six with visual hallucinations-72 years (64-90 years), seven with

23 differences in ocular pathology between the visual hallucination and non-visual hallucination groups

24 hy patient group included hyper-religiosity, visual hallucinations and cross-modal sensory experience

25 Visual hallucinations and illusions are thought to be ca

26 Fifty patients with visual hallucinations and illusions secondary to degener

27 ctively contribute to the pathophysiology of visual hallucinations and may explain their predominantl

28 s associated with the presence of persistent visual hallucinations and persistent delusions.

29 d cognitive decline with new-onset recurrent visual hallucinations and progressive lethargy.

30 variables persecutory ideation, auditory and visual hallucinations, and diagnosis of probable psychos

31 he possible neuronal events at the origin of visual hallucinations, and further suggests that brain o

32 ypes include myoclonic seizures, auditory or visual hallucinations, and renal failure.

33 Procaine-induced visual hallucinations appeared associated with greater g

34 Visual hallucinations are frequent, disabling complicati

35 in Parkinson's disease and, with respect to visual hallucinations, are an important predictor of cog

36 predominant effects induced by LSD included visual hallucinations, audiovisual synesthesia, and posi

37 n in Alzheimer's disease, as were persistent visual hallucinations, but patients who had dementia wit

38 -HT2A receptors may lead to the formation of visual hallucinations by increasing cortical excitabilit

39 Visual hallucinations can be a major hallmark of late st

40 Here we show that visual hallucinations can be induced in the normal popul

41 between the presence of the Ser23 allele and visual hallucinations (chi2 = 7.5, df = 1, P = 0.006) (P

42 estones of advanced disease (frequent falls, visual hallucinations, cognitive disability and need for

43 older and had an extremely low frequency of visual hallucinations compared with Parkinson's disease.

44 grey matter atrophy patterns associated with visual hallucinations, comparing Parkinson's disease hal

45 he link between abnormal visual function and visual hallucinations, considering current models for me

46 ionships of persistent psychiatric symptoms (visual hallucinations, delusions, depression) to plaques

47 estones of advanced disease (frequent falls, visual hallucinations, dementia and need for residential

48 ribed by several different authors as formed visual hallucinations due to disturbances of the visual

49 , the hallucinators, all of whom experienced visual hallucinations, exhibited grey matter atrophy wit

50 ons to the hippocampus and cortex, while the visual hallucinations experienced by subjects with Demen

51 ch in peduncular hallucinosis, a syndrome of visual hallucinations following subcortical lesions long

52 Patients in the visual hallucinations group had similar disease duration

53 ogy between the visual hallucination and non-visual hallucination groups.

54 ses revealed that patients with a history of visual hallucinations had lower fractional anisotropy in

55 cted for multiple comparisons, patients with visual hallucinations had significantly greater disease

56 Visual hallucinations, illusions and extrapyramidal trac

57 profile correlated strongly with ratings of visual hallucinations, implying that intrinsic brain act

58 nary incontinence in 50% and dementia in 39%.Visual hallucinations in 0%.

59 The literature related to visual hallucinations in ophthalmological settings from

60 The exact pathogenesis of visual hallucinations in Parkinson's disease is not know

61 mographic and ophthalmological correlates of visual hallucinations in Parkinson's disease, the combin

62 endently contributing to the pathogenesis of visual hallucinations in Parkinson's disease.

63 connectome, and lead to the hypothesis that visual hallucinations in patients with COS may be becaus

64 reases appeared to correlate positively with visual hallucination intensity.

65 Current research into visual hallucination is predominantly ophthalmology-led,

66 Complex visual hallucinations may affect some normal individuals

67 Transient monocular visual loss or complex visual hallucinations may lead to neuroanatomic ambiguit

68 tion and the wider clinical context in which visual hallucinations occur.

69 ness and transient delusions and auditory or visual hallucinations occurred in the majority.

70 where loss of vision leads to complex, vivid visual hallucinations of objects, people, and whole scen

71 ent, migraine coma, Charles Bonnet syndrome (visual hallucinations of the blind), schizophrenia, hall

72 any drug (OR, 9.41; 95% CI, 5.26-16.85), and visual hallucinations (OR, 2.13; 95% CI, 1.10-4.13).

73 The milestones of dementia (P < 0.0005) and visual hallucinations (P = 0.02) as well as the accumula

74 ng cognitive impairment, attention deficits, visual hallucinations, parkinsonism, and other neuropsyc

75 ficits included a small visual field defect, visual hallucinations, prosopagnosia, topographical diso

76 ns in subjects with Parkinson's disease with visual hallucinations, relative to those without visual

77 2.1 [5.1] years; P = .03), had more frequent visual hallucinations that did not achieve significance

78 ommon and sometimes curious, if not bizarre, visual hallucinations, the forms of which suggest that e

79 to both fMRI and iEEG, induced a topographic visual hallucination: the patient described seeing indoo

80 cking of this oscillation is correlated with visual hallucinations, thought disorder, and disorganiza

81 significant and consistent risk factors for visual hallucinations, together with new evidence to sug

82 The Charles Bonnet syndrome is a disorder of visual hallucinations typically occurring in older perso

83 For many years, visual hallucinations (VH) in idiopathic Parkinson's dis

84 ht into the pathophysiological mechanisms of visual hallucinations (VHs) in patients with Parkinson d

85 Visual hallucinations (VHs) occur in macular degeneratio

86 The frequency of delusions and visual hallucinations was increased in Parkinson's disea

87 in neuropathological correlate of persistent visual hallucinations was the presence of less severe ta

88 bodies, which is hypothesized to account for visual hallucinations, we found connectivity with this r

89 at death 75.5 +/- 8.0 years) and persistent visual hallucinations were documented in 63%.

90 ies and Alzheimer's disease for auditory and visual hallucinations were especially pronounced for pat

91 Visual hallucinations were more common in the group with

92 n model the four independent determinants of visual hallucinations were rapid eye movement sleep beha

93 erential diagnosis, depicting other forms of visual hallucination which result from a variety of non-